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Nutrition, Airways, & Mandibular Fractures Nutrition, Airways, & Mandibular Fractures

Nutrition, Airways, & Mandibular Fractures - PowerPoint Presentation

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Uploaded On 2022-08-02

Nutrition, Airways, & Mandibular Fractures - PPT Presentation

Dr Hani AlSheikh Radhi Nutrition Starvation 75 kg reservoir of 200000 cal Theoretically sustain the patient for 35 months Reality below 140000 cal Death can occur Body attempts to conserve energy resources and cellular function ID: 932447

patient airway airways state airway patient state airways feeding body tube cal fat mask oral obstruction energy starvation maneuvers

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Slide1

Nutrition, Airways, & Mandibular Fractures

Dr. Hani Al-Sheikh Radhi

Slide2

Nutrition

Starvation

75 kg reservoir of 200,000 cal. Theoretically sustain the patient for 3-5 months.

Reality below 140,000 cal. Death can occur.

Body attempts to conserve energy resources and cellular function

No exterior source of energy. The body will consume fat and muscles. (1kg of muscles will provide 800 cal, 1kg of fat will provide 7000 cal)

Hepatic and GIT systems will be first affected. Gradually drop in Glucose levels. After depletion the brain will depend on fat as source of energy.

Fat can’t cross the blood brain barrier it will be transformed into ketone acids to pass the barrier and that will create state of acidosis. Which can cause depression of the CNS and its function

Slide3

Stress related starvation

Stress means trauma, burn, severe infection. Affect physiology of body

Neuroendocrine mediated process. Catabolism Process controlled by hormones (catecholamine, cortisol, and Glucagon) (Interleukins and Cytokines)

Result

HYPERMETABOLIC STATE

Increase temp., Increase in O2 consumption, Overactive circulation, decrease in insulin, decrease in body mass, lipolysis, increase lactate and pyruvate.

Neuroendocrine Starvation State

Peaks 48-72 hrs. Subside 3-4 days, result in severe loss of vitamins and minerals, severe malnutrition develops within days.

Slide4

Hyper-metabolic state patient treated by metabolic support

Treatment is different than treating real starvation.

Assess Body Mass Index (BMI), Basic Energy Expenditure formula (BEE), Assess Urine Albumin and Nitrogen

Nitrogen in Urine is very important indicator to the levels of protein destruction. Which if positive indicates continuous state of muscular destruction hence continues state of hyper-metabolism.

All food contain hydrogen and oxygen but only proteins contain nitrogen

Treatment

: well nourished patient expected to be back on oral feeding within 1 week we give I.V. 5% Dextrose equivalent to 500 Kcal/day.

Treatment

: Bad nourishment, +/- more than 1 week before oral feeding we need alternative source of feeding.

Slide5

Slide6

NON-ORAL METHODS OF NUTRITIONAL THERAPY

NG Tube (Nasogastric feeding)

Tube inserted from the nose into the stomach

We give the patient

metocoplramide

, relax stomach sphincter pylorus

The tube can sustain the patient 4-6 weeks maximum.

Risk: patient with midface and skull base fractures, tracheostomy

Slide7

Slide8

NON-ORAL METHODS OF NUTRITIONAL THERAPY

PERCUTANEOUS ENDOSCOPIC GASTROSTOMY (PEG)

Old method named Gastrostomy.

No laparotomy, no scar, no peritoneal tearing, less infection risk

Immediate feeding with PEG, less post-op pain

Indication in OMFS,

Cnacer

H&N, Obstruction in pharynx and Larynx

Slide9

1. Initial assessment: Recognize airway obstruction.

2. Perform airway maneuvers, clear the airway, reposition patient

3. Use artificial airways, and perform bag-valve-mask ventilation

4. Perform endotracheal intubation.

5. Create a surgical airway if unable to intubate.Emergency Airway Management in the Traumatized Patient

Slide10

Initial Assessment

Look

1- Patients with compromised airways sits up right.

2- Look of terror on their face.

3- Anxiety and agitation are signs of hypoxia.

4- Altered Level of consciousness are signs of hypercarbia.Listen

FeelQuick way: Ask the patient to describe the injury. If he was able to do it then he doesn’t require rapid airway intervention. Obstruction in the upper airway usually cause noisy breathing,

stridor,and

wheezing.

feel for facial fractures, soft tissue swelling, subcutaneous air in the neck, laryngeal tenderness, and dental injuries during the primary assessment

Slide11

Maintain the patient’s airways

+ to the basic maneuvers

Oropharyngeal & Nasopharyngeal tubes

Temporary until definitive intubation

Oropharyngeal (OPA)Patients with no history no gagging

Determine the proper size and insertion method Nasopharyngeal (NPA)Patients with no history no gaggingDetermine the proper size and insertion method

Slide12

Oropharyngeal (OPA)

Slide13

Nasopharyngeal Airway(NPA)

Slide14

BAG-VALVE-MASK (BVM) VENTILATION

Provides positive pressure

Used usually with other maneuvers

MOANS Difficulty Test (Mask, Obesity/obstruction, Age, No teeth, Stiffness [Asthma, COPD])

Used usually with other maneuvers

Slide15

ENDOTRACHEAL INTUBATION

placement of a flexible plastic tube into the trachea to maintain an open airway

2 types: 1- Direct

Laryngeoscopy

2-

Nasotracheal IntubationIndications1- Apnea2- Hypoxia less than 90% SPo23- Inadequate Mask seal.

4- No patent airways.5-GCS less than 9.6- Unstable midface fracture.7- Shock BP less than 80 mmHg

Slide16

Slide17

Adjunct Medications

Premedication

I.V. Lidocaine (anesthesia)

Opioids (Analgesia)

Atropine (Decrease Secretions)

Etomidate

(Sedation)Succinylcholine (Muscle Relaxing)

Slide18

SURGICAL AIRWAY

Slide19