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Honey Herce, MD Department of Ophthalmology, Honey Herce, MD Department of Ophthalmology,

Honey Herce, MD Department of Ophthalmology, - PowerPoint Presentation

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Honey Herce, MD Department of Ophthalmology, - PPT Presentation

Baylor College of Medicine and Texas Childrens Hospital Common Ophthalmic Findings in Children Objectives Be able to identify common ophthalmic findings in children Be able to perform a basic eye exam ID: 913925

refractive eye children amblyopia eye refractive amblyopia children findings strabismus ophthalmic high eyes hemorrhage orbital vision vitreous conjunctivitiscellulitiscommon errornldochalazionptosis

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Slide1

Honey Herce, MD

Department of Ophthalmology,

Baylor College of Medicine and Texas Children's Hospital

Common Ophthalmic Findings in Children

Slide2

Objectives

Be able to identify common ophthalmic findings in children.Be able to perform a basic eye exam.Be able to manage common ophthalmic findings in children.

Common Ophthalmic Findings in Children

Slide3

Slide4

Slide5

Slide6

Pediatric eye examVision PupilsOcular alignment and motilityGross observationPediatric Ophthalmologist will do a full exam (8-point eye exam) consisting of the following:Tonometry (checking eye pressure)

Slit lamp examinationRefraction (Checking for glasses prescription).Dilated fundus exam.

Slide7

Vision In ChildrenBlinks to light/Reacts to light: develops by GA 31 weeksFix and follow: develops by 2-3 mos gestational age.Pictures: Allen chartLetters: HOTV chart/matching, Snellen Chart, crowding bars

* With all of these, you want to look for symmetry*

Slide8

Fix and FollowDistraction is keyObserve if they watch you and if they can follow you.Check monocularly and see if they have a preference for an eye.

Slide9

Allen Chart

Tumbling E’s

Slide10

Inaccurate visual acuity tests!!!

Vision Tests for Verbal children

Slide11

Accurate visual acuity tests!!!!

Slide12

Ocular Alignment and motilityGross ObservationCorneal light reflex testingHirschberg testingKrimsky testingCover/Alternate cover testing:

Slide13

Slide14

Bruckner testSimple test for amblyopia risk factorsUse your direct ophthalmoscopeShine the light on each eye alone (red reflex test) Then both eyes togetherLook thru view finder

Slide15

Amblyopia risk factorsStrabismusHigh or asymmetric refractive errorsDeprivation: i.e. cataract

Slide16

Abnormal red reflexCorneal opacitiesCataractVitreous hemorrhageRetinal detachmentRetinoblastomaStrabismusRefractive errors

Slide17

Ocular Motility Document each function of each extraocular muscle

Slide18

Right Eye Left Eye

SR IO IO SR

LR MR MR LR IR SO

SO IR

Slide19

Ocular Anatomy

Slide20

Slide21

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide22

Strabismus* *G.

strabismos, squinting

= any Misalignment of the eyes~2-4% of all people

Slide23

Slide24

Strabismus Which direction in the misalignment?

Control of the deviation?

ESO – turning inwardEsophoria (tendency to turn in, usually controlled)

Esotropia

(constant misalignment)

Intermittent

esotropia

(intermittent misalignment)

EXO – turning outward

Exo

phoria

(tendency to turn out, usually controlled)

Exotropia

(constant misalignment)

Intermittent

exotropia

(intermittent misalignment)

Measurements are done at Distance (20 feet) and near (1/6 m)

Slide25

6 extra-ocular muscles (EOMs) per eye

Medial/

nose

Slide26

How do we assess Strabismus? Assess Alignment

Assess Movement of the eyes

Slide27

How do we assess Strabismus? Assess Alignment

Make assessments when they walk into the room and as you are examining them.Are the eyes straight looking straight head?If not

Describe the deviation/s

Slide28

Alignment Assessment

Gross observationObvious misalignment

Corneal light reflex centration In same place on eye? Eyes are straightMight look crossed but really is straightIn different positions on eyes? An eye is misaligned

Left eye is subtly

esotropic

Slide29

Alignment Assessment

Cover testing

Cover fixing eye and crossed eye shifts to pick up fixation

Gold standard test

Slide30

Congenital Esotropia a.k.a. Infantile EsotropiaPresentation before 6 mos.They usually will have a large deviation.Crossing in a baby that does not go away and as they get older gets more constant.

May run in families.Surgery is indicated.Increased frequency in Cerebral Palsy and hydrocephalus

Slide31

Accomodative EsotropiaCharacterized by:Onset after age 18 mos. Usu intermittent at onset but then becomes constant. when tired or sick, can appear acutely

They can develop amblyopia on the eye that turns in more.Glasses keep the eyes straight.Associated findings:+3.00D to +10.00D- High refractive error.Often hereditaryAmblyopia frequent- 2/3

Slide32

TreatmentTreat Amblyopia (patching therapy).Correct refractive error with glasses.Surgery for residual eye crossing with the glasses.

Slide33

Intermittent ExotropiaIntermittent misalignment, however if long standing can turn into a constant deviation. Eyes usually are straight then becomes exotropic and the patient blinks and they go back to straight. Onset varies from 6

mos – 6 yrsCan be progressive 75%TreatmentGlasses if high refractive error.Alternate occlusion therapyPrism therapy (base-in prisms)Over-minus glasses: this induces an accomodative convergence.

Surgery

Slide34

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide35

“Lazy Eye”

Not a medical termUsed by patients, relatives to describe: - Strabismus - Amblyopia

- A blind eye (any cause) - Ptosis - Almost anything

Slide36

Amblyopia*

*G. amblyopia, dimness of vision

Definition: poor vision in an eye (or both eyes) even though the retina and optic nerve are normal and even though the correct glasses are in placeSo why can’t the eye see?

Usually unilateral

~2-5% of the population

Slide37

The eye can’t see because it has been “turned off”

or suppressed by the brainso that even with the right

glasses correction - the vision isn't right3 Causes of suppression

are the causes of amblyopiaStrabismus

(misaligned eyes) – brain suppresses the deviated eye

Refractive errors

(high degrees of nearsightedness, farsightedness, or astigmatism, especially if the refractive errors are asymmetric) – brain suppress the eye/s w blurrier vision

Deprivation

(lack of form vision due to blockage of light getting to retina – cataract, ptosis, vitreous hemorrhage) – brain suppress the eye/s

Slide38

How do we test for amblyopia?Check vision! Check for the risk factors that may lead to amblyopia

What are the THREE risk factors for amblyopia??

Slide39

3 Amblyopia Risk FactorsStrabismusHigh or asymmetric refractive errors (uncorrected)

DeprivationCataract Ptosis

Slide40

Amblyopia TreatmentClear the ocular media (

ie cataract extraction, ptosis surgery, etc.) - if needed

Provide glasses - if neededPatch the better eye or Blur the better eye with atropine drops

Atropine 1 drop, to the good eye, 2d a week

Patch good eye 2h/d

Slide41

FollowupCheck vision every 2-3 months while undergoing patching or atropine Stop treatment whenVision is equal/normal in each eye

ORVision improvement in amblyopic eye stalls in spite of therapy (cant cure all amblyopia)Continue follow up to ensure amblyopia doesn’t recur (won’t after age 7)

Slide42

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide43

Leukocoria* Means “white pupil”This occurs when the pupil is white rather than black or when there is an abnormal red reflex.The red reflex appears red because when light enters the eye, the retina reflects a small amount of light. The reddish-orange color is the normal color of the retina. Anything obscuring it will change the red reflex.

Slide44

LeukocoriaThis can be due to many causes.CataractRetinoblastomaVitreous hemorrhageRetinal detachmentRetinopathy of prematurity

Intraocular infectionHigh refractive error or Asymmetric refractive error

Slide45

Slide46

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide47

Nasolacrimal duct obstructionObstruction at distal end of duct: Valve of Hasner6% of newborns90% resolve by 6 months95% resolve by 9 monthsTearing, mucous dischargeWHITE eye

Slide48

NLDO TreatmentMassage sac with each feed Topical antibiotic drops after massagewhen infectedProbe in office age 9-12 monthsProbe in OR anytime after 6-9 months– can stent with tubes or do balloon dilation.

Slide49

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide50

ChalazionA localized bump on the eyelid due to blockage of one or more meibomian glands. (oil glands) This is different from a stye or a hordeolum which is a small abscess due to the glands getting infected.

Slide51

Slide52

TreatmentFrequent warm compresses and lid scrubs help unclog the oil glands. Anti-inflammatory eye drops and ointments sometimes are needed.For long-standing chalazia, surgical drainage may be necessary.

Slide53

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide54

PtosisPtosis is a drooping of the upper eyelid.Can cause amblyopiaObstruction of the visual axisCan cause anisometropic astigmatismWatch the patient when they walk in

Chin up head posture is good: means they are using the eye.

Slide55

Causes of ptosisCongenital ptosis: frequently hereditary3rd nerve palsyHorner’s syndrome1-2 mm ptosis

of upper and lower lidsAnisocoria worse in the darkMyasthenia gravisMarcus Gunn jaw wink

Slide56

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide57

ConjunctivitisIt is an inflammation or infection of the conjunctiva. The conjunctiva is the loose connective tissue that covers the surface of the eyeball. It is one of the most common complaints that present to the emergency room department. 30%There are many causes of a conjunctivitis ranging from allergic to viral to bacterial to chemical.

Slide58

Pediatric red eye

Slide59

ConjunctivitisAge and presentation will dictate managementNeonatal: gonorrhea, chlamydia, HSV

URI Symptoms: adenovirusPurulent: bacterialAllergic

Slide60

ConjunctivitisViralAcute or subacute onset with usually an exposure history.Minimal pain level .Clear watery discharge, photophobia and itching*

Very contagiousTreatment: Self limited, supportive care with artificial tears and cool compresses.BacterialAcute onset, with minimal pain. Chemotic conjunctiva, copious thich and purulent dischargeTreat with antibiotic eye drops. i.e.

PolytrimAllergicAcute ro subacute onset. May be seasonalHallmark of symptom is itching and a clear watery discharge. Treat with antihistamine drops and mast cell stabilizers. I.e. Zaditor, Pataday

Slide61

Neonatal ConjunctivitisThree etiologies: Chemical, Bacterial, Viral.Chemical conjunctivitis: within the first 24 hours of life.Neisseria Gonorrhea: 3-5 days after birth.Present with acute conjunctivitis with severe lid edema and

mucopurulent discharge.C. Trachomatis: 5-14 days after birth.Mild swelling and mucopurulent discharge. Important to also treat systemically to prevent PneumoniaHSV: 1-2 weeks.

Slide62

Strabismus

AmblyopiaLeukocoriaCongenital CataractRetinoblastomaROPOthers: vitreous hemorrhage, high refractive errorNLDOChalazionPtosis

ConjunctivitisCellulitisCommon Ophthalmic Findings in Children

Slide63

Preseptal CellulitisInflammation and infection of the tissues anterior to the orbital septum.Swollen and erythematous lid, but the eye is white and motility is full. Three main routesDirect inoculation with eyelid trauma i.e. insect bites.Spread from contiguous structures

Paranasal sinuses i.e. ethmoid diseaseChalazia/HordeoleumDacryocystitisImpetigoHSVHematogenous

Slide64

Preseptal CellulitisTreatment: Oral antibioticsIf the patient does not respond to oral antibiotics in 48 hours or if suspecting orbital disease- IV antibiotics needed.CT scan if suspecting orbital cellulitis.

Slide65

Orbital CellulitisInfection of orbital contents posterior to the orbital septumEtiology:SinusitisDacryocystitis

DacryoadenitisDental infectionsIntracranial infectionsTraumaPostorbital surgery

Slide66

Orbital CellulitisSymptoms:Decreased visionPain with eye movements

DiplopiaSigns:FeverLid erythema and edemaProptosis

Motility restrictionsRelative afferent pupillary defectConjunctival injectionOptic nerve swelling

Slide67

Orbital CellulitisCT to further assess extent and presence of orbital abscessBlood culturesBroad spectrum antibiotics

Special considerations if immunocompromisedConcern for fungal (Mucormycosis)Consultations

OphthalmologyENTInfectious Disease

Slide68

Questions?

Slide69