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GASTRIC OUTLET OBSTRUCTION GASTRIC OUTLET OBSTRUCTION

GASTRIC OUTLET OBSTRUCTION - PowerPoint Presentation

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Uploaded On 2022-06-18

GASTRIC OUTLET OBSTRUCTION - PPT Presentation

AND ITS SURGICAL MANAGEMENT DEFINITION Gastric Outlet Obstruction is clinical or pathophysiological consequence of any disease process that produces mechanical impediment to gastric emptying ID: 920568

metabolic gastric outlet obstruction gastric metabolic obstruction outlet carcinoma patient ulcer stomach large tube sodium xray disease leads pyloric

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Slide1

GASTRIC OUTLET OBSTRUCTION AND ITS SURGICAL MANAGEMENT

Slide2

DEFINITIONGastric Outlet Obstruction is clinical or pathophysiological consequence of any disease process that produces mechanical impediment to gastric emptying

Slide3

Gastric Outlet Obstruction

Slide4

EtiologyBENIGNPeptic Ulcer disease

Ingestion of Caustics

Trichobezoars

( Hairballs)

Adult hypertrophic Pyloric stenosis

Pyloric mucosal diaphragm

Pancreatic

Pseudocysts

Slide5

BARIATRIC PROCEDURESVertical banded gastroplasty

Roux-en-Y gastric bypass

Slide6

ETIOLOGYMALIGNANTCarcinoma of StomachPeriampullary

carcinomas

Carcinoma Head of pancreas

ampullary

carcinoma

Carcinoma of second part of duodenum

cholangiocarcinomas

Slide7

PRESENTATIONHISTORY

Epigastric

or left

hyochondrial

pain

> which is most common feature in peptic ulcer disease

vomiting

>unpleasant smell

> copious amounts

>projectile

>

NON BILIOUS

>contains undigested food particles taken hours to several days ago

Slide8

HistoryFeeling of unwellAnorexia NauseaEarly satiety

Weight loss

Abdominal swelling

Slide9

EXaminationGENERAL PHYSICAL EXAMINATIONChronically ill looking patient

Wasted

Dehydrated

Pale

Left

supraclavicular

lymphadenopathy

(with malignant obstruction)

Slide10

ExaminationABDOMINALDistended stomach ( fullness in epigastrium)Visible Gastric

Peristalisis

Succussion

splash

Hepatosplenomegally

Look for

Ascities

( sign of Carcinoma spread)

Slide11

Epigastric fullness

Slide12

Succussion splashSloshing sound heard through stethoscope place over

epigastrium

during sudden movement of the patient

Slide13

.

Slide14

CLINICAL FEATURESMETABOLIC EFFECTSVomiting of Hydochloric

acid ( HCL) leads to

hypochloremic

metabolic alkalosis

Kidneys respond by excreting Bicarbonate and conserving chloride

This bicarbonate is excreted with sodium

So with time patient becomes more profoundly dehydrated and

hyponatremic

Slide15

Metabolic effectsBecause of dehydration body responds to preserve intravascular volume by sodium retentionNow potassium and hydrogen are excreted in preference to preserve sodium

This leads to paradoxical

aciduria

( acidic urine despite metabolic alkalosis)

Hypokalemia

ensues

Slide16

Metabolic effectsAlkalosis leads to lowering of circulating ionised calcium This can lead to

tetany

Slide17

INVESTIGATIONSCBCS/E

LFT

Test for H pylori

Slide18

InvestigationsABG s : Metabolic AlkalosisUrine C/E: paradoxical aciduria

Slide19

RadiologyPlane Xray Erect Abdomen:

Large Gastric shadow and Large amount of Gastric fluid

Slide20

Plane Xray Abdomen

Slide21

Barium Meal6 hour peroid of fasting is observed prior to studyBarium

sulphate

is ingested by the patient

Xray

images are taken at 20 to 30 minutes interval in supine position

Slide22

Barium meal

Slide23

Upper GI endoscopy visualize Gastric Outlet Biopsy

Slide24

Gastric outlet obstruction

Slide25

CT scanFor extraluminal obstruction Periampullary

carcinomas

Slide26

ManagementTwo Aims1.Correct metabolic abnormality

2.Deal with mechanical obstruction

Slide27

Correcting Metabolic AbnormalitiesPass double large Bore IV linePass wide bore

nasogastric

tube to empty the stomach

Sometimes an

orogastric

tube is required to

lavage

and empty the stomach as

nasogastric

tube may not be sufficiently large to deal with contents of the stomach

Slide28

Correcting metabolic abnormalitiesIntravenous Normal Saline (0.9% NaCI) with Potassium Supplementation

Correct anemia

Slide29

managementEarly cases may settle with conservative management

NPO

ANTACIDS

PPI

as the edema around the ulcer diminishes as the ulcer is healed

Slide30

Surgery for benign GOO

Slide31

Pyloroplasty with vagotomy

Slide32

Truncal Vagotomy and Antrectomy and Billroth Reconstructions

Slide33

PYLOROPLASTYFINEYJABOULEYSHeineke-Mikulicz

Slide34

Slide35

Slide36

Slide37

GASTROJEJUNOSTOMY

Slide38

BALLOON DIALATATIONENDOSCOPIC DALATION Repeated dilatations neededMay cause

perforation

Slide39

Endoscopic stenting for unresectable tumor

Slide40

stenting