Psych Rehab Nursing Fall 2009 Outline Overview of anatomy and physiology Diagnostic tests Musculoskeletal trauma Problems of the musculoskeletal system Osteoporosis Osteoarthritis Rheumatoid arthritis ID: 775020
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Slide1
Musculoskeletal Disorders
Psych Rehab Nursing
Fall 2009
Slide2Outline
Overview of anatomy and physiology
Diagnostic tests
Musculoskeletal trauma
Problems of the musculoskeletal system
Osteoporosis
Osteoarthritis
Rheumatoid arthritis
Gout
Other musculoskeletal problems
Slide3Review
Purpose of the musculoskeletal system
-
Supporting framework for attachment of muscles
and other tissues
- Protects vital organs and soft tissues
- Joints, bones, cartilage make up the skeletal
system
- Bones enable movement of the body by acting as
levers and points of muscle
attachments
(muscles ONLY contract: opposing muscles allow flexion & extension. Muscles are conductors of electrical energy)
Slide4Bones
Function
Framework for the attachment of muscles and other tissues
Protection
Production of blood cells in the red marrow
-
Which bones are involved in the process?
-
Facilitate movement by contracting muscles
-
Storage area for calcium and phosphorus
-
Lipids (energy sources) are stored in adipose
cells of the yellow marrow
Slide5Bones
Blood cells are produced in bone marrow (see below)
Red marrow in flat bones (sternum, scapulae, skull, ribs, vertebrae, pelvis, epiphyseal ends of long bones, i.e., femur and humerus (marrow runs all the way through the bone, but the cells are released from the epiphyseal ends)
Femur head; view of cortex; view of red bone marrow; view of yellow bone marrow (2009)
Slide6Bones
Ligaments
(fibrous connective tissue connecting bone to bone)
Tendons
(connect muscle to bone) Example: Achilles tendon attaches calf muscles to the ankle bone while a ligament holds the calf and thigh bones together at the knee joint
(
Pollick
, 2008)
Slide7Bones: Types
Long bones
Short bones
Flat bones
Irregular bones
Sesamoid (or round) bones
Sutural or Wormian bones
Slide8Long Bones
Long bonesLonger than they are wideA shaft with two ends - the shaft widens at the end of the bone Contain yellow bone marrow and red bone marrowHumerus (proximal) ; radius and ulna (distal) of the upper extremity; femur (proximal), tibia (anterior/distal) and fibula (posterior/distal) of the lower extremity
Slide9Long Bones
Examples of long bonesMetacarpals Phalanges9 14 10 1511 1612 13
Slide10Red and Yellow Bone Marrow
Half of the bone marrow is red (in adults)Red marrow is found mainly in the flat bones (hip bone, sternum, skull, ribs, vertebrae and shoulder blades)Also found in the spongy material in the proximal ends of the long bones – femur and humerus Yellow marrow is found in the hollow interior of the middle portion of long bones
Femur head; view of cortex; view of red bone marrow; view of yellow bone marrow (2009)
Slide11Bone Marrow
Hematopoesis (blood formation)RBC storageProduction of WBCsPlatelets (formed in bone marrow; aid in clotting)High fat content (long bones) fat embolus
“Fat embolism syndrome, a condition characterized by hypoxia, bilateral pulmonary infiltrates, and mental status change, is commonly thought of in association with long-bone trauma. … Although studies suggest that embolization events infrequently result in clinically apparent fat embolism syndrome, clinicians should be vigilant in considering fat embolism as a causative agent for postoperative respiratory distress.”(Glazer & Onion, 2001)
Slide12Short Bones
Short bones
Cube-like; about as long as they are wide
Contain mostly spongy bone
Outside surface consists of a thin layer of compact bone
Located in the hands and feet (metacarpals)
Patella
Slide13Long Bones
??
Slide14Flat Bones
Flat bones
Thin and flat
Found where the need is for a broad surface area for muscular attachment or where extra protection is needed
Examples: Skull; Pelvis; Sternum; Rib cage; Scapula
In adults most RBCs are formed in flat bones
Slide15Bones: Microscopic Anatomy
Osteoprogenitor
cells are
multipotential
skeletal cells; the stem cells for the skeletal system.
Osteoblasts
are bone forming cells; they lay down the bond tissue. Can be stimulated in cancer.
Osteocytes
are mature bone cells that become trapped at maturity in a matrix; they maintain the bone
Osteoclasts
are bone reabsorbing cells that destroy old bone. Slow process. Inc cancer, these cells are destroyed and not replaced (osteoporosis).
Collagen
is soft, strong connective tissue that supports and reinforces the mineralized matrix – stronger than steel.
Slide16Irregular Bones
Examples are the vertebrae; sacrum; coccyx; temporal; sphenoid;
ethmoid
(in skull);
zygomatic
(cheek bone);
maxilla; mandible; palatine; inferior nasal
concha
; and hyoid
Serve as protection (example: vertebrae protects spinal cord)
Allowing multiple anchor points for skeletal muscle (example: sacrum)
Slide17Bones
Ligaments (bone to bone): fibrous connective tissue connecting bone to bone
Tendons: (connect muscle to bone) Example:
achilles
tendon attaches calf muscles to the ankle bone while a ligament holds the calf and thigh bones together at the knee joint.
Slide18Sesamoid (Round) Bones Sutural, or Wormian bones
An example of a Sesamoid bone is the patella Sesamoid bones are embedded within tendons; Act to protect tendonsSutural or Wormian bones occur between the sutures of the cranial bones
Slide19Bones: Types
Long Bones: weight bearing, strong, curved
Short bones: compact on surface, spongy center &
cuboidal
(metatarsals)
Flat bones: parallel surfaces of the body,
protecitve
funciton
, broad attachment surface (ribs, pelvis, skull,
ilium
)
Irregular bones: various functions: vertebrae & some facial bones
Sesamoid
(or round) bones: embedded in tendons, allow change of direction of movement, like a pulley (patella)
Sutural
or
Wormian
bones: between sutures of skull (between large flat bones of skull)
Slide20Joints
A
di
arthrosis
joint is a freely moveable joint – always a synovial joint
Types of synovial joints
Gliding
(Two sliding surfaces) (Example - between carpals)
Hinge
(Concave surface with convex surface) (Example – between
humerus
and ulna)
Pivot
(Rounded end fits into ring of bone and ligament (Example – between atlas (C1) and
axia
(C2) vertebrae
Slide21Joints
Synovial Joint
Diarthrosis Joint
(all are synovial joints)
Slide22Joints, Synovial types, cont.
Ball and socket joint
(Ball-shaped head with cup-shaped socket) (Example – Between femur and pelvis)
Condyloid
joint
(Oval
condyle
with oval cavity) (Example – between metacarpals and phalanges)
Saddle joint
(Each surface is both concave and convex) (Example – Between
carpus
and the first metacarpal)
Hinge joint
(
interphalangeal
joints)
Pivot joint
(able to rotate – neck, forearm, knees)
Slide23Muscles
Source of power and movement
Three types…
Skeletal Muscle
Cardiac Muscle
Smooth Muscle
Slide24Skeletal Muscle
Skeletal Muscle
Attached to bones and causes movements of the body.
Also called striated
muscle (
actin
&??)
because of its banding pattern, or voluntary muscle (because muscle contraction can be consciously controlled
)
Slide25Cardiac Muscle
Cardiac muscle
Responsible for the rhythmic contractions of the heart
Muscle is involuntary
Generates its own stimuli to initiate muscle contraction
Microscopically striated like skeletal muscle
Striations join together in bundles to allow coordinated action
Involuntary and
autorhythmic
. Some cardiac muscles function as built in pacemakers.
Slide26Smooth Muscle
Smooth Muscle
Lines the walls of hollow organs
(Example: lines the walls of blood vessels and of the digestive tract where it functions to advance the movement of substances.
Contraction is relatively slow and
involuntary
Microscopically smooth (not striated)
Slide27Slide28Basic components
Muscles are stimulated by motor neurons.
Richly supplied w/ arteries and veins and have intimate contact w/a rich capillary network b/c of high energy demands.
Slide29Diagnostic Tests forMusculoskeletal
X-ray
Electromyogram (EMG)
Arthroscopy
Arthrogram
Computerized Axial Tomography (CT)
Magnetic Resonance Imaging (MRI)
Bone Scan
Arthrocentesis
Laboratory Testing
Antinuclear Antibodies (ANA)
Ca+, P
Rheumatoid Factor (RF)
Erythrocyte Sedimentation Rate (ESR)
Uric Acid
Slide30Chemical action in muscle
ATP – ADP energy cycle. Critical to muscles ability to get energy they need to do the work they need to do.
Slide31Soft Tissue Injuries
Sprains/Strains
Sports-related
Dislocation/Subluxation
Carpal Tunnel Syndrome
Rotator cuff
Repetitive Strain
Meniscus Injury
Bursitis
Muscle spasms
Slide32Dislocation / Subluxation
Dislocation of jointSubluxation (partial dislocation of joint)Candidate joints are shoulders, fingers, kneecaps
Slide33Trauma to Bone
Highest incidence
MALES 15-24 years of age
or
elderly females 65 years of age, or older
Why are females in this age group affected?
A result of a blow to the body, a fall, or another accident
Slide34Rotator Cuff Injury
A rotator cuff injury includes any type of irritation or damage to the rotator cuff muscles or tendons. Causes of a rotator cuff injury may include falling, lifting and repetitive arm activities — especially those done overhead, such as throwing a baseball or placing items on overhead shelves (Mayo Clinic, 2008)
Slide35Meniscus Injury
Symptoms of medial meniscus tear A history of trauma or twisting of the knee Pain on the inner surface of the knee joint Swelling of the knee within 24-48 hours of injury Inability to bend knee fully- this may be associated with pain or a clicking noise A positive sign (pain and/or clicking noise) during a "McMurrays test" Pain when rotating and pressing down on the knee in prone position (video). "Locking" of the knee Inability to weight bear on the affected side (SIC, 2008)
Slide36Muscle Spasm
Inflammation that occurs when a muscle is over-stretched or tornThe back is a common area for inflammation to occurWhy? Poor body mechanics (cold or heat might be effective, reduce inflammation & relax muscles)
Slide37Bursitis:Inflammation of the Bursa(Pain, fluid build up, calcium deposits & loss of motion in the joint. Prevention is to build up activity gradually. Treatment: avoid what is causing the problem, NSAIDS, steroids, ice. In some cases surgery might be necessary.
Slide38Types of Complete Fractures
Closed (Simple)
Open (Compound
):
riskiest due to infection, bone breaks through skin.
Transverse
Oblique
Spiral
Comminuted
Colles
’ fracture:
fracture of wrist on inside portion of wrist. Treatment is reduce it & cast it. (happens when someone falls down). Responds well to cast.
Slide39Fractures
Fractures commonly tear blood vessels, producing a hematoma
This area of hematoma is commonly used as an area to anesthetize the
periosteum
because
thisis
where the blood vessels are located.
Reduction usually requires anesthesia
Slide40Healing stages
After several weeks the
periosteum
is beginning to heal & lay down scar tissue.
Trabecular
cone has begun to grow over the break. There is callus formation w/ the
osteoblasts
After several months…..
Slide41Healing stages, cont
Remodeling: over the next months or years, the bone shape returns to normal as
osteoclasts
absorb extra cells and
osteoblsats
generate new cells and bone. The bone will be thickened somewhat at the fracture…
Fully healed fractures in children are indistinguishable from the original bone b/c the growth plates are open. However, multiple
fxs
in various stages of healing are a strong indicator of child abuse
Slide42Closed Simple Fracture
Only bone damageLittle or no soft tissue damage Does not penetrate skin
Slide43Open Fracture
Probably need surgery… lot of time spent cleaning the wound
Wound may be left open for a while…
Slide44Transverse Bone Fracture
Often caused by direct traumatic injuryBone has been broken giving rise to a transverse break or fissure within the bone at a right angle to the long portion of the bone
Slide45Oblique Bone Fracture
Extremely rare type of breakAn oblique break in the bone which is very unstable (break at an angle)Bone still together…
Slide46Spiral Fractures
Bone is broken due to twisting-type motionUnstable fractureLooks like corkscrew – runs parallel with the axis of the broken bone
Slide47Comminuted (crushed) Fracture
More than two fragments of bone have been broken offHighly unstable with many bone fragments(Fixed w/ rods & screws after taking all the pieces out)
Slide48Types of Incomplete Fractures
Greenstick
Torus (closed) (side of the bone bends but does not break)
Bowing
Stress
Slide49Greenstick fracture
Usually seen in childrenBone is usually “bent” and broken on the outside of the bendIf kept straight, heals quickly(sometimes doesn’t need a cast… sometimes overtreated)
Slide50Stress Fracture
Incomplete fractureCaused by “unusual or repeated” stress – this in contrast to other type fractures resulting from traumaCommon sports injury and among soldiers from marchingTiny hairline fractures
Most common symptom is pain
Most are not associated with swelling or redness, but tenderness to palpation
Tibia (shin splints-runners)
and metatarsal bones affected in runners
Slide51Stress Fracture
Example of a stress fracture Common runner-type fracture
Slide52Other Types of Fractures
Pathologic Fracture
May occur during normal activity or after minimal injury
(Is associated with what
?) common in elderly w/
osteoarthrits
or osteoporosis
Fatigue or Stress Fracture
The muscles associated with the bones are unable to absorb energy as they usually do
Avulsion Fracture
A strong ligament or tendon pulls a fragment of the bone away from the rest of it
Impacted Fracture
Fracture fragments are pushed into each other
Slide53Clinical Manifestations
Pain
Loss of normal function
Obvious deformity
Excessive motion
Crepitus
(fluid builds up on joint, can be felt at the joint)
Edema
Warmth
Ecchymosis
Loss of sensation
Signs of shock
X-ray evidence
Slide54Factors that hinder good callus formation
Inadequate reduction of the fracture
Inefficient immobilization
Excessive edema at the fracture site, impeding the supply of nutrients
Too much bone lost at time of injury to permit bridging of broken ends
Infection at the site of injury
Bone necrosis
Anemia or other systemic conditions
Endocrine imbalance (parathyroid not enough Ca)
Poor dietary intake
Slide55Neurovascular Assessment (5 Ps)
Pain
Paresthesia (tingling, pricking, or numbness of the skin)
Pallor
Pulses
Paralysis
Slide56Goals
Prevent injury
Maintain strength
Promote comfort
Maintain intact neurovascular status
Slide57Treatment Objectives for Fracture
Reduction of fracture
Maintenance of fragments in correct alignment
Prevention of excessive loss of mobility and muscle tone
Slide58Collaborative Management
Health History
X-ray/CT/MRI/Scan
Fx
Reduction
Fx
Immobilization
MEDS
Analgesics
Antibiotics
Tetanus
Toxoid
(good for 10 years)
Slide59Immobilization of Bones
1)
Physiologic
Splintage
- naturally occurring phenomenon related to pain that causes guarding, muscle spasms, and avoidance of further use. There is a desire to rest the whole body until some repair has occurred.
2)
External orthopedic
splintage
- with devices such as casts
3)
Internal Fixation
with screws, pins, rods or plates to hold the opposing ends of the fracture in place
Slide60Bone Healing
1.
Hematoma formation
(situates between broken fragments)
2.
Fibrin meshwork formation
(blood vessels grow into a jelly-like matrix of the blood clot – WBCs are brought to the site
3.
Invasion of
osteoblasts
(produces matrix that becomes mineralized)
4.
Callus formation
(usually shows up by x-ray 6 weeks in adults/less time in children
5.
Remodeling
(bones are constantly changing –
osteoclasts
break down old bone so
osteoblasts
can replace it with new bone tissue – a process called remodeling.
What can impair bone healing?
Slide61Bone Healing
Slide62Fractures: terms commonly used
Reduction: re-establishment of the normal position of ..
Dislocation
Fracture
Internal fixation is the surgical placement of steel material into the bone to hold it in place. Used when healing would be impaired or immobility of the
fx
is a problem.
Slide63Factors that Hinder Good Callus Formation
Inadequate reduction of the fracture
Inefficient immobilization
Excessive edema at fracture site, impeding the supply of nutrients
Too much bone lost at time of injury to permit bridging of broken ends
Infection at the site of injury
Bone necrosis
Anemia or other systemic conditions
Endocrine imbalance
Poor dietary intake
Slide64Immobilization and Care
External fixation devices
Casts (plaster casts can be bi-
valved
to allow for swelling)-this means it is cut into two pieces, lengthwise, so that the area beneath the cast can be observed.
Splints
Brace or cast-brace
Traction (weights should hang freely)
Skin traction
Skeletal traction
Balanced suspension
Counter-traction
Internal
Fixators
Plates and screws
Rods
Prosthetics
Bone Stimulation
Slide65External Fixator
Used when a cast would not allow proper alignment/immobilization of the fractureProper cleaning to prevent infection is required No cast, just curlex (gauze)
Slide66Traction
Aligns the ends of a fracture by pulling the limb into a straight position
Helps manage muscle spasm r/t
fracture
(weights should hang freely)
Skin traction
Skeletal traction
Balanced suspension
Counter – traction
Skin integrity or back massage might be helpful. Often used pre-op to keep them immobile
Slide67Bone Stimulation
Used when satisfactory healing is not occurring naturally Application of a low electrical current to the fracturePromotes the speed of bone healing
Slide68Nursing Diagnoses
Risk for Neurovascular Dysfunction
Pain
Risk for Infection
(Risk for) Impaired Skin Integrity
Risk for Nutrition Deficit
Risk for Injury
Knowledge
Deficit (teaching about care of the site)
Risk for impaired perfusion
Slide69Complications
Who is at risk?
: 0.5%-2% long bones, 10% hip
DVT
Stroke or Pulmonary Embolus
S/S of PE:
Hemoptysis
(coughing up of blood),
pleuritic
chest pain,
dyspnea
,
rales
Fat Embolism Syndrome (When fat enters the circulation)
12-48 hrs after fracture
Usually associated with
fx
of long bone or pelvis (
fx
of hip is at highest risk)
Men 20 to 40 years of age and older adults 70 to 80 years of age at greatest risk for development
Mental status changes (hypoxemia),
tachypnea
,
dyspnea
, tachycardia, temperature,
petechia
of upper body and
axilla
, feeling of impending doom
Slide70Complications
Compartment Syndrome 4-12 hrs after fractureCompartments are areas of the body in which muscles, blood vessels, and nerves are contained within fasciaProgressive pain distal to fracture, 5 Ps (pain, pressure, paralysis, paresthesia, pallor and pulselessness), pressure inside compartment >30mmHg (normal 0-8 mmHgFasciotomy (opening in the fascia)
Slide71Deep Vein Thrombosis
Slide72Deep Vein Thrombosis
Route of DVT embolus
Pulmonary embolism
Slide73Hip Fractures
Currently more than 250,000 hip fractures annually
Associated costs exceed $7 billion
Repair of a fractured hip is the most common procedure performed in people over 85.
30% of patients with a hip fracture die within 1
year r/t surgery (complications) & immobility
Osteoporosis is biggest risk factor
Slide74Classification of Hip Fractures
IntracapsularExtracapsularIntertrocantericSubtrocantericTranscervicalImpacted at base of the neck(Capsular refers to the proximal 1/3 of bone)
Slide75Signs of Symptoms of Hip Fracture
Medical emergency
Severe pain at the fracture site
Inability to move leg voluntarily
Classic
shortening
and external rotation of the leg
One-third of elderly individuals with hip fracture die within one year of injury
Typically the bone is rotated externally & shortening of the leg… try to get the leg back into
allignment
…
Slide76Medical Management –Hip Fracture
Conservative management
Prolonged immobility - 12-16 wks BR(avoids the risks associated with anesthesia)
Surgical Management
Reduction and stabilization of fracture with insertion of internal fixation device
Stable plate and screw (non-w/b 6 weeks to 3 months)
Telescoping nail (minimal to partial w/b 6 weeks3 months)
Prosthetic implant -replaces femoral head and neck
position restriction 2 weeks->2 months
partial weight bearing 2 months
Slide77Follow-up Instructions –Hip Fracture
DO NOT
flex more than 90
o
force hip into adduction or internal rotation
cross legs
put on own shoes and socks x 8 weeks
sit in chairs that do not have armrests
Slide78Follow-up Instructions –Hip Fracture
DO
Keep in extension and abduction
use toilet seat raiser
place/use shower chair
use pillow between legs x 8 weeks
notify of increased pain
inform dentist of prosthetic device
Slide795 Ps of neurovascular assessment
May be a test question
Pain
Paresthesia
(tingling, prickling or numbness of the skin)
Pallor
Pulses
paralysis
Slide80Hip Fracture
Slide81Hip Fracture
Severe pain at the fracture siteInability to move leg voluntarilyExternal Rotation
Slide82Total Hip Replacement
Preoperative care-consider psychosocial issues-family support-comfort-safety
Postoperative care-hip is kept in extension and abduction to prevent dislocation of the hip
Slide83Osteomyelitis
Acute or chronic infection of bone (confirmed by positive wound culture)
Usually staph
aureus
Direct or indirect
Pain, temperature, swelling, warmth, redness
Wound culture, bone scan, CT, MRI
Aggressive antibiotics 6-8
wks
(my pt at
Speciality
)
Nursing diagnoses: Pain, Impaired physical mobility, Ineffective therapeutic regimen, Risk for impaired skin integrity
Nursing care: Aseptic technique, no heat or exercise to affected area which will increase circulation
Slide84Osteomyelitis
Slide85Osteoporosis
A disorder in which bone mass is lost to the point where the skeleton is no longer able to withstand unexpected or normal mechanical forces. Most common skeletal disorder and second only to arthritis as a cause of musculoskeletal morbidity in the elderly. A/K/A porous bone
Bone resorption > bone formation
Slide86Osteoporosis
Slide87Osteoporosis Risk Factors
Aging
Gender (female)
Race (white)
Family History
Postmenopausal
(and not taking calcium supplement)
Chronic calcium deficiency
Sedentary
lifestyle (wt bearing exercise)
Small frame-low body weight
Slide88Osteoporosis – Related Risk Factors
Chronic smoking
Diet high in protein and fat (phosphorous depletes calcium – found in animal fat/protein)
Chronic alcohol use
Excessive caffeine intake (phosphorous depletes calcium)
Postmenopausal (estrogen helps body absorb calcium)
Glucocorticoids
(involved in protein and fat metabolism; Aluminum containing antacids (reduce amount of calcium in the body
)
Horonal
imbalances (estrogen and testosterone) are the primary causes of osteoporosis
Slide89Osteoporosis – Secondary Risk Factors
Endocrine disorders: Hyperthyroidism, hyperparathyroidism
GI Disorders
malabsorption syndrome, Hyperthyroidism, parathyroidism
COPD
glucocorticoids
Drug
Glucocorticoids
, heparin, anticonvulsants, loop diuretics, barbiturates
Slide90OsteoporosisClinical Manifestations
Back Pain- (
fx
of vertebra)
Chronic dull ache
Sudden onset of acute pain greatly intensified with coughing, sneezing or movement
Fracture
Proximal femur
Distal radius
Proximal
humerus
Ribs
Thoracic
kyphosis
(Dowager’s hump)
Loss of more than 2 in. standing height
Slide91Osteoporosis Collaborative Management
Diagnosis
Labs may not be helpful
X-rays (at 30-50% loss)
CT , MRI, Bone Scan
Bone Mineral Density Measurement
Medication
Estrogen Replacement: Prevention
Calcium Supplements
Calcitonin
: Nasal spray->irritating
Fosamax
,
Boniva
(1X/ month)
Actonel
: Before breakfast without food, sit upright X 30 min., full glass water
Vertebroplasty
,
Kyphoplasty
Slide92OsteoporosisNursing Management
Nursing diagnoses: Pain, Impaired physical mobility, Risk for injury, Imbalanced nutrition: less than body requirements
Promote calcium intake
Review diet to include:
Decrease caffeine
Excess calcium loss
Decrease protein and fat
High protein diet causes bone loss secondary to calcium loss
Promote exercise
active weight bearing, walking
Slide93Paget’s Disease
Excessive bone resorption followed by bone formation leading to weakened bone, bone pain, arthritis, deformity leading to pathologic fractures and osteogenic sarcoma
Bone marrow replaced by vascular fibrous connective tissue leading to formation of larger, disorganized, weaker bone
X-ray, increased alkaline phosphatase, positive bone scan
Medication (Fosamax, Actonel) with slowing of disease with early diagnosis
Slide94Paget’s Disease
Slide95Osteoarthritis (DJD)
Degenerative Joint Disease
Most common form of arthritis in the elderly
#1 cause of disability and limitation in those over 74
Non-inflammatory disease of moveable joints
Deterioration in articular cartilage and formation of new bone at the joint
Slide96Osteoarthritis (DJD)
Slide97Osteoarthritis
Slide98OsteoarthritisRisk Factors
Age
Obesity
Repetitive joint injuries
Genetics
Slide99Osteoarthritis -Cardinal Symptoms
Pain
After movement relieved by rest
Characterized as aching
Poorly localized
On motion with weight bearing
Slide100Osteoarthritis -Cardinal Symptoms
Stiffness
On awakening and in AM
After activity
Of relatively short duration
Slide101OsteoarthritisOther Signs and Symptoms
Crepitus
Limitation of motion
Weight-bearing joints weaken (asymmetrical)
Heberden nodes
Lateral enlargements of the distal phalangeal joints
Bouchard’s nodes (found at the proximal interphalangeal joints)
Slide102Bouchard Nodes
Slide103Heberdan Nodes
Slide104Osteoarthritis – Diagnosis
X-Ray
Decreased joint space, bony sclerosis, spur formation
Fluid analysis
Slide105Osteoarthritis - Treatment
Symptomatic relief and minimization of further joint destruction
Exercise
Rest
Weight-loss
NSAIDs
Gastric irritation, bleeding
Disturbance in platelet formation
Slide106Rheumatoid Arthritis
Chronic, systemic inflammatory disease of moveable joints
6 million Americans
Prevalence increases with age for both men and women
Slide107Rheumatoid Arthritis
Slide108Rheumatoid Arthritis
Slide109Rheumatoid Arthritis –Cardinal Symptoms
Unexplained periods of exacerbation and remission
Painful, swollen joints,
Bilateral, symmetrical
Morning stiffness
Lasting longer than 1 hour
In AM and after inactivity
Slide110Rheumatoid Arthritis –Cardinal Symptoms
Abnormal labs
Elevated ESR (
sed
rate)
Rheumatoid
factor
Anemia
WBC in synovial fluid
X-Ray
Bone degeneration
Slide111Rheumatoid Arthritis –Diagnosis
Based on cluster (4 for 6 weeks)
Morning stiffness
Arthritis in three or more joints
Arthritis of hand joints
Symmetrical
Rheumatoid nodules
Serum rheumatoid factor
Radiographic changes
Slide112Rheumatoid ArthritisTreatment Goals
Reduce inflammation
Alleviate pain
Preserve function
Prevent
deformity
(treatment is the same as OA, pretty much)
Both RA & OA cause severe loss of quality of life
Slide113Rheumatoid Arthritis –Management
Rest
PT
Aspirin
Corticosteroids
Gold salts
Disease modifying antirheumatic drugs (DMARDs)
Plaquenil, Azulfadine
Methotrexate
Slide114Rheumatoid Arthritis
Slide115Comparison of OA to RA
OA
Degenerative joint dzAffects articular cartilageNeed to add more
RA
Autoimmune joint
dx
“inflammatory arthritis”
Affects synovial membrane
Membrane thickens and immobilizes the joint
Slide116Gout: Gouty Arthritis
Affects middle aged to elderly men
Associated with renal stones r/t sodium
urate
crystal deposition
Treated with drugs that promote uric acid excretion (
allopurinol
and
probenicid
)
Treated with diet-limiting protein(organ meats, red meats, i.e., high
purine
food)
(Gout is associated with increased uric acid in the body;
Purines
increases uric acid levels in the body)
Unlike other forms of arthritis absolute rest of the joint is necessary. So painful it causes extreme pain just to touch the joint.
Primary form is inherited
(aspirin not indicated, can make the uric levels higher)
Slide117Mandible Fracture
Preoperative care-history of injury, pain control, gentle oral care, psycho-social
Postoperative care-pain control, observe for s/s infection, suction equipment at bedside, liquid diet, careful monitoring of airway, ready to cut wires if client vomits or has respiratory emergency(wirecutters at bedside)
Slide118Mandibular Fracture
Slide119Amputation
Preoperative care-nurse does history(how it occurred if trauma), concurrent illnesses, habits,
ie
. Smoking, current meds, psycho-social
Postoperative care-pain management including phantom pain, prevent infection, observe for adequate tissue perfusion
Nursing
dx
: prevention of infection, promotion of mobility, preparation for prosthesis…
Slide120Amputation
Accidental
Surgical
Slide121Bone Cancer
Multiple Myeloma
Osteogenic
sarcoma,Osteoclastoma
, Ewing’s Sarcoma,
Metatastic
Bone
Disease
Slide122Primary Bone Cancer
Multiple Myeloma (Plasma cell myeloma)
Invades sternum, ribs, spine, clavicles, pelvis, long bones.
Sx
: back pain, anemia, thrombocytopenia.
Osteogenic
sarcoma
Metaphyseal
area of long bones, esp. distal femur, proximal tibia, proximal
humerus
.
10-25 y/o males
Osteoclastoma
(Giant cell tumor)
Cancellous
bone: distal femur, proximal tibia, distal radius.
20-35 y/o.
Swelling local pain, bone destruction on
Xray
Ewing’s Sarcoma
3
rd
most common
Males under age 30
Medullary
cavity of long bone esp. femur, pelvis, tibia, ribs.
Mets to lung
Metastatic bone lesions
from breast, GI tract, lungs, prostate, kidney, ovary, and thyroid
Slide123Other Musculoskeletal Problems
Muscular Dystrophy
Low Back Pain-Laminectomy
Herniated Intervertebral Disk