Fat Cow Syndrome Dr. Vivek Kr. Singh Assistant
Description: Fat Cow Syndrome Dr. Vivek Kr. Singh Assistant Professor Department of Veterinary Clinical Complex UNIT-3 Synonyms FATTY LIVER IN CATTLE FAT-MOBILIZATION SYNDROME HEPATIC LIPIDOSIS PREGNANCY TOXEMIA IN CATTLE Introduction It is a disease of
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slide1. Fat Cow Syndrome Dr. Vivek Kr. Singh
Assistant Professor
Department of Veterinary Clinical Complex UNIT-3<br>
slide2. Synonyms FATTY LIVER IN CATTLE
FAT-MOBILIZATION SYNDROME
HEPATIC LIPIDOSIS
PREGNANCY TOXEMIA IN CATTLE<br>
slide3. Introduction It is a disease of high producing cows
Overfed cattle during dry period may develop fatty liver syndrome just before or after calving (Transition period)<br>
slide4. Etiology Mobilization of excessive body fat to liver during periods of negative energy balance at time of parturition or in early lactation of dairy cows and late pregnancy of beef cows<br>
slide5. Pathogenesis Increase energy demands + Reduced feed intake
Negative Energy Balance
Mobilization of fat from reserve
Fat is transported to liver for gluconeogenesis
Ruminant liver has limited capacity to transport VLDL
Hepatic Lipidosis<br>
slide6. Clinical findings Anorexia
Depression
Weakness
Persistent weight loss
Recombency
Ketosis (Do not respond to glucose treatment)
Some animals show nervous signs like star gazing and tremors of head and neck
Coma and death<br>
slide7. Necropsy Findings Gross enlargement of liver
Liver is pale yellow in color
Greasy and friable
Histopathology reveals
Enlarged hepatocytes with fatty changes<br>
slide8. Diagnosis Elevated NEFA
Ketone bodies
Eleveted LDH
Tatal bilirubin
AST
GGT
The liver sample which floats in water and copper sulphate solution having sp. Gravity ranging between 1.025 – 1.055 indicates presence of 34% lipid content in the sample<br>
slide9. Treatment Continuous supplementation of glucose along with calcium and magnesium salt to correct negative energy balance
Long acting protamine zinc insuline @ 200 units/ cow every 12 hrly
S/c administration of choline chloride (25 g in 250 ml NSS)
Niacin@ 6-12g/cow/day helps in reducing hepatic lipidosis
Antioxidants
Anabolic steroids @300mg/cow<br>
slide10. Thank You<br>
Assistant Professor
Department of Veterinary Clinical Complex UNIT-3<br>
slide2. Synonyms FATTY LIVER IN CATTLE
FAT-MOBILIZATION SYNDROME
HEPATIC LIPIDOSIS
PREGNANCY TOXEMIA IN CATTLE<br>
slide3. Introduction It is a disease of high producing cows
Overfed cattle during dry period may develop fatty liver syndrome just before or after calving (Transition period)<br>
slide4. Etiology Mobilization of excessive body fat to liver during periods of negative energy balance at time of parturition or in early lactation of dairy cows and late pregnancy of beef cows<br>
slide5. Pathogenesis Increase energy demands + Reduced feed intake
Negative Energy Balance
Mobilization of fat from reserve
Fat is transported to liver for gluconeogenesis
Ruminant liver has limited capacity to transport VLDL
Hepatic Lipidosis<br>
slide6. Clinical findings Anorexia
Depression
Weakness
Persistent weight loss
Recombency
Ketosis (Do not respond to glucose treatment)
Some animals show nervous signs like star gazing and tremors of head and neck
Coma and death<br>
slide7. Necropsy Findings Gross enlargement of liver
Liver is pale yellow in color
Greasy and friable
Histopathology reveals
Enlarged hepatocytes with fatty changes<br>
slide8. Diagnosis Elevated NEFA
Ketone bodies
Eleveted LDH
Tatal bilirubin
AST
GGT
The liver sample which floats in water and copper sulphate solution having sp. Gravity ranging between 1.025 – 1.055 indicates presence of 34% lipid content in the sample<br>
slide9. Treatment Continuous supplementation of glucose along with calcium and magnesium salt to correct negative energy balance
Long acting protamine zinc insuline @ 200 units/ cow every 12 hrly
S/c administration of choline chloride (25 g in 250 ml NSS)
Niacin@ 6-12g/cow/day helps in reducing hepatic lipidosis
Antioxidants
Anabolic steroids @300mg/cow<br>
slide10. Thank You<br>