S Joshi Associate Prof Dept Of Anesthesia LEARNING OBJECTIVES At the end of the lecture the student shall be able to Describe anatomy of spinal cord Enumerate physiological effects of spinal anaesthesia on various systems ID: 775199
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Slide1
SPINAL ANAESTHESIA
Dr.
Pradnya
S. Joshi
Associate Prof.
Dept.
Of Anesthesia
Slide2LEARNING OBJECTIVES
At the end of the lecture, the student shall be able to
Describe anatomy of spinal cord
Enumerate physiological effects of spinal anaesthesia on various systems
Enumerate indications & contraindications of spinal anaesthesia
Enumerate advantages & disadvantages of spinal anaesthesia
Enumerate complications of S.A.
Slide3ANATOMY OF SPINE
Spine : composed vertebral bones &
fibro cartilaginous discs
.
There are : 7 cervical
: 12 thoracic
: 5 lumbar
: sacrum which is a fusion of 5
sacral vertebrae
: Coccyx 4-5
Slide4VERTEBRAL COLUMN
Four curves:Cervcial &lumbar convex anteriorly (lordosis).Thoracic & sacral concave anteriorly (kyphosis)Spinal nerves:8 cervical 12 thoracic5 lumbar5 sacral1coccygeal.Total 31
Slide5SPINAL CORD
Spinal cord continues
cephalad
with brain stem through foramen magnum and terminates distally in the
conus
medullaris
at
lower border of L1 in adults and upper border of L3 in children
.
Slide6SPINAL CORD
Meningeal
coverings
:
From with in to periphery
Pia
mater
Arachanoid
membrane
Dura mater
Slide7Slide8SUBARACHANOID SPACE
Lies between
pia
mater and
arachnoid
mater, ends at S2
Contents
:
CSF
Spinal nerves
Blood vessels supplying spinal cord
Slide9Slide10CIRCULATION OF CSF
Choroid plexus in lateral ventricles Foramen of Monroe 3rd ventricle Aqueduct of Sylvius 4th ventricle Foramen of Luschke and Magendie Subarachanoid space.Re absorption of CSF occurs in arachanoid villi.
Slide11August Bier 1885
Slide12AUTONOMIC NERVOUS SYSTEM
Has 2 components:
Sympathetic fibers
Arises from T1 - L2 segment.
Parasympathetic fibers
From craniosacral segment
Cranial nerves giving rise to parasympathetic fibers: 3,7,9,10
Sacral fibers arises from S2,S3,S4 spinal segment.
Slide13S.A. results in predominantly sympathetic blockade ,because parasympathetic fibers carried by vagus have higher origin
Slide14MECHANISM OF ACTION
Site of action for neuraxial blockade in sub arachnoid space: nerve roots
Blockade of posterior nerve roots interrupts somatic and visceral sensation.
Blockade of anterior nerve root fibers prevents efferent motor and autonomic outflow.
Slide15ORDER OF BLOCKING NERVE FIBRES
Autonomic fibers
Temperature fibers
Pain fibers
Touch fibers
Somatic motor fibers
Slide16T4---Nipple
T5---
Inframammary
fold
T6---
Xiphoid
T10---Umbilicus
L1---Groin
S2-4---Perineum.
Slide17CVS CHANGES
HYPOTENSION
Occurs because of venous and arterial dilatation
BRADYCARDIA
Blockade of cardiac sympathetic fibers ->
decrease in H.R.
EFFECTS ON GIT
Nausea and vomiting
caused by unopposed parasympathetic (vagal) activity
This also results in small contracted gut which provide excellent operative conditions.
Slide19URINARY TRACT MANIFESTATIONS
Blockade of both sympathetic and parasympathetic control of bladder function results in urinary retention.
Slide20ADVANTAGES
Low cost
Decrease blood loss
Less metabolic derangement
Better for severe respiratory impairment
Allows verbal communication
Postoperative Analgesia
Avoid complications of G.A.
Slide21DISADVANTAGES
Discomfort in prolonged surgeries
Potential of nerve damage
Introduction of infection in CSF
Post operative leg weakness & urinary
retention
Slide22INDICATIONS
Primary anesthetic technique in lower abdominal procedures(LSCS), inguinal hernia repair,
urogenital
, rectal and lower extremity surgeries.
S.A. preferable in
geriartic
age group for peripheral
orthopaedic
,vascular and urological procedures.
Obstetric patients with full stomach
Slide23ABSOLUTE CONTRAINDICATIONS
Patient refusal
Infection at injection site
Coagulopathy
or bleeding diathesis
Low fixed cardiac output states:
Severe constrictive
pericarditis
, cardiac
tamponade
, severe M.S., severe A.S.
Severe
hypovolemia
Raised ICT
Slide24RELATIVE CONTRAINDICATIONS
Sepsis
Uncooperative patient
Spinal deformity:
congenital, traumatic, post
laminectomy
Severe anemia
Slide25TECHNIQUE OF S.A.
Equipment
Spinal needle - small needles reduce the incidence of post
dural
puncture headache.
Drugs
Lidocaine
(5%)
Bupivacaine(0.5%)
Ropivacaine(0.5%) available in isobaric form only
Slide26Spinal Needle
Slide27Sitting Position
Slide28LATERAL POSITION
SITING POSITION
Slide29Holding for Spinal
Slide30Slide31INTRAOPERATIVE COMPLICATIONS
Due to exaggerated physiological responses
Hypotension
Management:-
Maintain O2 supply
IV fluids bolus 0.5-1L
RAISE LEGS
Use of
vasopressor
drugs like Ephedrine or
Mephenteramine
Slide32Bradycardia
When PR. < 60/Min
P.R.< 50 - Inj. Atropine 0.6mg
Inj. Epinephrine 50-100µg
Slide33TOTAL SPINAL BLOCK:
Level of block extend above T1 involving cervical segments
Nausea, vomiting, difficulty in breathing, hypotension,
bradycardia
.
Management
:
Maintain adequate airway & ventilation with 100%O2 supply.
Immediately do intubation
Maintain circulation with iv fluids.
Vasopressor
drugs :
Inj.
Phenylephrine
, Dopamine .
Slide34Urinary retention
Nausea &vomiting
Slide35POSTOPERATIVE COMPLICATIONS
Postdural
puncture headache
occurs 12-72 hrs post operatively.
Cause -
Loss of CSF through
dural
puncture -> decreased ICP -> traction on
dura
& blood vessels
Bilateral frontal or retro orbital or occipital headache.
Increases in sitting & relieves in lying down
Slide36Management
Prevention
:
use smaller size needle
Prevention of dehydration
Treatment
:
Recumbent position, analgesics
I.V or oral fluid administration
I.V. caffeine
Epidural blood patch.
Slide37Delayed complications
Transient neurological symptoms
Cranial nerve disturbances
Visual and auditory disturbances
Paralysis of 6
th
cranial nerve
Backache
Spinal
haematoma
Meningitis or
arachnoiditis
Slide38Slide39