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Haemorrhage M Kamil Department of Surgery Haemorrhage M Kamil Department of Surgery

Haemorrhage M Kamil Department of Surgery - PowerPoint Presentation

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Haemorrhage M Kamil Department of Surgery - PPT Presentation

20182019 Haemorrhage hypovolaemic shock T issue trauma hypovolaemic shock Acute T raumatic Coagulopathy ATC ATC TraumaInduced C oagulopathy TIC ID: 934698

control haemorrhage coagulopathy blood haemorrhage control blood coagulopathy surgical hypothermia resuscitation acidosis pressure surgery concealed direct damage bleeding due

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Presentation Transcript

Slide1

Haemorrhage

M Kamil

Department of Surgery

2018-2019

Slide2

Haemorrhage →

hypovolaemic shock

Tissue trauma + hypovolaemic shock → Acute Traumatic Coagulopathy (ATC)ATC → Trauma-Induced Coagulopathy (TIC) This is multifactorial:1. Ongoing bleeding with fluid and red blood cell resuscitation → dilution of coagulation factors which worsens the Coagulopathy2. Hypoperfusion → hypothermia → Coagulopathy and further haemorrhage

Haemorrhage

Slide3

3. Hypoperfusion

Under perfused muscle → Acidosis + Hypothermia → poor coagulation functions →further haemorrhage → hypoperfusion and worsening Acidosis and Hypothermia. 4. Cold Intravenous blood and fluids → Hypothermia. 5. Surgery → opened body cavity → further Hypothermia 6. Surgery → further bleeding → resuscitation with acidic crystalloid fluids (e.g. normal saline has a pH of 6.7) →

Acidosis

Haemorrhage

Slide4

Coagulopathy

+

Acidosis (pH < 7.2) + Hypothermia (< 35o C)

Slide5

Slide6

Grossly:Revealed haemorrhage

Concealed haemorrhage

Classification:

Slide7

Revealed haemorrhage

Slide8

Concealed Haemorrhage

Slide9

Concealed Haemorrhage

Slide10

According to time frame (time of occurrence)

Primary

haemorrhage occurring immediately due to an injury (or surgery)Reactionary delayed haemorrhage (within 24 hours) and is usually due to dislodgement of clot by resuscitation, normalisation of blood pressure and vasodilatation. Reactionary haemorrhage may also be due to technical failure, such as slippage of a ligatureSecondarydue to sloughing of the wall of a vessel. It usually occurs 7–14 days after injury and is precipitated by factors such as infection, pressure necrosis (such as from a drain) or malignancyClassification:

Slide11

Surgical Haemorrhagedue to a direct injury and is

amenable to

surgical control (or other techniques such as angioembolisation)Non-surgical Haemorrhagethe general ooze from all raw surfaces due to coagulopathy and cannot be stopped by surgical means (except packing). Treatment requires correction of the coagulation abnormalitiesClassification:

Slide12

Blood volume normally is around 5 liters

Adults and children = 70ml/kg; Neonates = 80ml/kg

Estimation of the amount of blood that has been lost is difficult, inaccurate and usually underestimates the actual valueIn the theater; blood collected in suckers is counted. Swabs socked in blood is weighedBlood collected in drains can be countedBUT concealed haemorrhage is difficult to estimateHaemoglobin level is a poor indicatorVital signs; CVP; pH; Base Deficit and Dynamic Response to Fluid Therapy Estimation of the amount of blood lost

Slide13

Slide14

Management

Immediate resuscitative measures:

Direct pressure should be placed over the site of external haemorrhageAirway and breathing should be assessed and controlled as necessary Large-bore intravenous cannulablood drawn for typing and cross-matching. Emergency blood should be requested if the degree of shock and ongoing haemorrhage warrants this

Slide15

Identification of the site of concealed haemorrhage

:

Aim: to define the next step in haemorrhage control (operation, angioembolisation, endoscopic control)Clues:History: (previous episodes, known aneurysm, non-steroidal therapy for gastrointestinal (GI) bleeding) Clinical examination: (nature of blood – fresh, melaena; abdominal tenderness, etc.). For shocked trauma patients, the external signs of injury may suggest internal haemorrhage, but haemorrhage into a body cavity (thorax, abdomen) must be excluded with rapid investigations (chest and pelvis x-ray, abdominal ultrasound or diagnostic peritoneal aspiration).Management

Slide16

Control of haemorrhage:

Must be rapid to prevent triad of

Coagulopathy – Acidosis – HypothermiaUn-necessary actions and investigations is omittedProlonged volume resuscitation with crystalloid is not beneficialBlood components to correct coagulopathy is desirableResorting to “Damage Control Surgery” (DCS), part of strategy of “Damage Control Resuscitation” (DCR)Once haemorrhage and sepsis controlled; aggressive resuscitation; rewarming and correction of coagulopathy should followManagement

Slide17

What is meant by Damage Control Resuscitation?

A recent strategy with FOUR main components:

First: anticipate and treat Acute Traumatic Coagulopathy (ATC) to prevent trauma-induced Coagulopathy (TIC)Second: Permissive Hypotension therapy until haemorrhage controlledThird: limitation of Crystalloid and Colloid infusion to avoid Dilutional coagulopathyFourth: Damage Control Surgery (DCS) which entails:Arresting haemorrhageControl of sepsisProtection from further injuryNothing else Management

Slide18

Haemorrhage control- direct pressure

Slide19

Haemorrhage control- direct pressure

Slide20

Haemorrhage control- surgical clamps

Slide21

Haemorrhage control- surgical clamps

Slide22

Haemorrhage control- packing

Slide23

Haemorrhage control- packing

Slide24

Haemorrhage control- balloon inflation

Slide25

Haemorrhage control-

balloon inflation

Slide26

Haemorrhage control- haemostatics

Slide27

Haemorrhage control- bone wax

Slide28

Haemorrhage control- electrocautery

Slide29

Haemorrhage control- photocoagulation

Slide30

Bleeding control: Ligature and clips

Slide31

Slide32

Slide33

That is the end

Thanks for listening