20182019 Haemorrhage hypovolaemic shock T issue trauma hypovolaemic shock Acute T raumatic Coagulopathy ATC ATC TraumaInduced C oagulopathy TIC ID: 934698
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Slide1
Haemorrhage
M Kamil
Department of Surgery
2018-2019
Slide2Haemorrhage →
hypovolaemic shock
Tissue trauma + hypovolaemic shock → Acute Traumatic Coagulopathy (ATC)ATC → Trauma-Induced Coagulopathy (TIC) This is multifactorial:1. Ongoing bleeding with fluid and red blood cell resuscitation → dilution of coagulation factors which worsens the Coagulopathy2. Hypoperfusion → hypothermia → Coagulopathy and further haemorrhage
Haemorrhage
Slide33. Hypoperfusion
→
Under perfused muscle → Acidosis + Hypothermia → poor coagulation functions →further haemorrhage → hypoperfusion and worsening Acidosis and Hypothermia. 4. Cold Intravenous blood and fluids → Hypothermia. 5. Surgery → opened body cavity → further Hypothermia 6. Surgery → further bleeding → resuscitation with acidic crystalloid fluids (e.g. normal saline has a pH of 6.7) →
Acidosis
Haemorrhage
Slide4Coagulopathy
+
Acidosis (pH < 7.2) + Hypothermia (< 35o C)
Slide5Slide6Grossly:Revealed haemorrhage
Concealed haemorrhage
Classification:
Slide7Revealed haemorrhage
Slide8Concealed Haemorrhage
Slide9Concealed Haemorrhage
Slide10According to time frame (time of occurrence)
Primary
haemorrhage occurring immediately due to an injury (or surgery)Reactionary delayed haemorrhage (within 24 hours) and is usually due to dislodgement of clot by resuscitation, normalisation of blood pressure and vasodilatation. Reactionary haemorrhage may also be due to technical failure, such as slippage of a ligatureSecondarydue to sloughing of the wall of a vessel. It usually occurs 7–14 days after injury and is precipitated by factors such as infection, pressure necrosis (such as from a drain) or malignancyClassification:
Slide11Surgical Haemorrhagedue to a direct injury and is
amenable to
surgical control (or other techniques such as angioembolisation)Non-surgical Haemorrhagethe general ooze from all raw surfaces due to coagulopathy and cannot be stopped by surgical means (except packing). Treatment requires correction of the coagulation abnormalitiesClassification:
Slide12Blood volume normally is around 5 liters
Adults and children = 70ml/kg; Neonates = 80ml/kg
Estimation of the amount of blood that has been lost is difficult, inaccurate and usually underestimates the actual valueIn the theater; blood collected in suckers is counted. Swabs socked in blood is weighedBlood collected in drains can be countedBUT concealed haemorrhage is difficult to estimateHaemoglobin level is a poor indicatorVital signs; CVP; pH; Base Deficit and Dynamic Response to Fluid Therapy Estimation of the amount of blood lost
Slide13Slide14Management
Immediate resuscitative measures:
Direct pressure should be placed over the site of external haemorrhageAirway and breathing should be assessed and controlled as necessary Large-bore intravenous cannulablood drawn for typing and cross-matching. Emergency blood should be requested if the degree of shock and ongoing haemorrhage warrants this
Slide15Identification of the site of concealed haemorrhage
:
Aim: to define the next step in haemorrhage control (operation, angioembolisation, endoscopic control)Clues:History: (previous episodes, known aneurysm, non-steroidal therapy for gastrointestinal (GI) bleeding) Clinical examination: (nature of blood – fresh, melaena; abdominal tenderness, etc.). For shocked trauma patients, the external signs of injury may suggest internal haemorrhage, but haemorrhage into a body cavity (thorax, abdomen) must be excluded with rapid investigations (chest and pelvis x-ray, abdominal ultrasound or diagnostic peritoneal aspiration).Management
Slide16Control of haemorrhage:
Must be rapid to prevent triad of
Coagulopathy – Acidosis – HypothermiaUn-necessary actions and investigations is omittedProlonged volume resuscitation with crystalloid is not beneficialBlood components to correct coagulopathy is desirableResorting to “Damage Control Surgery” (DCS), part of strategy of “Damage Control Resuscitation” (DCR)Once haemorrhage and sepsis controlled; aggressive resuscitation; rewarming and correction of coagulopathy should followManagement
Slide17What is meant by Damage Control Resuscitation?
A recent strategy with FOUR main components:
First: anticipate and treat Acute Traumatic Coagulopathy (ATC) to prevent trauma-induced Coagulopathy (TIC)Second: Permissive Hypotension therapy until haemorrhage controlledThird: limitation of Crystalloid and Colloid infusion to avoid Dilutional coagulopathyFourth: Damage Control Surgery (DCS) which entails:Arresting haemorrhageControl of sepsisProtection from further injuryNothing else Management
Slide18Haemorrhage control- direct pressure
Slide19Haemorrhage control- direct pressure
Slide20Haemorrhage control- surgical clamps
Slide21Haemorrhage control- surgical clamps
Slide22Haemorrhage control- packing
Slide23Haemorrhage control- packing
Slide24Haemorrhage control- balloon inflation
Slide25Haemorrhage control-
balloon inflation
Slide26Haemorrhage control- haemostatics
Slide27Haemorrhage control- bone wax
Slide28Haemorrhage control- electrocautery
Slide29Haemorrhage control- photocoagulation
Slide30Bleeding control: Ligature and clips
Slide31Slide32Slide33That is the end
Thanks for listening