PPT-Hemodynamic Disorders, Thromboembolic Disease, and Shock

Practical session DrMohanad Total extracellular tissue fluid volume hydrostatic pressure colloid osmotic pressure lymphatic drainage Mechanism of edema formation in heart renal and liver failure. Healthy red blood cells are round and they move through small blood vessels carrying oxygen to all parts of the body In SCD the red blood cells become hard and sticky and look like a Cshaped farm tool called a sickle Sickle cells die early which cau Grant M. Greenberg MD, MA, MHSA. Overview. Identify the challenges in diagnosis of Venous Thromboembolic Disease.  . Diagram current protocols/pathways for . evaluation . and treatment of VTE. Review current anticoagulation . Matthew Velkey. matt.velkey@duke.edu. . 454A Davison, Duke South, Green Zone. Fluid Homeostasis. Total extracellular tissue fluid volume . = hydrostatic pressure . – [. colloid osmotic pressure . ulmonary Hypertension. Dr Dolores Taboada MD. Consultant Cardiologist. Pulmonary Vascular Disease Unit. Papworth Hospital, Cambridge, UK. My disclosures. I have received honoraria . from . Actelion. , Bayer, . Shock may be defined as a condition in which circulation fails to meet the nutritional needs of the cells and at the same time. A final common pathway for many potentially lethal clinical events (hemorrhage,trauma, burns, large MI, massive pulmonary embolism microbial sepsis) fails to remove the metabolic waste products. Polydactyly. Down Syndrome. Hemophelia. Progeria. Hypertrichosis. Sickle Cell Anaemia. INTRODUCTION. What is genetic diseases/disorders?. The human body is composed up of cells , each one specializing a particular function like sensing light, smelling . Alžbeta. . Kráľová. . Trančíková. Department of Pathophysiology. alzbeta.trancikova@jfmed.uniba.sk. Ingestion of food. Digestion. mechanical digestion of food particles. breaks up food particles. Shock. Decreased tissue perfusion . . inadequate O2 delivery . . . tiessue. ischemia. Common Clinical Features of Shock. 1. HYPOTENSION. SBP < 90 mm Hg. MAP <60 mm Hg. Acute decreased in SBP of > 40 mm Hg. A genetic disorder that is caused by abnormality in an individual's DNA. Abnormalities can range from small mutation in a single gene to the addition or subtraction of a whole chromosome or set of chromosomes.. Fig. 1. and 2. Widespread purpuric and ecchymotic rash covering all four limbs. 2. Discussion 2. 1. Pathophysiology and risk factors Meningococcal disease (MD) caused by the gram negative diplococc DR. SUVANKAR GHOSH. BANKERS HEART INSTITUTE AND RESEARCH CENTRE. Physiology . Myocardial Oxygen Supply and Demand. a,b. Goals of Hemodynamic Support During High-Risk PCI. Types of Hemodynamic Support in the . George W. Vetrovec, MD, MACC, MSCAI. Professor Emeritus, VCU Pauley Heart Center. Virginia Commonwealth University. Richmond, Virginia . USA. Disclosures:. Consultant: Abiomed, Merck, FDA. What Hemodynamic Support is Available?. Inborn Errors of Metabolism . Inborn Errors of Metabolism(IEM) . Disorders in which a gene defect causes a clinically significant block in a metabolic pathway(enzyme deficiency . or transport protein . An embolus: is a detached intravascular solid, liquid, or gaseous mass that is carried by the blood to a site distant from its point of origin. . . Embolism: - . This is the passage and eventual trapping within the vasculature of any of a wide variety of mass objects..