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ACUTE AND CHRONIC TONSILITIS ACUTE AND CHRONIC TONSILITIS

ACUTE AND CHRONIC TONSILITIS - PowerPoint Presentation

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ACUTE AND CHRONIC TONSILITIS - PPT Presentation

Dr kcsudeep ANATOMY Palatine tonsils are two in number Situated on lateral aspect of oropharynx between the anterior and posterior pillars Actual size of the tonsil is bigger than the one that appears from its surface as parts tonsil extend upwards into the soft palate downwards i ID: 614662

tonsillitis acute tonsil tonsils acute tonsillitis tonsils tonsil surface chronic crypts abscess enlarged artery infection treatment soft tonsillar palate 000 complications membrane

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Slide1

ACUTE AND CHRONIC TONSILITIS

Dr. kcsudeepSlide2

ANATOMY:

Palatine tonsils are two in number.Situated on lateral aspect of oropharynx between the anterior and posterior pillars.

Actual size of the tonsil is bigger than the one that appears from its surface ,as parts tonsil extend upwards into the soft palate, downwards into the base of tongue and

anterirly

into

palatoglossal

arch.

Has got medial , lateral surface and upper and lower pole.Slide3

Medial surface is covered by non

keratinising stratified squamous epithelium which dips into the substance of tonsil in the form of crypts.Lateral surface is covered by well defined fibrous capsule. In between bed of tonsils and fibrous capsule there is loose areolar tissue site for collection of pus in

peritonsillar

abscess.

Upper pole of tonsil extends into soft palate and presence of

supratonsillar

fossa

.

Lower pole attached to tongue .

Tonsillolingual

sulcus

may be the seat of carcinoma

Bed of the tonsil: it is formed by superior constrictor and

styloglossus

muscles.The

glossopharyngeal

nerve and

styloid

process, if

enlarged,may

lie in relation to the lower part of

tonsillar

fossa

.Slide4
Slide5
Slide6

BLOOD SUPPLY:

1)

Tonsilar

branch of facial artery(main artery)

2) Ascending pharyngeal artery from external carotid.

3) Ascending palatine , a branch of facial artery.

4)Dorsal

linguae

branches of lingual artery.

5) Descending palatine branch of maxillary artery.Slide7
Slide8

VENOUS DRAINAGE: veins from the tonsils drain into paratonsillar

vein which joins the common facial vein and pharyngeal venous plexus.Slide9

LYMPHATIC DRAINAGE:

Lymphatics jugulodigastric (tonsillar) node situated below the angle of mandible.Nerve supply:

Lesser palatine branches of

sphenopalatine

ganglion (CNV) and

glossopharyngeal

nerve provide sensory nerve supply.Slide10

ACUTE TONSILLITIS

Primarily , tonsil consist ofa) surface epithelium continous with oropharyngeal lining.

b) crypts which are tube like invaginations from surface epithelium.

c) lymphoid tissues.Slide11

1) Acute catarrhal or superficial tonsillitis:

here tonsillitis is a part of generalised pharyngitis and mostly seen in viral infection.

2) Acute follicular tonsillitis:

infection spreads into crypts which become filled with purulent material, presenting at the openings of crypts as yellowish spots.Slide12
Slide13
Slide14

3) Acute

parenchymatous tonsillitis: Tonsil substance is affected. Tonsil is uniformly enlarged and red.

4)Acute membranous tonsillitis:

It is a stage ahead of acute follicular tonsillitis when exudation from the crypts coalesces to form a membrane on the surface of tonsil.Slide15
Slide16

AETIOLOGY

Mostly affects school-going children, but also affect adults.

Haemolytic

streptococcus most common.

Other causes: staphylococci,

pneumococci

, H. influenza.Slide17

SYMPTOMS:

Sore throat.Difficulty in swallowing.Fever.Earache.Constitutional symptoms  headache, malaise, abdominal pain .Slide18
Slide19

SIGNS:

Breath is foetid and tongue is coated.Hyperaemia of pillars, soft palate and uvula.Tonsils are red and swollen with yellowish spots of purulent material(acute follicular tonsillitis) or

whitis

membrane on medial surface of tonsils(acute membranous tonsillitis).

Tonsils may be enlarged or congested so much they almost meet in midline with edema of uvula and soft palate.(acute

parenchymatous

tonsillitis.)

Jugulodigastric

lymphnodes

are enlarged and tender.Slide20
Slide21

Laboratory results:

 blood work indicative of inflammation, an increase in white blood cell count, gradual decrease.A general examination is necessary, including examination of heart and circulation and urine analysis.Slide22

TREATMENT:

Bed rest and plenty of fluids.Analgesics Antimicrobial therapy.COMPLICATIONS:Chronic tonsillitis with recurrent acute attacks.

Peritonsillar

abscess.

Parapharyngeal

abscess, cervical abscess.

Rheumatic fever, acute

glomerulonephritis

, acute

otitis

media.Slide23

FAUCIAL DIPTHERIA

Aetiology : gram positive bacillus, corynebacterium diptheriae.

Spreads by droplet infection.

Incubation period 2-6 days.

CLINICAL FEATURES:

oropharynx is commonly involved and larynx nasal cavity may also be affected.

In oropahryx , a greyish white membrane forms over tonsils .it is tenacious and causes bleeding when removed . cervical lymphnode enlarged giving ‘bull- neck” appearance.

Patient is ill and toxemic.Slide24

COMPLICATIONS:

Exotoxin produced by this bacilli is toxic to heart and nerves. It cause myocarditis, cardiac arrhythmias and acute circulatory failure.Neurological complications: paralysis of soft palate, diaphragm and ocular muscles.In larynx, diptheritic membrane may cause airway obstruction.Slide25

Treatment:

Aim is to neutralise free toxins and killing this bacilli.Check for hypersensitivity.Diptheria of <48 hrs

20,000 to 40,000 units.

Diptheria

of >48hrs and confined to tonsils80,000 to 120,000 units.

Antibiotics used are benzyl penicillin 600mg 6

hrly

for 7 days.

What antibiotics if hypersensitive to

benzyl penicillin?Slide26

CHRONIC TONSILLITIS:

Aetiology :May be complication of acute tonsillitis.Subclinical infections of tonsils without acute attack.Mostly affect child and young adults.

Chronic infection in sinuses or teeth may be predisposing factor.Slide27

TYPES:

Chronic follicular tonsillitis: tonsillar crypts are full of infected cheesy material which shows on surface as yellowish spots.Chronic

parenchymatous

tonsillitis: hyperplasia of lymphoid tissue . tonsils are very much enlarged and may interfere with speech, deglutition and respiration.

Chronic fibroid tonsillitis: tonsils are small but infected, with history of repeated sore throats.Slide28
Slide29

CLINICAL FEATURES:

Recurrent attack of sore throat or acute tonsillitis.

Chronic irritation in throat with cough.

Bad taste in mouth and halitosis due to pus in crypts.

Difficulty in swallowing and choking at night.

EXAMINATION:

Tonsils may show varying degree of enlargement.

Yellowish beads of pus on medial surface of tonsil

Flushing of anterior pillars compared to rest of pharyngeal mucosa.

Enlargement of lymphnodes.Slide30

TREATMENT:

Conservative treatment: attention to general health, diet, treatment of co-existent infection of teeth, nose and sinuses.Tonsillectomy is indicated when it interfere with speech, deglutition and respiration or recurrent attack.COMPLICATIONS:

Peritonsillar

abscess.

Parapharyngeal

abscess, intra

tonsillar

abscess.

Tonsilloliths

.

Tonsillar

cyst.