PPT-Viruses and Cancer

Folder Title CxVirus NoTP Updated April 12 2017 See Chapter 3 Weinberg 2 nd Edition pages 71 to 102 Importance of Virology to Cancer Biology and Cancer Medicine Need to Control Potentially Infectious Exposure. Chapters 12 and 13. Viral Characteristics and Structure. Why Viruses aren’t Alive. General Characteristics of Viruses. Name derives from the Latin for “poison”. Obligatory intracellular parasites. Parasitic Worms. Kingdom . Animalia. : . eukaryotic. , no cell walls, heterotrophic nutrition, specialized tissues. A. Platyhelminthes. Flatworms. e.g. planarians . Platyhelminthes. Trematoda. = flukes . HIV infected T-cell. Viral Structure. not . cells. small . infectious particles . w/. DNA or RNA enclosed . in a protein coat . (capsid). in . some cases. , a membranous envelope. Viral Genomes. genomes . Days . W. ork . in Biotech. Steven . Pincus. , AVP Virology and Analytical Development. Cari. . Sadowski. Associate Principle Scientist Group Leader Virology. Health . Science Teachers Conference . EQ: Why are viruses considered to be non-
living?. Characteristics:. *Nonliving particles made of . nucleic acid. and a . protein coat . enlosed in a . capsid. shell... *In order to reproduce, all viruses must have a . 8.1 What Is a Virus? Plus 9.1, 9.2 (parts). 8.2 Structure of the Virion. 8.3 Overview of the Virus Life Cycle. 8.8 . Temperate Bacteriophages and . Lysogeny. 8.10, 9.6, 9.7 Overview of Animal Viruses. . . The process of transfer of virus into host is referred as . TRANSMISSION. and this is an important step in the life cycle of viruses.. Person to person infections are said to be . HORIZONTAL. . Carcinogenesis. Carcinogenesis or . Oncogenesis. =. creation of cancer= normal cells are transformed into cancer cells= characterized by . a progression of changes on cellular and genetic level . that ultimately reprogram a cell to undergo uncontrolled cell division, thus forming a malignant mass.. Table showing the Differences between Hepatitis A, B, C, D and E.  . Hepatitis A. Hepatitis B. Hepatitis C. Hepatitis D. Hepatitis E. Aetiology (cause). Hepatitis A Virus. Hepatitis B Virus. Hepatitis C Virus. Animalia. : eukaryotic, no cell walls, heterotrophic nutrition, specialized tissues. A. Platyhelminthes. Flatworms. e.g. planarians . Platyhelminthes. Trematoda. = flukes . leaf-shaped. parasitic. complex life cycles with several larval forms. As a group discuss for 1 minute. Be able to defend your answer for the class!. Are . VIRUSES. alive?. Growth and Development. Reproduce. Change Over Time. Obtain and Use Energy. Maintain Homeostasis. AL-. Fahadawi. Oncogenic . viruses. HPV. EBV. CMV. Oncogenesis. . -An abnormal . growth of . tissue resulting . from . uncontrolled, progressive multiplication . of . cells . and serving no . physiological function. . :. o. 1. • Self-sufficiency in growth signals. • Insensitivity to growth inhibitory signals. • Evasion of cell death. • Limitless replicative potential. • Development of sustained angiogenesis. basics. Dr. Baha Eldin Khalid Elamin. Viruses enter the body of the host. in a variety of ways, for example... . .  .                                             .