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SEPSIS SEPSIS

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SEPSIS - PPT Presentation

EM Student Lecture Series CASE STUDY A 53year old woman presents complaining of several days of fever generalized malaise nausea amp vomiting She has a PMH of diabetes and HTN and takes Glucophage ID: 434308

amp sepsis septic egdt sepsis amp egdt septic mortality abcs lactate mild patient ventilation measure fluid mmhg shock admit oxygenation organ source

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Slide1

SEPSIS

EM Student Lecture SeriesSlide2

CASE STUDY

A 53-year old woman presents complaining of several days of fever, generalized malaise, nausea & vomiting. She has a PMH of diabetes and HTN and takes

Glucophage

and

Lisinopril

.

Initial VS: 105/54 110 24 100.4 O2 sat 96%

PE: significant for mild lethargy (but she is A&Ox4); mild diffuse abdominal tenderness to palpation – otherwise WNLSlide3

WHAT NOW?

List 5 initial steps in the management of this patient

List 5 differential diagnoses

List 5 tests or interventionsSlide4

SEPSIS

A continuum … from

SIRS

Sepsis

Severe Sepsis

Septic Shock

DEATHSlide5

SIRS

Requires

2 out of 4

of the following:

Temp >38.0 or <36.0

HR>90 RR>24 or PaCO2<32

WBC<4000 or >12000 OR bands >10%Slide6

SEPSIS

Systemic response to host infection

SIRS + A SOURCE

Encompasses body’s own response to pathogen – characterized by derangements in inflammation, coagulation &

fibrinolysis

May progress to abnormal

vasodilation

, tissue

hypoperfusion

, microcirculation thrombosis … to ORGAN DYSFUNCTION

Increased risk in ...Slide7

SEVERE SEPSIS

Sepsis + organ failure OR lactate level >4

CNS

Pulmonary (ALI)

Heme

(

coags

& platelets)

Liver (

bili

)

Kidney (AKI)

Circulatory systemSlide8

SEPTIC SHOCK

Sepsis + hypotension

Unresponsive to initial bolus (20-30 cc/kg)

Most septic patients are UNDER-resuscitated

Hypotension = SBP<90 OR 40 mmHg below baseline

OR MAP <65 mmHg or >25mmHg below baselineSlide9

EPI/PATH OF SEPSIS

10

th

leading cause of mortality

750,000 hospitalizations/year

Most common sources:Lung

Abdomen

GU

Skin/soft tissue

CNSSlide10

ED WORKUP OF SEPSIS

CAREFUL history

Complaints may be nonspecific, especially very old/young

VITAL SIGNS ARE JUST THAT … but lack of fever rules out

nothing

CAREFUL physical

Inspect every inch/every orifice

BE SUSPICIOUSSlide11

ED WORKUP OF SEPSIS

Labs

The

usuals

– CBC, CMP, U/A, CXR, EKG

The unusuals

:

Lactate

?

procalcitonin

?

Cultures of every fluid

Imaging

XR

US – RUSH protocol/IVC collapse

CTSlide12

>50% collapse during inspiration indicates low CVP/likely fluid responsivenessSlide13

TREATING SEPSIS: EGDT

Landmark study (2001) showed that “bundling” sepsis management techniques and starting them in the ED showed

mortality benefit

(NNT=6)

Focuses on aggressively managing

Preload

Afterload

Oxygenation

Source controlSlide14

EGDT ALGORITHMSlide15

THE ABCs of EGDT

“Are you OK?”

Rapid

identification of the septic patient

Initiating

diagnostic steps immediately (IV, monitor, early lactate measurement)Slide16

THE ABCs of EGDT

A & B – oxygenation status & work of breathing

Obvious airway compromise/respiratory distress = easy!

Measures of poor oxygenation:

Lethargy, restlessness, altered MS

Pulse Ox/RR/PaCO2

ScvO2

– what the heck is that??

poor

oxygen delivery to tissues/overwhelming oxygen

debt

(<70% = poor O2 delivery)

Early intubation

& mechanical

ventilationSlide17

THE ABCs of EGDT

Other adjuncts to A&B

Transfusion

if

hematocrit

<30%

Lactate

– measure of anaerobic metabolism of tissues

Even mild elevations (>2) associated with increased mortalitySlide18

THE ABCs of EGDT

C – circulatory status

BP is an imperfect gauge of true circulation!

Look for subtle signs of

hypoperfusion

… like:

Going IN: Rapid

central venous

access (<2hr)

Preload –

multiple

IVF boluses

Afterload

pressors

(generally

norepinephrine

)

Coming OUT: measure strict

UOPSlide19

THE ABCs of EGDT

D&E – disability & exposure

WHERE IS THE SOURCE?? Full inspection of the patient

Lung – most common

Kidneys/bladder

Skin/soft tissue

GI

GU/GYN

Other (FBs, CNS, bone, etc)

UNKNOWN in up to 1/3 of cases

BROAD

Abx

coverage until you know what bug (culture, culture, culture!)Slide20

GOALS OF EGDT – when to stop?

Airway/Breathing

ScvO2 >70%

By means of: intubation/ventilation;

transfusion +/- addition of

inotrope

if

Hct

<30%

Improving lactate level

Circulation

CVP 8-12

(must measure thru central line; also use IVC)

Uop

>0.5 cc/kg/hr

MAP 65-90 mmHgSlide21

ADJUNCT SEPSIS THERAPIES

Steroids – very controversial

Generally reserved for the patient in septic shock unresponsive to

pressor

& fluid therapy

Mechanical ventilation lung-protective strategies

Low TV, low plateau pressures

Aspiration precautions

Tight glucose control

GI/ulcer & DVT prophylaxisSlide22

PATIENT DISPO

Admit, admit, admit!!

To the floor ONLY if mild sepsis and responding to ED therapy

THESE PATIENTS GET WORSE QUICKLY

Mortality rates

20% sepsis

40% severe sepsis

60% septic shock

Increase with every organ system involvedSlide23

BACK TO THE CASE …

Significant labs:

WBC 9,000 15% bands

H/H 9.2/28.3

Glu

186

HCO3 16

U/A + nitrites

CXR clear

How would you manage this patient??Slide24

SEPSIS: TIME=MORTALITY