24 This is hidradenitis in the opening of a hair follicle that is con Staphylococcus aureus infects Fig 2410 Staphylococcus in the epidermisIt occurs most frequently in infants and children up t ID: 162921
Download Pdf The PPT/PDF document "Hidradenitis suppurativa" is the property of its rightful owner. Permission is granted to download and print the materials on this web site for personal, non-commercial use only, and to display it on your personal computer provided you do not modify the materials and that you retain all copyright notices contained in the materials. By downloading content from our website, you accept the terms of this agreement.
24 Hidradenitis suppurativa This is hidradenitis in the opening of a hair follicle that is con- Staphylococcus aureus infects ( Fig. 24.10 Staphylococcus in the epidermis.It occurs most frequently in infants and children up toage 6. A fever and reddening around the mouth or eyesfirst appear, followed by painful exfoliation, erosion andblistering. Systemic management and care, and administration ofantibiotics are the main treatments. Outline Clinical features , Clinical images are available in hardcopy only. Clinical images are available inhardcopy only. Fig. 24.12 Pyodermia chronica glutealis. Large infiltrating plaques accompanied by pus- C.Systemic infections Go Back to the Top To Order, Visit the Purchasing Page for Details C. Systemic infections 461 24 characterized by reddening of the tongue (strawberrytongue) and dense erythema on the whole body.Eruptions do not appear around the mouth (perioral pal-lor).Increased antistreptolysin O has diagnostic value. Peni-cillin is administered. tongue. At the early stages, tongue fur which is often referred tothe tongue fur resolves in a day or two, leaving the typical straw-axillary fossae, and medial thighs, spreading over the wholeDiffuse erythema appears on the face, except at the peripheries ofthe mouth and nasal alae (perioral pallor). Hemorrhagic lesionsand systemic lymph node enlargement also occur. As the feverto disappear. After exfoliation, the eruptions heal without post- Fig. 24.16 . Eruptions are caused on the whole body by an exotoxin pro-duced by Streptococcus pyogenes . This bacterium first infects thePost-infectious complications of Streptococcus pyogenes Elevated levels of ASO and ASK, leukocytosis, and left shiftof the nuclei in leukocytes are caused by streptococcal infection.Bacteria are detected from the primary infection site, such as theRubella, Kawasaki disease and drug eruptions should be dif- Fig. 23.23 Oral penicillin G is the first-line treatment. Although eruptions Streptococcus may proliferate again in the phar- Treatment , Prognosis Differential diagnosis Laboratory findings Complications Pathogenesis Clinical features Fig. 24.16 Clinical course of scarlet fever(streptococcal infection). Clinical images are available in hardcopy only. 462 24Bacterial Infections 24 ynx, causing complications such as nephritis or rheumatic fever.After termination of medication, periodic examinations such as It is an acute bacterial infection in subcutaneous tissueand superficial fascia ( Fig. 24.3 ). The extremities andgenitalia of persons middle-aged and older are most fre-quently affected.The main systemic symptoms are reddening andswelling of skin, ulceration, and fever accompanied byintense pain.High doses of antibiotics at the early stages and surgicaldébridement are the main treatments. Multiple organ fail-ure may lead to death. The extremities (lower legs in particular), genitalia andswelling that rapidly progress with marked systemic symptoms. Fig. 24.17 reduced according to the progression of the fasciitis. Even whenthe periphery of the lesion appears normal to the naked eye, thesubcutaneous tissue is affected. Necrotizing fasciitis is character-arthralgia, muscle pain, shock and multiple organ failure. Necro-tizing fasciitis of the genitalia is called FournierÕs gangrene.Necrotizing fasciitis frequently occurs as a complication of toxic- Streptococcus pyogenes anaerobes such as Bacterioides fragilis and Peptostreptococcusanaerobius. Streptococcus pyogenes leading to a sudden onset of necrotizing fasciitis. Anaerobic bac-teria tend to infect individuals with an underlying disease, suchas diabetes. In some cases, a micro-injury or tinea pedis inducesclear leukocytes occur from the lower dermal layer to the under- Pathology Pathogenesis Clinical features Outline 4.Necrotizing fasciitis MEMO syndrome (TSLS) Synonym: Severe invasive streptococcalinfectionThe main cause is streptococcal pyogenicexotoxin (SPE), produced by streptococci andso-called Òkiller bugs.Ó Swelling in theextremities and fever occur, rapidly progress-ing to necrotizing fasciitis (described later),multiple organ failure and shock. MEMO The immunological response between anti- Go Back to the Top To Order, Visit the Purchasing Page for Details