PPT-DIABETIC EMERGENCIES DIABETIC KETOACIDOSIS (DKA)

and HYPEROSMOLAR HYPERGLYCAEMIC STATE HHS By Dr Shamanth Soghal JSD FY2 Medical Rotations Queen Elizabeth Hospital Birmingham OBJECTIVES THINGS NOT COVERED Hypoglycemia Euglycemic DKA. Mary . Manandhar. Carmel Dolan. Paul Rees-Thomas. Pascale Fritsch. Background. Ageing world. : today, more people aged 60 and above than children under 4. Lack . of awareness and knowledge within the humanitarian sector. Endocrine Block. - . Diabetic Complications. - . Ketone bodies metabolism. - DKA. :. Definition . Causes and Mechanisms. Manifestations. Precipitating Factors. - Hyperosmolar . hyperglycaemic. state (HHS) = . Danielle’s Law Training. Responsibilities & Quality of Care. You have an important role in caring for individuals with . intellectual and developmental . disabilities. Recognizing medical issues and obtaining appropriate care is vital. DR PAVANI KALYANAM. PROFESSOR. DEPARTMENT OF ANAESTHESIOLOGY . OSMANIA GENERAL HOSPITAL. HYDERABAD. ENDOCRINE. . SYSTEM. Growth and development. Metabolism. Control of body temperature, heart rate and blood pressure. Objectives. Introduce a patient case on diabetic ketoacidosis (DKA). Discuss the clinical presentation of DKA. Review the differences between DKA and HHS. Guidelines for DKA treatment and best practices.  . Ann Going & Chris Hall. 2020. Ketones. DKA IS A DANGEROUS AND POTENTIALLY LIFE THREATENING CONDITION WITH EARLY APPROPRIATE MEDICAL INTERVENTION ESSENTIAL.. What are . Ketones. Metabolism of fats can provide the body with energy when glucose is not available to the cells. Failure to take insulin. Failure to increase insulin. Illness/Infection. Pneumonia. MI. Stroke. Acute stress. Trauma. Emotional. Medical Stress. Counterregulatory hormones. Oppose insulin. Stimulate glucagon release. October 2019. DKA background. Diagnosis. Hyperglycemia (glucose > 200). Acidosis (venous pH < 7.3). Ketosis (urine or serum ketones). Risk factors. Not previously diagnosed with diabetes. 1/3 of children with type 1 diabetes first present with DKA. Dr. S. . Parthasarathy. . MD., DA., DNB, MD (. Acu. ), Dip. . Diab. . DCA, Dip. Software . statistics . PhD . (. physio. ). Mahatma Gandhi Medical college and research institute , . puducherry. India . Dr. Faryal Baddia. Objectives. General. Specific. Type of study design. Data collection method. “. Guidelines for developing DR Program in India. . By . Arvind. Eye Care System.”. Materials & Methods. Paediatric emergency medicine . spr. Learning outcomes. Be able to diagnose DKA. Start appropriate management. Discuss recent research / updates. Prescribing session. introduction. incidence of DM . 25 per 100 000 . Tom Heaps. Consultant Acute Physician. Case 1. 82-year-old Caucasian female with T2DM (>30 years) and HTN. BMI 19. Metformin. 500mg TDS , . Gliclazide. 40mg BD, . Ramipril. 5mg OD. Admitted with 1/52 of increasing drowsiness, confusion and reduced oral intake. page ITC1-2 page ITC1-5 Treatmentpage ITC1-9 Practice Improvementpage ITC1-14 page ITC1-16 Christine Laine, MD, MPHBarbara J. Turner, MD, MSEDSankey Williams, MDJennifer F. WilsonThe content of In the Insulinomas. ). Acute:. 1. Diabetic . Ketoacidosis. . 2. Hyperglycemic . Hyperosmolar. state. 3. Hypoglycemia: (patients under treatment). complications. Diabetic . Ketoacidosis. and . Hyperosmolar.