PPT-Skin Pathology III Pemphigus Vulgaris

Type II Hypersensitivity Reaction IgG Autoantibodies against Desmogleins that disrupt intercellular adhesions and result in the formation of blisters Loss of intercellular attachment . s. kin biopsies. Dr. Claire Murray. For IMF. Normal Skin. Perilesional. skin. Lesion. For histology. Procedure for biopsy. Ellipse incisional biopsy helps preserve an intact blister. Punch biopsies are more likely to disrupt the roof. It can really get under your skin. By. Tammy Chamness. Autoimmune disease. Characterized by the presences of autoantibodies that are produced by the immune system, but act against the body’s healthy cells and tissues .. DISEASES. کنفرانس ضایعات تاولی پوست . 25/12/94. Dr. : E. . Salimi. . Dermatologist. Kermanshah university of medical sciences. Key features. . Pemphigus is a group of autoimmune blistering . Introduction to Medical Terminology, Ehrlich. Components of the Skin. Protection. – Barrier to sun & invasion of pathogens, holds moisture in & prevents body tissues from drying out. Sensory Perception . Emerging Treatments . Description of Program. This program will include a discussion of investigational agents not approved by the FDA for use in the US.. Overview of Pemphigus Vulgaris. Pemphigus: Autoimmune Blistering Diseases. Pemphigus Vulgaris. Introduction. M. onitoring Treatment Response. Case 1. Nikki. Physical Examination. Physical Examination (Cont.). Assessment and Treatment Plan. Diagnosis. Supportive Care and Topical Treatment. Part 12. . Pathology and diseases of . the musculoskeletal and nervous systems. Section A: musculoskeletal . system. Part II: general pathology, infections and neoplasia of muscle . Prepared by Judith Handlinger . 2000-2009 . By Dr. . Hisham. . Arda. , M.D. Ph.D., Associate Prof. AN-. Najah. Univ.. Nablus-Palestine. . Acne . Vulgaris. . is primarily a chronic inflammatory process of the . pilosebaceous. unit. . Dr. Amrita . Upadhyaya. AP. Dermatology. 21st. . july. 2017. Blister: fluid filled cavity formed within or beneath the epidermis. Vesicle: blister < 0.5cm. Bullae: >0.5cm. Mechanism of blister formation:. د. رزان. OBJECTIVES. 1.Classification.. 2.presentation.. 3.differentiation.. 4.outline management.. Immunoblistering. skin diseases:. Group of disorders characterize by having . bullae. . or vesicles. A vesiculobullous disease is a type of mucocutaneous disease that is characterized . by . vesicles and bullae (i.e. blisters. ).. . Both vesicles and bullae are fluid-filled lesions, and they are distinguished by size (vesicles being less than . ous membranes.Included in this group ispemphigus vulgaris,a bullous disease involv- here is a wide variety ofblister-ing diseases,some ofwhich can Bullous dermatoses can be debilitating and possibly f Associate Professor. Hair Transplant. . & Laser . Department of Dermatology- KSU. Blistering Diseases. . Objectives. To know the definition . & classification of Blistering diseases. To recognize the primary presentation of different types of main blistering diseases. Dr. Abdullah ALAKEEL. Assistant . P. rofessor. & Consultant. Department. of . Dermatology. KKUH. Circumscribed skin lesions containing fluid (If the size ≤ 5mm = vesicle If the size > 5mm = bulla) .