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Etiology of Dental Caries Etiology of Dental Caries

Etiology of Dental Caries - PowerPoint Presentation

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Uploaded On 2016-02-28

Etiology of Dental Caries - PPT Presentation

DrRai Tariq Masood Early Theories Worm Theory Humour Theory Parasitic Theory Vital Theory Chemical Theory Chemoparasitic Theory Proteolytic Theory Proteolysis Chelation Theory ID: 234444

surface caries theory enamel caries surface enamel theory tooth acid saliva lesion factors classification cavitation flow based demineralization arrested

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Slide1

Etiology of Dental Caries

Dr.Rai

Tariq

MasoodSlide2

Early Theories

Worm Theory

Humour

Theory

Parasitic Theory

Vital Theory

Chemical Theory

Chemo-parasitic Theory

Proteolytic

Theory

Proteolysis-

Chelation

TheorySlide3

Current Concepts of Caries Etiology

Keyes Circles

Caries is multi-factorial disease comprising of four factors

Susceptible Tooth Surface

Micro-organism

Diet (Sucrose)

Appropriate time

Each one of them is of equal importance in

aetiology

of cariesSlide4

Classification Based on Morphology

Occlusal

Caries ( Pit & Fissure Caries)

Smooth Surface Caries

Buccal

& Lingual Caries

Proximal CariesSlide5
Slide6

Classification Based on Severity & Progression

Rampant Caries

Early Childhood Caries ( Baby Bottle Tooth Decay)

Radiation CariesSlide7

Classification Based on Part of Tooth Involved

Enamel Caries

Dentinal Caries

Cemental

CariesSlide8

Classification Based on Activity

Primary Caries

Secondary Caries

Residual Caries

Arrested CariesSlide9
Slide10

Clinical Manifestations of Caries Process

1-

Early Changes

First time demineralization of enamel when PH falls below 5.2 – 5.5

Demineralization can not be detected clinicallySlide11

2- White Spot Lesion

First visible clinical presentation

Caused by sub-surface enamel demineralization

Surface is intact

It may or may not progress to frank

cavitationSlide12
Slide13

3- Hidden or Occult Caries

Calcium and Phosphate moves from subsurface to the surface.

Calcium and Phosphate along with fluoride from saliva precipitate on effected surface enamel.

It will occlude the pores that limits demineralization of surface enamel.

Hence intact surface enamel and caries in subsurface level.

Not clinically visible.Slide14

4- Frank

Cavitation

Sub-surface carious lesion increases in dimensions.

Collapse of surface layer

Cavitation

More plaque accumulation so rapid tooth destruction.

It takes 18 (+- 6 months) to progress from white lesion to

cavitation

.Slide15

5- Arrested Caries

Carious lesion can become arrested at any stage.

If the causal factors are changed or protective factors are increased.

Example :Proximal Carious lesion and if adjacent tooth is lost then it becomes self cleansing.Slide16

Micro-Biology of Dental Caries

Streptococcus

Mutans

Ability to stick to tooth surfaces

Ability to produce lactic acid

Resist the

acidogenic

environment

Produce intracellular polysaccharide

Streptococcus

Sobrinus

LactobacillusSlide17

Formation o

f Plaque

Adherence of bacteria to pellicle or enamel surface.

Adhesion between bacteria by polysaccharide chains

Subsequent growth of bacteriaSlide18

Risk Factors/Protective Factors

Total oral Bacterial population

Tooth Morphology

Salivary secretion rate

Intake of carbohydrates

Oral Hygiene Habits

Use of FluoridesSlide19

Role of Saliva in Caries

Also called

Liquid Enamel

because of high mineral content

Cleansing Action

Buffering Capacity

Antibacterial Action by

Lysozyme,Lactoperoxidase,hemoprotein

enzyme (Prevents bacterial colonization)

Saturated with Calcium and Phosphate

Most prominent antibody in saliva IGA.

Proteins like

statherin

protects

hydroxyapetite

crystals.Slide20

Flow rate:

Role of saliva, with respect to caries, is in the removal of bacterial and debris. Average un-stimulated flow rate is 0.3 ml/minute and amount prior to swallowing 0.9-1.2 ml

Quantity

: Normal is 700-800 ml/day. Less leads to rampant caries as seen in

Xerostomia

.

Viscosity:

Thick saliva associated with high caries but not confirmed.

pH:

Depends on bicarbonate

content.Saliva

may be slightly acidic as it is secreted at

unstimulated

flow rates but may reach PH of 7.8 at high flow rates.Slide21

Buffering Action

Bicarbonates are most important buffers

It reacts with acid and release weak carbonic acid.

Carbonic acid is rapidly decomposed into water and carbon dioxide.

So acid is completely removed.

When there is excess sucrose

intake,intense

acid production will breakdown the buffers.Slide22

Thank you