/
Syncope Syncope

Syncope - PowerPoint Presentation

cheryl-pisano
cheryl-pisano . @cheryl-pisano
Follow
555 views
Uploaded On 2017-04-21

Syncope - PPT Presentation

Alexander Thai Emergency Medicine Resident PGY1 Disclaimer I have no affiliations or financial benefits from this lecture Disclaimer I have no affiliations or financial benefits from this lecture ID: 539920

syncope cardiac physical qtc cardiac syncope qtc physical soccer exam fainting history cardiology testing presentation prior case reports presents minute step consultation

Share:

Link:

Embed:

Download Presentation from below link

Download Presentation The PPT/PDF document "Syncope" is the property of its rightful owner. Permission is granted to download and print the materials on this web site for personal, non-commercial use only, and to display it on your personal computer provided you do not modify the materials and that you retain all copyright notices contained in the materials. By downloading content from our website, you accept the terms of this agreement.


Presentation Transcript

Slide1

Syncope

Alexander ThaiEmergency Medicine Resident PGY-1Slide2

Disclaimer

I have no affiliations or financial benefits from this lectureSlide3

Disclaimer

I have no affiliations or financial benefits from this lectureBut if you're interested in sponsoring me, I am quite availableSlide4

Case presentation

Holtz is a 12-year-old male who presents for his sports physical prior to trying out for a traveling soccer team. During your history-taking, he admits to fainting last week during a soccer practice. He thought it was due to possible dehydration so he never mentioned it to his parents. When asked further about this episode, he recalls that he was running on the field then suddenly collapsed. He was unconscious for about a minute and “came to” when his teammates roused him. No previous episodes of fainting. Mom reports that her brother (Holtz’s uncle) died of drowning when he was a teenager.Slide5

Case presentation

Holtz is a 12-year-old male who presents for his sports physical prior to trying out for a traveling soccer team. During your history-taking, he admits to fainting last week during a soccer practice. He thought it was due to possible dehydration so he never mentioned it to his parents. When asked further about this episode, he recalls that he was running on the field then suddenly collapsed. He was unconscious for about a minute and “came to” when his teammates roused him. No previous episodes of fainting. Mom reports that her brother (Holtz’s uncle) died of drowning when he was a teenager.

Are you concerned about Jack? Why or why not?Slide6

Case presentation

Holtz is a 12-year-old male who presents for his sports physical prior to trying out for a traveling soccer team. During your history-taking, he admits to fainting last week during a soccer practice. He thought it was due to possible dehydration so he never mentioned it to his parents. When asked further about this episode, he recalls that he was running on the field then suddenly collapsed. He was unconscious for about a minute and “came to” when his teammates roused him. No previous episodes of fainting. Mom reports that her brother (Holtz’s uncle) died of drowning when he was a teenager.

Are you concerned about Jack? Why or why not?

Yes

. Red flags: Syncope with exertion, family history of possible sudden cardiac death – both concerning for etiology of syncopeSlide7

Case presentation (Question 1)

Holtz is a 12-year-old male who presents for his sports physical prior to trying out for a traveling soccer team. During your history-taking, he admits to fainting last week during a soccer practice. He thought it was due to possible dehydration so he never mentioned it to his parents. When asked further about this episode, he recalls that he was running on the field then suddenly collapsed. He was unconscious for about a minute and “came to” when his teammates roused him. No previous episodes of fainting. Mom reports that her brother (Holtz’s uncle) died of drowning when he was a teenager.

What is the next step in management?

Cardiac event monitoring

CT scan of the brain

Electrocardiography

Electroencephalography

Tilt Table

Testing

EchocardiographySlide8

Case presentation (Question 1)

Holtz is a 12-year-old male who presents for his sports physical prior to trying out for a traveling soccer team. During your history-taking, he admits to fainting last week during a soccer practice. He thought it was due to possible dehydration so he never mentioned it to his parents. When asked further about this episode, he recalls that he was running on the field then suddenly collapsed. He was unconscious for about a minute and “came to” when his teammates roused him. No previous episodes of fainting. Mom reports that her brother (Holtz’s uncle) died of drowning when he was a teenager.

What is the next step in management?

Cardiac event monitoring

CT scan of the brain

Electrocardiography

Electroencephalography

Tilt Table

Testing

EchocardiographySlide9

Case presentation (Question 1)

Electrocardiography should be a part of the initial evaluation for all patients who presents with syncope.

Why not the others?

Cardiac event monitoring, tilt table testing, and an echo are usually done after an abnormal ECG and a cardiology consultation has been done

Neuro-imaging not indicated if pt does not present with focal neurologic findingsSlide10

Case presentation (Question 2)

So you get the EKG…Slide11
Slide12

Diagnosis?Slide13

I’ll give you a clue…Slide14

I’ll give you a clue…

Calculate the QTcSlide15

I’ll give you a clue…

Calculate the QTc

QTc = QT/sqrt(RR)Slide16

QTc = QT/sqrt(RR)Slide17

QTc = QT/sqrt(RR)

4 big boxes = 0.8 or 800 ms

~3 big boxes = 0.6 secs or 600 msSlide18

QTc = QT/sqrt(RR)

4 big boxes = 0.8 or 800 ms

~3 big boxes = 0.6 secs or 600 ms

QTc = 0.6/0.9 = 0.666 or 666msSlide19

This child has prolonged QTc

Next step?Slide20

This child has prolonged QTc

Next step?Cardiology consultationSlide21

This child has prolonged QTc

Next step?Cardiology consultation

Treatment?Slide22

This child has prolonged QTc

Next step?Cardiology consultation

Treatment?

Beta-blockersSlide23

So back to our main topic… SyncopeSlide24

Syncope DefinedSlide25

Syncope DefinedSlide26

Syncope Defined

Brief sudden loss of consciousness with loss of postural tone that resolves spontaneouslyPre-syncope refers to feeling faint without true loss of consciousness

Literature reports occurs in 15-50% of adolescents

(highly sensitive literature)Slide27

What causes syncope?Slide28

What causes syncope?

In peds, most common cause is vasovagalSlide29

What causes syncope?

In peds, most common cause is vasovagalHowever, your job is to rule out the life threatening causes

Dysrhythmias (usually tachydysrhythmias)

Cardiac outflow obstructions

Toxic exposures

Hypoglycemia

Ectopic pregnancySlide30

Beware of mimicsSlide31

Beware of mimics

SeizuresMigrainesHyperventilation

Choking games

H

ysteria/conversionSlide32

How to evaluate?Slide33

How to evaluate?

HistoryWhat was happening around pt?

Feeling/sensation before event?

What position was pt in when it happened?

Did pt have chest pain or HA before/after?

Witness?Slide34

How to evaluate?

HistoryWhat was happening around pt?

Feeling/sensation before event?

What position was pt in when it happened?

Did pt have chest pain or HA before/after?

Witness?

Warning signs!

Triggered by fright or sound

No prodrome

Exertional

Palpations or chest pain

Brief posturing

Family history of sudden cardiac death, known arrhythmia

Congenital heart diseaseSlide35

Other questions to think aboutSlide36

Other questions to think about

Additional hxMenstrual history

Medical problems

Access to medications/illicit drugs

Family history

Early cardiac death before 45

Known arrhythmia

Familial cardiomyopathySlide37

Physical ExamSlide38

Physical Exam

OrthostaticsChange from sitting to standing (decrease in SBP >20 or HR increase>20

More importantly, does this recreate symptoms?

Normal does not exclude cardiac dysrhythmiasSlide39

Physical Exam

OrthostaticsChange from sitting to standing (decrease in SBP >20 or HR increase>20

More importantly, does this recreate symptoms?

Normal does not exclude cardiac dysrhythmias

Full physical exam with emphasis on:

Detailed neurologic exam

Cardiac exam

Murmurs, gallops, and rubs

Signs of heart failureSlide40

Physical Exam

OrthostaticsChange from sitting to standing (decrease in SBP >20 or HR increase>20

More importantly, does this recreate symptoms?

Normal does not exclude cardiac dysrhythmias

Full physical exam with emphasis on:

Detailed neurologic exam

Cardiac exam

Murmurs, gallops, and rubs

Signs of heart failure

Document thoroughlySlide41

TestingSlide42

Testing

#1 thing in anyone with AMS?Slide43

Testing

#1 thing in anyone with AMS?AccucheckSlide44

Testing

#1 thing in anyone with AMS?AccucheckECG

Urine Preg

Hgb

Urine drug screen (if still altered)

No neurologic imaging indicated unless persistent focal neurologic abnormalitySlide45

Cardiac diagnoses to look for?Slide46

Cardiac diagnoses to look for?

Plumbing

Electrical ProblemsSlide47

Cardiac diagnoses to look for?

PlumbingHypertrophic cardiomyopathy

Anomalous coronary arteries

Ventricular cardiomyopathy

Aortic Stenosis

Pulmonary HTN

Acute Myocarditis

Dilated Cardiomyopathy

Electrical ProblemsSlide48

Cardiac diagnoses to look for?

PlumbingHypertrophic cardiomyopathy

Anomalous coronary arteries

Ventricular cardiomyopathy

Aortic Stenosis

Pulmonary HTN

Acute Myocarditis

Dilated Cardiomyopathy

Electrical Problems

Long QT

Brugada

Polymorphic VT

Pre-excitation (WPW)Slide49

Long QTSlide50

Long QTSlide51

Long QT

Delayed repolarizationMay be familial (family hx)QTc >460

Can cause syncope -> torsades de pointes -> v. fib arrest

Treatment = beta blockersSlide52

Short QTSlide53

Short QTSlide54

Short QT

QTc < 320Increased incidence of atrial fibrillationMay indicate an electrolyte abnormality (hypercalcemia for example)

High risk of ventricular dysrhythmia and sudden cardiac deathSlide55

Brugada

PatternSlide56

Brugada

PatternSlide57

Brugada

PatternRSBB or incomplete RBBB in V1-V2 with ST elevation

At risk for monomorphic and polymorphic v. tachycardia

Ultimately need pacemakers Slide58

WPWSlide59

WPW

Short PR interval and delta wave are diagnosticRepresents signal transmitting around the AV node through bundle of Kent

Can go into tachydysrhythmias… Beware… can be wide complex irregular tachycardia

If stable may want to discuss with cardiology prior to drug administration as adenosine and diltiazem can be problematic

Unstable… SHOCKSlide60

Who gets cardiology consultation/follow-up?Slide61

Who gets cardiology consultation/follow-up?

Family hx of sudden death or malignant arrhythmiaExercise related syncope

Cardiac history

If abnormal ECG, fax to cardiology (or text at Holtz) for an interpretation prior to admitting patientSlide62

High YieldSlide63

High Yield

If pt at baseline, little need for extensive workupScreening ECG, through low yield, will screen for most life-threatening cardiac syncope

Look for anemia, hypoglycemia

Always check U-preg

No indication for ED neuro-imaging in a child without focal neurological signSlide64

Resources

5-minute PEM consulthttp://

www.nccpeds.com/ContinuityModules-Spring/Spring%20Faculty%20Modules/Syncope-Faculty.pdf

http://

www.slideshare.net/Odigia/syncope-17173349

Google images