PDF-7 : 1INTRODUCTNon cirrhotic portal fibrosis (NCPF) is a syndrome of ob

NONCIRRHOTIC PORTAL FIBROSIS K Gupta Jacob George rithvi Giri 463 ATY Gross pathology Liver may be normal to markedly nodularNodularity when present is limited to subcapsular zone in contrast. - clinical characteristics & risk factors Huisong Chen, Goolab Trilok, Fei Wang, Xiaolong Qi, Junjie Xiao & Changqing Yang Division of Gastroenterology & Hepatology, Digestive Disease Institute, Tongj and Understandable II – . Patterns of Inflammation. Dr Ian Chandler. February 2013. With acknowledgements to Prof S . Hubscher. , Birmingham. Patterns of Inflammation in the Liver. Portal inflammation. OF HEPATIC PERFUSION DISORDERS . I MARZOUK MOUSSA, F KSONTINI, L BEN FARHAT, A MANAMANI, N DALI, L HENDAOUI. MEDICAL IMAGING AND INTERVENTIONNAL DEPARTEMENT, MONGI SLIM HOSPITAL, LA MARSA TUNISIA. GI12. in congenital. hepatic fibrosis. ABSTRACT I.D . – IRIA 1066. BACK GROUND. Congenital hepatic fibrosis (CHF) is a rare autosomal recessive disorder that belongs to . fibropolycystic. group of diseases. . Li Z, Wang Z, Yin Z, Zhang Y, . Xue. X, Han J, Zhu Y, Zhang J, . Emmert. MY, Wang H.. Department of Cardiovascular Surgery, Shenyang Northern Hospital, China; Clinic for Cardiovascular Surgery, University Hospital Zurich. –. Surgical Procedure. 2. Courtesy and with permission from . Dr. Rick Selby. Professor of Surgery. Division Chief, . Hepatobiliary. and Pancreatic . Surgery and Abdominal Organ . Transplantation. Richa Shukla. Faculty mentor: Dr. Sahil Mittal. February 12, 2015. Case discussion. Reason for presentation: Abdominal pain. 41F with medical history significant for HLD, HTN and DM. Presented to ED with 6 months of waxing and waning RUQ pain, worse over 3-4 weeks prior to presentation. Richa Shukla. Faculty mentor: Dr. Sahil Mittal. February 12, 2015. Case discussion. Reason for presentation: Abdominal pain. 41F with medical history significant for HLD, HTN and DM. Presented to ED with 6 months of waxing and waning RUQ pain, worse over 3-4 weeks prior to presentation. Table of Contents Presentation 5 Presentation Resources 6-7 Teacher/Faculty Curriculum 8-9 Teacher/Faculty Resources 10 K-2nd Grade Activities/Materials 11 3rd-5th Grade A Director Dell Children’s Medical Center CF Center. October 28, 2017. Objectives. Pathophysiology of CF. Genetics of CF. Diagnostic evaluation for CF. Respiratory issues in CF. GI and nutrition issues in CF. Differential diagnosis. . of. . chest diseases. Micronodular. (. miliary. )disease. . TB. Histoplasmosis. Chicken box. Sarcoidosis. LCH. Pneumoconiosis. Alveolar . microlithiasis. Metastasis . Dr. Muntadher Abdulkareem Abdullah. M.B.Ch.B,CABM,FIBMS,FIBMS(GE. .&HEP.). Objectives. :. At the end of this lecture you must know:. 1.What . is . portal hypertension ?. 2.What is . etiology of portal hypertension ?. Ascites. Defined as the accumulation of fluid in the peritoneal cavity. . It is a common clinical finding, with many . extraperitoneal. and peritoneal causes , but most common from liver cirrhosis .. Hepatocellular Carcinoma. Risk factors for HCC. HBV – the most common causative risk factor. Increased risk :. Earlier infection. Higher DNA load (>2000 IU/L). Higher level of quant. . HbsAg. (>10,000 U/L).