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Acute Pain Management Shiv Chavan Acute Pain Management Shiv Chavan

Acute Pain Management Shiv Chavan - PowerPoint Presentation

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Acute Pain Management Shiv Chavan - PPT Presentation

Consultant Anaesthetist College Tutor QEHB What is Pain An unpleasant sensory and emotional experience associated with actual or potential tissue damage or described in terms of such damage Types of Pain ID: 1036089

pain block wrist nerve block pain nerve wrist acute regional patient skin infraclavicular interscalene spinal post signs toxicity damage

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1. Acute Pain ManagementShiv ChavanConsultant AnaesthetistCollege TutorQEHB

2. What is Pain?An unpleasant sensory and emotional experience associated with actual or potential tissue damage, or described in terms of such damage

3. Types of PainAcutePain due to clear causeUsually goes away after the healing processUsually moderate to severeChronicPersistent pain even after tissue healingUsually dull, aching painDifficult to manage

4.

5. Why should we manage acute pain?Pain causes physical and emotional sufferingDelays wound healingLeads to other complicationsCan become chronicMore hospital stay, loss work etc

6. Acute Pain ManagementWHO Analgesic ladderReverse WHO Analgesic ladder

7. Acute Pain ManagementEnteralOralSublingualParenteralIntravenousIntramuscularSubcutaneous

8. Enteral RouteAdvantagesSimpleNo need for IV accessTolerated by mostDisadvantagesSlow onsetBioavailabilityAlmost useless in severe painPt may be NBMSome pt cant swallowIntolerence

9. ParenteralIV route is most effective and popular routeIM and SC can be used but slow to workCan be titrated to the effectQuick onsetBut need IV access, nursing expert and monitoring

10. IntravenousIntermittent bolusPatient Controlled AnalgesiaContinuous Infusion

11.

12. Patient Controlled AnalgesiaMost popularNeeds bolus to start withSafety features Better patient satisfaction

13. Adjuvants in acute pain managementAnti emeticsHydrationOxygen

14. Non Pharmacological interventionsImmobilisationProper dressingsPsychological interventions

15. Regional AnaesthesiaLocal InfiltrationNerve blocksNeuraxial blocksEpiduralSpinal (opioids)

16. Regional Anaesthesia for Core Surgical Trainees

17. Why do you need to know?Most of your patients would have had RA or will have one…..You might be called to see pt who has had a block/spinalYou might have to do something to give pain relief before surgeryPrescribe infusions/treat complications in emergency

18. What do you need to know?Common blocksSome side effects and complicationsLocal anaesthetic drugs and toxicitySpecific concerns with trauma patients

19. Regional anesthesia - UsesProvide anesthesia for a surgical procedure Provide analgesia post-operatively or during labor and deliveryDiagnosis or therapy for patients with chronic pain syndromes

20. Regional anesthesia - typesTopicalLocal/FieldIntravenous block (“Bier” block)Peripheral (named) nerve, e.g. radial n.Plexus - brachial, lumbarCentral neuraxial - epidural, spinal

21. AdvantagesRegional anaesthesia avoids GA in high risk patientsMay avoid GA in unstarved pts who can’t waitEarly ambulation – day caseUseful for post op analgesia

22. FacilitiesSkilled assistantResus facilitiesMonitoringAsepsisNerve stimulator / paraesthesiaNeedlesB-bevel (non cutting, 45 degree)InsulatedTube syringe-needle (aspirate!)

23. Common Peripheral Nerve BlocksUpper limb:InterscaleneSupraclavicularInfraclavicularAxillaryIndividual nerve blocksWrist blockRing blocks

24. Interscalene Block

25. Interscalene Block

26. Interscalene Block

27. Complication of Interscalene BlockHorner’s syndromePhrenic nerve palsyCervical spinal/epiduralIntra-arterial injectionPneumothoraxNerve damage

28. Supraclavicular block

29. Supraclavicular Block

30. Infraclavicular Block

31. Infraclavicular Block

32. Infraclavicular Block

33. Complications of Supra/Infraclavicular blockPneumothoraxIntravascular injectionsDamage to surrounding structures

34. Axillary Block

35. Axillary Block

36. Elbow BlockNot popular as no advantage over wrist block but much easier using USThree injectionsMedian medial to brachial arteryUlnar in ulnar groove (between olecranon and medial epicondyle)Radial lateral to biceps tendon

37. Forearm blocks

38. Wrist BlockPreferred to elbow for small hand surgeryEasy but not commonly practiced at this centre Hand procedures/post op3 injections

39. Wrist Block – MedianFlexor carpi radialisPalmaris longus2cm proximal to skin crease1cm deep3-5 ml1 ml to skin for palmar cutaneous branch

40.

41.

42. Wrist Block – UlnarFlexor carpi ulnarisMedial to arteryPalmar or ulnar approachUlnar approach allows cutaneous branch blockade

43.

44.

45. Wrist Block – RadialField block in area of anatomical snuff box

46.

47. Wrist Block – MedianFlexor carpi radialisPalmaris longus2cm proximal to skin crease1cm deep3-5 ml1 ml to skin for palmar cutaneous branch

48. Digital Nerve Block4 nervesBlock from dorsal side1-2 ml deep0.5 ml superficialMain complication vascular insufficiency – gangreneNO ADRENALINE

49.

50. Intravenous Regional Anaesthesia (Bier’s block)Reliable, easy, usually safeUsually done for short proceduresNeed some experience Double tourniquetLocal anaesthetics agents0.5-1% Lignocaine0.5-1% Prilocaine- Rarely some additives

51. Lower limb blocksLumbar PlexusFemoral/ 3 in 1 SciaticAnkle

52. Lumbar PlexusNerve stim/US guidededHip proceduresSingle shot/CatheterLA toxicity

53. FemoralThigh/Knee surgeryNerve stim/US guide3-in-1Pre op #NOF

54.

55. SciaticTransgluteal approch

56. SciaticKnee/Foot surgerySeveral approchesCurvilinear probe

57. Distal Sciatic Nerve BlockPopliteal Nerve blockFoot/ankle surgery

58. Ankle Block

59. Central Neuraxial BlocksSpinalEpiduralCombined spinal epidural

60. Physiological effectsCVSDrop in SVR leading to drop in BPBradycardiaRespMinimal effectHigh spinal- reduced MVGIIncreased peristalsisRenalIncreased RBF

61. What are local anesthetics?Drugs which produce reversible inhibition of excitation and conduction in peripheral nerve fibres and nerve endings, and thus produce loss of sensation in circumscribed area of the body.

62. ClassificationEstersCocaineProcaineChlorprocaineAmethocaineTetracaineAmidesLignocaineBupivacaineLevobupivacaineRopivacaineMepivacainePrilocaine

63. Mechanism of ActionThey act by blockade of sodium channels so that:1- The threshold for excitation increase2- Impulse conduction slow3- The rate of rise of the action potential declines, and4- The ability to generate an action potential is abolished or canceled

64. AgentpKaLipid SolubilityPotencyProtein binding %OnsetDurationProcaine8.91111SlowShortLidocaine7.7150265FastModPrilocaine7.750255FastModBupivacaine8.11000895ModLongRopivacaine8.1400694ModLong

65. Safe DoseAgentDoseDose( with adrenaline)Lignocaine 3mg/Kg 7mg/kgBupivacaine2mg/Kg 2.5mg/kgPrilocaine 6mg/kg 9mg/kgLevobupivacaine2.5mg/kg3mg/kgRopivacaine3mg/kg4mg/kg

66. Local Anesthetics - ToxicityTissue toxicity - RareCan occur if administered in high enough concentrations (greater than those used clinically)Usually related to preservatives added to solutionSystemic toxicity - RareRelated to blood level of drug secondary to absorption from site of injection.Range from lightheadedness, tinnitus to seizures and CNS/cardiovascular collapse

67. Safety Issues Related to Local AnestheticsDrugDoseSite of administrationCondition of the patient

68. CNS ToxicityTends to occur first (relative to CVS toxicity)See excitatory signs and symptoms firstFollowed by depressant signsCircumoral and tongue numbnessLightheadedness and tinnitusVisual disturbanceMuscle twitchingConvulsionsComaRespiratory arrestCVS depression

69. CVS ToxicityAlteration in the excitatory mechanism slower depolarization decreased HR prolonged PR interval widened QRSArrythmias bradycardia ectopic beats ventricular fibrillationDecreased cardiac output on the basis of HR contractility

70. Treatment of ToxicityIdentify the problem signs and symptoms temporal relationship IV injection 40-60 min post for peak plasma levelsCNS treatment with benzodiazepines

71. Signs and Symptoms of ToxicityRange of signs and symptomsCNS:Lightheadedness, faintPeri oral numbnessConfusion, drowsinessLOC, convulsionsCVS: Bradycardia, hypotensionCardiac arrhythmias, cardiac arrest

72. TreatmentStop injectingCall for helpSecure Airway, 100% oxygenLarge bore accessTreat ConvulsionsClose monitoring of cardiovascular status

73. TreatmentTreat cardiovascular symptomsIf in cardiac arrest, start CPR (ALS algorithm)Intralipid emulsion:1.5ml/kg over 1 minFollowed by 15ml/kg/hrTwo additional bolus doses if refractoryDouble the infusion rateMax dose: 12mg/kg

74. TreatmentContinued ITU careBloods for drug levelPaper work

75. Trauma and RAAcute polytraumaShockCompartment syndrome

76. Compartment syndromePNB and Compartment syndromeDoes it really mask the compartment syndrome?Most case reports from Surgeons say it does, but if you look closely every patient was in severe pain!!!!