PDF-bone resorption are balanced by osteoblastic new bone forma

tion Figure 1 Skeletal aging is characterized as a gradual loss of bone mass due to an excess of bone resorption that is not matched by new bone formation There are two major types of bone. MSK Pharmacology -Virginia Lam. Case study. Mary is 78 years old female. She came in to AED after . a. fall. She said the floor was wet, she slipped. Her right leg appeared shortened and externally rotated. . VERTICAL RIDGE AUGMENTATION. Edentulism. Once the teeth are lost, a continuous . resorptive. process. Results. Diminished volume and strength of residual bone. Loss of facial vertical dimension. Impaired . Hematopoiesis. Bone Formation. Fetal. INTRAMEMBRANOUS OSSIFICATION. ENDOCHONDRAL OSSIFICATION. From birth to young adult. APPOSITIONAL (flat, short, irregular bones). Increase diameter for ALL bones. portion of a bone. The radio- graphic appearance of living bone is the same as that of dead bone ofEgyptian mummies or prehistoric skeletons. Consequently, bone necrosis has no specific radiographic a 50-70% Mineral. Hydroxyapatite crystal. . Ca. 10. (PO. 4. ). 6. (OH). 2. 20-40% Organic Matrix. Type I Collagen (90% of total bone protein). Other proteins*. 5-10% Water. Some Lipids. Cells of Bone. Otosclerosis. . Otosclerosis. is an . autosomal. dominant disease, in which the fixation of footplate of the stapes in the oval window will occur due to development of spongy bone from the bony labyrinth, causing immobilization of the footplate of the stapes, which reduces the transmission of vibration to the inner ear. Periodontal ligament and bone response . to sustained orthodontic force. Presented by:. Dr Somayeh Heidari. Orthodontist . . Reference: . . Contemporary Orthodontics . Chapter 8. William R. Proffit, Henry W. Fields, David M.Sarver. 2013. Mosby. 1- Houses the roots of . teeth:Anchors. the roots of teeth to the alveoli, which is achieved by the insertion of . Sharpey’s. fibers into the alveolar bone proper. 2-Helps to move the teeth for better occlusion.. In general , metabolic bone disease affects the skeleton in one of two ways : there is either too much or too little calcified bone .The latter change , which comprises the majority of metabolic bone disease , is due either to decrease in the amount of bone formed , or to excessive resorption of bone. In turn this may be due to a variety of causes but most commonly to abnormalities of vitamin D and calcium metabolism , which in turn arise from abnormality of diet or renal function , endocrine abnormalities ( particularly of the parathyroid gland) , drug therapy or poisoning .. Ahmadi. Patient identification. A 35 y/o woman ,single, from and living in . Shahriyar. , housewife. Chief complain: generalized musculoskeletal pain. Present illness. This is a 35 y/o woman who was well until 7 month age that gradually developed bone . Are They Clinically Useful?. Dr.Zahedi. fellow of endocrinology. bone turnover markers. BTM can be measured in serum, plasma and urine and their levels relate to the activity of osteoblasts (bone formation markers) and osteoclasts (bone . large number of compounds with different side chains; they all have a strong affinity for metal ions like calcium and iron and are completely resistant to enzymatic hydrolysis. The second activity 8 Pathology Resource Registry Massachuse�s Eye and Ear 243 Charles Street Boston, MA 02114-3096 Free Brochures for your Oce or Clinic about Temporal Bone Research and Donation The Gi&# The metabolic roles of calcium and phosphorus in energy metabolism, signal transduction, and the formation and turnover of bone; . Calcium and phosphate homeostasis and affecting factors; and . Disorders of calcium and phosphorus homeostasis.