PPT-Optimizing Lung Protective Ventilation to Improve ARDS Outcomes

Brad Smith PhD University of Colorado Denver Anschutz Medical Campus Departments of Bioengineering and Pediatrics Background and Vision Background and Motivation Acute respiratory distress syndrome ARDS. Case presentation. A 45-year-old man develops ARDS after sustaining multiple broken bones in an automobile accident. The man weighs 70 kg. Mechanical ventilation is initiated in the AC mode with the following settings: (PEEP), 10 cm H2O; (FiO2), 70%; respiration rate, 12/min. . Ventilation. Alfred E. Baylor, III, MD. Critical Care Fellow. March 18, 2015. Objectives. To review the indications for standard mechanical ventilation. To evaluate the need to escalate therapy to rescue modes. Have we been killing our Patients?. Philip M. Hartigan, MD. Brigham & Women’s Hospital. Harvard Medical School. Case Report:. 54 y/o male. Smoking History. COPD. Persistent cough. CXR - Large RUL mass. Julia E. Linton. York College/ Wellspan Health Nurse Anesthesia Program. Objectives. Review patient case scenario. Review some basic principles of respiratory physiology. Describe indications for and complications with one-lung ventilation. Case presentation. A 45-year-old man develops ARDS after sustaining multiple broken bones in an automobile accident. The man weighs 70 kg. Mechanical ventilation is initiated in the AC mode with the following settings: (PEEP), 10 cm H2O; (FiO2), 70%; respiration rate, 12/min. . By: Yazmin Realivasquez. Stephen Huang . Jose Torres. What is ARDS?. ARDS is a respiratory condition characterized by hypoxemia, and stiff lungs, without mechanical ventilation most patients would die. ARDS represents a response to many different insults/injuries and evolves through different phases: alveolar capillary damage to lung resolution to a fibro-proliferative phase. The pulmonary epithelial and endothelial cellular damage is characterized by inflammation, apoptosis, necrosis and increased alveolar-capillary permeability, which lead to the development of alveolar edema. . ACNP Boot Camp 2013. Stephanie Davidson, ACNP-BC. Objectives. Review the causes and differentials for ARDS. Briefly discuss the pathophysiology . Discuss the clinical manifestations of ARDS. Understand evidence based treatment options. Lisa Pristas CRNA, MSN, MSHA. September 15, 2017. Objectives. Discuss the history of mechanical ventilation and the evolution of ventilators over the years.. List and describe the different modes of ventilation available on the newer gas machines and their uses.. et al). Incidence 1.5 -7.5/ 100000 population. 28 day mortality 25 – 30%. 1. Diagnosis clinical. Differential. . diagnosis. . . LVF. Fluid overload. Mitral . stenosis. Lymphangitis. . carcinomatosis. •Paralysis (GRADE 2A) . •Conservative fluid management (GRADE 2B) . •Bronchoscopy (GRADE 2C) . •Recruitment manoeuvres (GRADE 2C) . •Prone positioning for 16hrs (GRADE 2A. Consider tracking the Murray Score at all stages. . ARDS. Acute onset (<7 days). Bilateral opacities. “not fully explained by heart failure.”. Acute Respiratory Distress Syndrome. Moderate ARDS:. P/F 100-200. Mild ARDS: . P/F 201-300. Berlin Definition - 2012. One of the most important results of the LUNG SAFE study was that ARDS was not identied as such by the primary care clinician in almost 40% of cases []. is was particularly so for mild ARDS, in whic Multisystem failure and ARDS. Multisystem . failure. ARDS. pathology. clinical features. medical treatment. mechanical ventilation . physiotherapy. Predisposing factors. IV drug users (drug + access)…. Karrar. Nader AL-. Taie. Special complications . in . the ICU. Acute respiratory distress syndrome (ARDS):. Pneumothorax: . DVT. stress ulcer. Acute . Respiratory Distress Syndrome . (ARDS. ):. It is a state of acute diffuse alveolar damage characterized by increased capillary permeability, pulmonary edema and refractory hypoxia due to right to left shunting.