PPT-Alveolar Ventilation

Author : cheryl-pisano | Published Date : 2016-09-08

During General Endotracheal Anesthesia Valentyna Groelle RN BSN SRNA Review the physiology of alveolar ventilation Discuss pulmonary changes after induction of

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Alveolar Ventilation: Transcript


During General Endotracheal Anesthesia Valentyna Groelle RN BSN SRNA Review the physiology of alveolar ventilation Discuss pulmonary changes after induction of general . Dead Space. dead space. is the volume of air which is inhaled that does not take part in the gas exchange, either because it (1) remains in the conducting airways, or (2) reaches alveoli that are not perfused or poorly perfused. In other words, not all the air in each breath is available for the exchange of oxygen and carbon dioxide.. Julia E. Linton. York College/ Wellspan Health Nurse Anesthesia Program. Objectives. Review patient case scenario. Review some basic principles of respiratory physiology. Describe indications for and complications with one-lung ventilation. D. Sara . Salarian. , . Nov 2006. Kishore P.. Critical Care Conference. Why ventilate?. Improve oxygenation. Increase/maintain minute ventilation and help CO. 2. clearance. Decrease work of breathing. Submitted in partial fulfillment of the requirements in the course. N451 Clinical Management of Adult Health Nursing III. Old Dominion University. NORFOLK, VIRGINIA. Fall, 2013. Introduction. Presenters. Points of Discussions. Advanced Basics:. Flow and Time. Limit and cycling. Rise Time. Volume vs Pressure Control. Mandatory Modes of Ventilation. Controlled Mandatory Ventilation (CMV or IPPV). Triggered Modes of Ventilation. David Taylor. dcmt@liv.ac.uk. http://. www.liverpool.ac.uk. /~dcmt. When you have worked through this you should be able to. Describe the relationships between airflow, pressure gradients and resistance – gas laws. Classification of RF. Type 1. Hypoxemic RF **. PaO2 < 60 mmHg with normal or ↓ PaCO2. Associated with acute diseases of the lung. Pulmonary edema (Cardiogenic, noncardiogenic (ARDS), pneumonia, pulmonary hemorrhage, and collapse. PULMONARY FUNCTION & RESPIRATORY ANATOMY KAAP310 Respiratory Anatomy Larynx Hyoid bone Thyroid cartilage Lateral cricothyroid ligaments Cricoid cartilage http://apbrwww5.apsu.edu/thompsonj/Anatomy%20&%20Physiology/2020/2020%20Exam%20Reviews/Exam%203/larynx%20figure.jpg APRV. By: Jeff Cline, Angie Coon, Randy Hansen. November 19, 2012. Scenario. A new trauma surgeon has arrived at your hospital to direct Surgical Intensive Care. He trained and worked several years at Baltimore Shock Trauma, where they employ Airway Pressure Release Ventilation in many of their patients with ARDS after trauma. He tried to implement APRV on one of his patients and your Respiratory Therapy staff members were not able to assist. He has asked that you develop a protocol for APRV and an educational PowerPoint to inform the staff.. Lecture . – 4. Dr.Zahoor Ali Shaikh. 1. Elastic Behavior Of The Lungs. Elastic Behavior of the lungs is due to elastic connective tissue and alveolar surface tension.. During Inspiration – lungs expand.. 1- Houses the roots of . teeth:Anchors. the roots of teeth to the alveoli, which is achieved by the insertion of . Sharpey’s. fibers into the alveolar bone proper. 2-Helps to move the teeth for better occlusion.. Alveolar bone composed of. a) outer cortical plates. b) a central spongiosa . c) bone lining the alveolus (bundle bone). . Alveolar bone proper:. lines the tooth.. Contains Sharpey’s fibers embedded into it. and Minute Volumes. Determination of Alveolar and Dead Space Ventilation and Volumes. If we take . F. DCO2. . to be zero (b/c room air is defined as zero):. One way to easily find CO. 2. production: . Dušek D, Vince A, Kurelac I, Papić N, Višković K, Deplazes P, et al. Human Alveolar Echinococcosis, Croatia. Emerg Infect Dis. 2020;26(2):364-366. https://doi.org/10.3201/eid2602.181826.

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