PPT-Central venous pressure monitoring

Dr S Parthasarathy MD DA DNB PhDphysiology FICA Dip software based statistics CUGRAIDRA Professor Mahatma Gandhi medical college and research institute Pondicherry What is it . Method. Background. Results. Conclusions. Sixteen articles met inclusion criteria. Fifteen articles . were selected for review. Articles provided . evidence . supporting establishing . blood return. IN CASE OF . LOWER LIMB VARICOSE VEINS. ABSTRACT . . ID NO 99. INTRODUCTION. Varicose veins. Chronic venous insufficiency. Venous reflux. AIM. To . study the prevalence of deep venous reflux . as primary . Update: November 2016. . Steven E. Zimmet, MD . RPVI RVT FACPh. President, ABVLM. Disclosures. President, ABVLM. Overview. Background: VLM. Testing of Foundation Knowledge: ABMS specialties. ABVLM . Superficial system. :. Drains into the venous synuses. Central system. Drain in a large, short venous trunk – the great vein of Galien. Basal system. . Drains the blood into the basilar vein and then to the great vein of Galien. Evidence base and expert opinion put into practice. Christine Herb, . MS, FNP-C, CWON. Syracuse VA Medical Center. June 13, 2014. Venous. vs.. . Arterial. . vs. . Neuropathic. 1. st. it is critical to correctly determine the etiology!. Dr. . Aditya. . Jindal. 1/4/11. Introduction. Pulmonary artery and central venous catheter. Blood pressure. Cardiac output determination. ‘‘The same old Watson! You never learn that the gravest issues may . S. DR USMAN QURESHI. SURGICAL UNIT-1. 2. presentation outlines. Intoduction. Venous and lymphatic drainage of extermities.. Pathophysiology and Mx of specific venous diseases.. .Varicose vein. Pseudotumor. cerebri. (PTC) using digital subtraction angiography. (DSA). Dr.Mohamed. . hamdy. . ibrahim. . MBBC,MSc,MD. , PhD Neurology Degree Kings lake university (USA).. Fellow of Interventional Neuroradiology (FINR)SWITZERLAND. Assistant lecturer :. Noor Wafaa Hashim. Thrombosis is the development of a ‘thrombus’ consisting of platelets, fibrin, red cells and white cells in the arterial or venous circulation. If part of this thrombus in the venous circulation breaks off and enters the right heart, it may be lodged in the pulmonary arterial circulation, causing . 1 PresenterPayal Patel, MD, MPHInfection Diseases Physician and Assistant ProfessorUniversity of MichiganMedical Director of Antimicrobial StewardshipAnn Arbor VA Healthcare SystemContributions byVine JMCON. Central OBJECTIVE. At the end of teaching students acquire . knowledge about Haemo . dynamic monitoring and able to explain and apply this knowledge while working in the educational and clinical set up.. Baseline state with normal blood pressure, venous pressure, and ICP.. Dysregulated. cerebral hyperemia with hypertension, produces . peri. -arteriolar tissue . vasogenic. edema with elevated ICP. Increased venous pressure, at possible points noted by an asterisk, produces . Dr . S. . Parthasarathy. . MD. DA. DNB., . Dip.diab. . MD(. acu. ) , DCA, Dip. . PhD (. physio. ). . Mahatma Gandhi medical college and research institute , . puducherry. – India . What is it ??. Content Validated, Evidence Based “Guideline of Venous Ulcer Guidelines”. Using the AAWC Venous Ulcer (VU) Guidelines to Manage Venous Ulcers. 3 Steps to manage a VU patient:. Assess and document patient, skin & VU.