Jeremy Clorfene PhD Head Psychologist Advocate Weight Management Program Chicago Area 565 Lakeview Parkway Suite 102 Vernon Hills IL 60061 o 8479905770 c 8478771331 wwwjeremyclorfenephdcom ID: 723230
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Slide1
FOOD ADDICTION: FACTS, FICTION, CHALLENGES
Jeremy
Clorfene, Ph.D.
Head
Psychologist Advocate Weight
Management
Program (Chicago Area)
565
Lakeview Parkway, Suite 102
Vernon
Hills, IL 60061
o
847-990-5770
c
847-877-1331
www.jeremyclorfenephd.comSlide2
Advocate Weight Management
What do we do and what is our program’s mission?
…
to provide a comprehensive weight management program that treats obesity through medical and behavioral intervention by teaching the tools needed to lose weight and maintain the weight loss for a lifetimeSlide3
How
?
Organization, structure, programming (one-on-one with MD, Behavior, RD, Exercise, Clinician + groups)
Alter
food choices, food quantity, aid in improving physical activity, and medication/health management.
that
is…more nutritious foods, less carbs/sugar, more physical activity, and improve medical/health
profile.Slide4
Current Cause of Obesity
?
1
.
Disproportionate
food intake of carbohydrate and simple
sugars (in volume)
2
. Subsequent decrease in other foods such as protein, fats,
and
fiber (poor nutrition).
3
. Diminished physical activity
4
. Diminished sleep
5
.
Medications / Medical Issues
6.
Convenience
,
cheap sugar
7
. Genetics (only explains 1/3 variance for obesity and does not account for morbid obesity
)Slide5
Is
Obesity:
Is it Food Addiction?
Is
it compulsive overeating?
Is
it Binge Eating Disorder?
Is
obesity the result of being “fat”?
Is
obesity a pathology?
Is
it normal human response to environment with dire consequences?
Is it a dieting
disorder?
Is it a stress
disorder?
Is it a body
image problem?
Is it a sign
of a deeper emotional problem?Slide6
Obesity HistorySlide7Slide8
Why The Trend? History
(Graph
):
1
. 1970’s “Fat Free
Fallacy”–Med. Community (lipids bad)
2
. Food Pyramid (
grains
grains
grains)
3
. Agribusiness Policy (Earl “Rusty”
Butz
, sect. of
agric
) “Get Big
or Get Out” – lots and
cheap! Slide9
Obstacles we are up against? (our culture)
1. Sophisticated organized
system
to make
carbs, high sugar, high calorie foods
ubiquitously available, cheap, and tasty (e.g.,
soda,
bread, sweets, cereal, fast
fried food)
2. Longer work hours, longer
commutes (especially
women)
3
. Lots and lots of
pleasure
eating habits
(
always pressing the yummy
button – not easy to change)
4
. And … YOU GOT TO EAT! Slide10
Old School Food Market
New School
Food MarketSlide11
In Essence…
We
are
all the
antidote
(treatment) to
the
venom (poison) to what Kelly Brownell
(2004) has described
a culture as a “
Toxic Food Environment”
which is
responsible for the
rapid
increase obesity
rates.
What do we do?
Control
food, educate, build skills, create positive
experiences
, accountability, support, validate,
motivate
, attempt to transform
our patients
from ___________(?)
to
the __________(?)Slide12
REAL TIME PROBLEM
In
practice
we see powerful repeated behavioral patterns.
Identify the patterns, it then guides interventions and improves outcomes (transformation)!
Common pattern:
Can’t “stop
” eating forbidden foods (and quantity
)…
weight increases…emotional
distress
ensues…
adding
to negative belief
system…which
fuels more eating.
NOT IN CONTROL
. Slide13
In many cases it
feels like,
looks
like, and acts
like…
ADDICTION
.
But is it?Slide14
Is obesity (morbid obesity) really a “Food Addiction”?
Does “Food Addiction” best explain why people can’t stop eating and making poor choices?
Does “Food Addiction” best explain why people lose weight and regain it all back and more?
Do we want to label obese people as “addicts”?
If addiction explains some, then which ones?
The
idea that a person can be addicted to
food
(or certain foods) has recently received
attention from
science, but being addicted to a “necessity”
(
i.e., oxygen, water, or food) must be evaluated thoroughly and its clinical implications handled responsibly.Slide15
Addiction looks like?Slide16
ADDICTION DEFINED
Classic hallmarks of addiction include
:
impaired control over substances/behavior,
(CAN’T STOP EVEN WHEN TRY)
preoccupation with substance/behavior,
continued “seeking”
continued use despite negative consequences (impact on social, professional, financial, health), and often denial
immediate gratification (short-term reward)
coupled with delayed deleterious effects (long-term costs)
mood/thought altering (YOU GET HIGH)!Slide17
Addiction
Defined cont
.
Physiological dependence occurs when the body has to adjust to the substance by incorporating he substance into its ‘normal’ functioning
Creating condition of
tolerance
and
withdrawal
Tolerance
: body requires increasingly larger amounts to achieve the original effects.
Withdrawal
: the negative physical and psychological symptoms when reducing or discontinuing a substance
Symptoms
:
anxiety, irritability, intense cravings, nausea, hallucinations, headaches, cold sweats, tremors, possible seizures
DSM-IV, 2013 Slide18
Common Addictions are:
Alcohol (most common)
Weed
Nicotine
Heroine
Cocaine
Morphine (Vicodin)
Benzodiazepines (Xanax)
Ritalin
Gambling
Sex (porn)
Internet? Slide19
The Debate…the Evidence:
What about Food Addiction?
Is it real? Slide20
Press/Media Diagnosis/Treatment
Peer reviewed journals / research
Public
Science
Clinical
Food Addiction / “You are an Addict”?
Finlayson, Web 2013Slide21
I. Public
perception of food addition
…
Does it exist? Slide22
“Of course it exists!”
“Currently I’ve been binge eating everyday now for almost the past month. At first it only a couple days a week but now it’s daily. Eating until I feel like I’m going to die. For me it’s my coping mechanism for things in life. I believe it’s a slow for of suicide and the same goes for alcoholics and drug addicts. I just choose food as my drug.”
Anna B, 55. www.foodaddictsanonymous.orgSlide23Slide24
Self-help literature and Support groupsSlide25
“I
eat properly in front of other people and
then eat tons when
I get home and no one can see me
.”
“I
am fine for a while then I say just one bit of chocolate won’t hurt then it’s like I’ve blown it so who cares and I will start again tomorrow
.”
“I
eat in the car before I get home stuffing my face and if people knew what I
was
doing I couldn’t bear it
.”“I
get this craving in my head
for “it” and
I cant relax until I have had it
.”
“Once I start I can’t stop.”Slide26
Public perception of Food Addiction
…
Food addiction is real and foods can be addictive
Resonates with personal experience and relationships with food and eating – popular explanation for obesity
“Addiction” has entered into common usage but often mean any activity done to excess
E.G., TV, shopping, cell phone use, work, plastic surgery…
Metaphor vs. real medical condition
View is reinforced by reporting of Food Addiction in the media
1000-2000 new articles per month containing the term “Food Addiction.”
Chocolateaholic
!
Frequently reported with references to “junk food”, general obesity and illegal drug use
Lee
et al. 2013
PLOS
One
Slide27
II. Clinical
perspectives on
Food Addiction…
Does it exist? Slide28
Clinical says…
kinda
but not technically
Which addictions are “real” and which are
not, is extremely
important
for all clinicians: Psychiatrists, PCP’s, Psychologists, Counselors, Nurses,
RD’s
, Policy makers
,
Regulatory
bodies,
Pharmaceutical
and Insurance
Industries
•
APA’s
Diagnostic and Statistical Manual “Bible of Psychiatry
” (DSM) is most influential
point of reference for diagnosis of
addiction
• It evolves and changes: diagnosis validity can be questioned
DSM-I (1952, 130 pages, 106 mental disorders)…
Homosexuality was categorized as sociopathic personality disturbance
DSM-V (2013, 900 pages, 300 disorders) Slide29Slide30
No
mention of “addiction
”
“
Substance-Related Disorders” (1.Substance- abuse and 2.Substance- dependence
)
10
categories of
substance
Dependence
= 3+ symptoms (max 7) with significant impairment or
distress
With
or without physiological dependence (tolerance/withdrawal
)
Binge
Eating Disorder noted as requiring further
research
Food
Addiction NOT included
•
•
2000
-
2013
DSM-IV
TRSlide31
Substance-Related
and Addictive
Disorders
Continuum
of severity:
number of symptoms
Mild
= 2, Moderate = 4, Severe = 6
(
max 11
)
“
Dependence” seen as normal bodily response to a
substance
Gambling
disorder included as sole
behavioral addiction
BED recognized
as Feeding and Eating
Disorder
Internet
use disorder noted as requiring further
research
Food
addiction still NOT
included
May 2013
DSM-5Slide32
DIAGNOSIS: Substance
use disorders span a wide variety of problems arising from substance use, and cover 11 different
criteria (DSM-V):
1. Taking
the substance in larger amounts or for longer than the you meant to
2. Wanting
to cut down or stop using the substance but not managing to
3. Spending
a lot of time getting, using, or recovering from use of the
substance
4. Cravings
and urges to use the substance
5. Not
managing to do what you should at work, home or school, because of
substance
use
6. Continuing
to use, even when it causes problems in relationships
7. Giving
up important social, occupational or recreational activities because
of
substance use
8. Using
substances again and again, even when it puts the you in danger
9. Continuing
to use, even when the you know you have a physical or
psychological
problem that could have been caused or made worse
by
the substance
10. Needing
more of the substance to get the effect you want (tolerance)
11. Development
of withdrawal symptoms, which can be relieved by taking
more
of the substance.Slide33
“DSM…The
strength of the DSM has been “reliability” – to ensure clinicians use the same terms in the same way. The weakness is its lack of validity –common symptoms may have different causes and treatments. For research purposes, we need new/better ways of classifying mental disorders based on dimensions of
observable
behavior and neurobiological measures
.”
NIMH
Dir. Thomas
Insel
,
MDSlide34
Is
Food Addiction
a type of Binge Eating Disorder
?
Slide35
Binge Eating Disorder (BED):
finally
got its due in the DSM-V!
Binge eating disorder will now have its own category as an eating disorder. In the DSM-IV, binge-eating disorder was not recognized as its own disorder but rather was diagnosable under the category
Eating
Disorder Not Otherwise Specified (
EDNOS
).Slide36
DSM-V (Binge Eating Disorder)
Recurrent
episodes of binge eating. An episode of binge eating is characterized by both of the following:
eating, in a discrete period of time (for example, within any 2-hour period), an amount of food that is definitely larger than most people would eat in a similar period of time under similar circumstances
a sense of lack of control over eating during the episode (for example, a feeling that one cannot stop eating or control what or how much one is eating)
The binge-eating episodes are associated with three (or more) of the following:
eating much more rapidly than normal
eating until feeling uncomfortably full
eating large amounts of food when not feeling physically hungry
eating alone because of feeling embarrassed by how much one is eating
feeling disgusted with oneself, depressed, or very guilty afterwards
Marked distress regarding binge eating is present
.
The binge eating occurs, on average, at least once a week for three months.
The binge eating is not associated with the recurrent use of inappropriate compensatory behavior (for example, purging) and does not occur exclusively during the course Anorexia Nervosa, Bulimia Nervosa, or Avoidant/Restrictive Food Intake Disorder.Slide37
This revision is intended to increase awareness of the significant differences between BED and overeating.
According
to the American Psychological Association, “recurrent binge eating is much less common, far more severe, and is associated with significant
physical
and psychological problems.”Slide38
Food Addiction
may only be relevant to Binge Eating Disorder and not
necessarily relevant
to all
(most) individuals dealing
with
obesity
Truth is…we don’t know (limited evidence)
And…Food Addiction
may be the result obesity rather than a
cause
Gary
Taubes
(2010)…”People are fat not cause they eat too much, rather they eat too much because they are fat.”Slide39
III. Science perspectives
on
Food Addiction
…
Does
it exist? Slide40
Science says maybe…
been looking at this issue for sometime
Most
evidence for food’s
“addictive”
properties lies in the biological realm. Food and drugs of abuse exploit similar pathways in the
brain:
D
opamine
and
Opiate
systems
(
pleasure centers
).Slide41
Science cont
.
More
rewarding the food or drug is evaluated to be, the greater the release of extracellular dopamine, conversely when you block dopamine receptors reduce the reward value of sugar and drugs
PET
scans on
Subst. Abuse and
Obese pts show lower dopamine receptor levels…need more to get reward
Inverse
relationship between overeating and illicit drug use
Wang et al., 2004Slide42
The Rats…
Rats
more often choose water with sugar…when remove the sugar, withdrawal symptoms emerge.
But rat studies have clear limitations
Major
factors beyond palatability are …
availability
, visual appeal, economics, incentives,
alternative
reinforcement,
advertisements, and carbs are fun!Slide43
Drug
treatments for
Substance Abuse
and Obesity
?
Same
drugs used for SA could they be effective for Obesity?
Drugs
that improve dopamine functioning such as Naltrexone
Naltrexone blocks opiate rec that regulate release of dopamine has modest / inconsistent effects on alcohol dependence
Naltrexone with BED and Obesity early research
are scant.
Most drugs for Obesity is to impact appetite, urges, calorie absorption, moodSlide44
Sept, 2014.
Contrave
is a combination of two existing generic drugs in an extended-release formulation. One is naltrexone, which is used to treat alcohol and opioid dependence. The other is bupropion, which was approved under the name Wellbutrin to treat depression and under the name
Zyban
to help people quit smoking.Slide45
Comparison: Substance Abuse (SA),
Binge Eating Disorder (BED), and Obesity (FA?).
Great description of Addiction is that it’s a
“
Chronic
Relapsing
Disorder
”
Temporal course of relapse patterns is different
(
but measuring slightly different factors)
The
disorders have uniform but strikingly different patterns of relapse over time.
Wilson,
G.T
. (2010) Slide46
Temporal Course:
Substance Abuse
Nicotine
, Heroin, and Alcohol relapsed early post-treatment, and thereafter, relapse rates decelerated dramatically. The amount of accumulated time abstinent may be the transcending variable.
That
is, “abstinence begets abstinence.” Slide47
Temporal Course:
Binge Eating Disorder:
Binge
Eating Disorder is somewhat in the middle in that you get a significant reduction in binging early in treatment with mixed long-term results. However, no change in weight.
Striegel
-Moore, et al., 2010Slide48
Temporal Course:
Obesity
:
Obesity
initial weight loss is rapid and then slowly declines. Weight regain then begins and continues gradually until weight stabilizes somewhat below baseline levels. This temporal pattern is fairly independent of initial weight loss. Efforts to prevent relapse in the treatment of obesity via aggressive maintenance strategies have been able to slow the rate of regain over time but have failed to alter the basic accelerating function of the relapse curve.Slide49
Clear Issue in attempting to compare and establish validity is how we measure
these constructs:
Different
patterns will emerge for
sure
What are we measuring
?
Substance Abuse = drug relapse
BED = binge relapse
Obesity/FA = weight change and/or carb, sugar relapse?
What
do we need to
measure and treat if
we are to be
successful treating Food Addiction?
Slide50
Gearhardt
,
A.N
. et al., 2011Slide51
• One difference compared to BED is significant impairment
or
distress.
One difference regarding substances is the desire to get “high” from food/carbs/sugar?
Is it plausible that “DSM-V Substance Abuse criteria apply to food as a substance? Slide52
Substance use disorders span a wide variety of problems arising from substance use, and cover 11 different
criteria (DSM-V):
1. Taking
the substance in larger amounts or for longer than the you meant to
2. Wanting
to cut down or stop using the substance but not managing to
3. Spending
a lot of time getting, using, or recovering from use of the
substance
4. Cravings
and urges to use the substance
5. Not
managing to do what you should at work, home or school, because of
substance
use
6. Continuing
to use, even when it causes problems in relationships
7. Giving
up important social, occupational or recreational activities because
of
substance use
8. Using
substances again and again, even when it puts the you in danger
9. Continuing
to use, even when the you know you have a physical or
psychological
problem that could have been caused or made worse
by
the substance
10. Needing
more of the substance to get the effect you want (tolerance)
11. Development
of withdrawal symptoms, which can be relieved by taking
more
of the substance.Slide53
Psychometric measurement of “Food Addiction”
Development
of Yale Food Addiction
Scale (
YFAS
)
The
YFAS
is a measure that has been developed to
identify
those who are most likely to be exhibiting
markers and symptoms of substance
dependence
(criteria in DSM-IV-R) by the consumption
of high fat/high sugar foods
.
(larger amounts, can’t quit, time spent, impacts other areas of life, use despite
neg
consequences, tolerance, withdrawal, impairment)
Persistent
desire or un-successful effort to cut down endorsed by 71% sample
High
internal reliability:
α=.75
Convergent
validity with Eating Disorder and Binge Eating Scales
Discriminant
validity from Problem Alcohol Use
Scales
Gearhardt
,
A.N
. et al.,
2009Slide54Slide55Slide56
Challenges with
psychometric assessment of
“
Food Addiction” for
research
Assumption
that addiction is a stable, measurable concept
•
Assumption that Food Addiction exists to be
quantified
• No
clear threshold for ‘addictive’ versus ‘normative’ eating
behavior
•
Overlap with existing scales for severity of Binge Eating,
Disinhibition
, Emotional Eating, Food Craving, etc
.
•
Does it give any additional information
?
•
Is it necessary or helpful to ‘
medicalise
’ common
behaviors with
diagnostic labels? Slide57
More Challenges…
Problems
with food as “drug
”
Validity
of food addiction, misuse or abuse requires
new
categories of food –
(again it is essential
for
survival)
e
.g
.
“junk
”,
“hyper-palatable”, “high-sugar
” versus
“healthy
”,
“natural
”, “Wholesome
”
No
objective threshold for addictive potential of
foods (e.g., blood alcohol levels)
Bread
was found to be food “most difficult to control eating” in sample of obese “Food Addicts
”
Gearhardt
(2012) Yahoo Health
Slide58
If Food Addiction is cause of obesity it is
not following
other subst. addiction rates (stable/flat over time), thus calling into
question
Food Addiction’s
validity
and/or may
have to be evaluated and categorized
differently.
Alcohol
Statistics Teen Drug Use
Adweek
, 2015
www.monitoringthefuture.org
, 2014Slide59
RECAP
So…Does Food Addiction exist?
Yes it does!
How do we know? Cause we see it everyday, but it is not clean!
FA is unlikely to be the dominant (only) causal pathway to explain obesity, which is heterogeneous syndrome and profoundly environmental (“toxic food environment”).
Is
100% relapse prevention the goal
? Prevention of what?
Maintenance
strategies can slow but seldom prevent relapse.
Moreover
, the expenditure of time and money required to implement resource-intensive maintenance strategies renders them impractical for any large-scale, community-based intervention (Brownell, 2010).
Future Research –
YFAS
is key, and ask pts “yes or no” as FA.Slide60
Treatment Implications
Can
we market our program as a
Food Addiction treatment
program?
NOPE!...why?
Can
we call our patients food addicts?
(Isn’t
that a bit offensive
?)
NOPE!...why?Slide61
So, if Food Addiction does exist how to best treat?
Begs the question:
What is the key to treating addictions?
A. Abstinence
Or
B: Teach our patients how to eat and control their trigger, self-proclaimed addicted foods?
Slide62
Key
themes
for
treating Obesity/FA
:
Abstinence
Build history of alternative foods
Environment (home, work, drive-thru)
Build Control
Skills (impulses, emotional
)
Build a system/language about FASlide63
Treatment keys
:
1. Abstinence, Abstinence, Abstinence!
(abstinence for all addictions is key treatment goal*)
a.
pt
identifies self as a food addict
b. identify the “can’t control food”
c. minimum one year sobriety from that food (2yrs ideally)
d. track it, reward abstinence, make it public
*
National Institute on Drug Abuse, 2015Slide64
Treatment keys
:
2. Build
history of alternative
foods
a. changing biochemical nature of the body and brain
b. nutrition goes up cravings go down
c.
lowers valence of the “addiction food”Slide65
Treatment keys
:
3. Environment
(home
,
drive-thru
)
a. Environment is the true cause, then must be significant part of the solution!
b. The girl-scout cookie effect
c. kid’s
gotta
have it – no the don’t
d. Get it out of the house!
e. Eating out = weight gain
Slide66
Treatment keys
:
4. Build Control and More Control
a
.
positive self-talk
b
.
reinforce success
c
. get
back on plan if get off planSlide67
Treatment keys
:
5
. Skills, skills, skills
(impulses, emotional)
a
.
stress management
b
.
emotional eating
c
.
delay impulses
d. manage relationshipsSlide68
Treatment keys
:
6. Build
a
system
about
Food Addiction
a
.
review food addiction model
b
.
review abstinence
c
.
entire team on same page
d
.
not powerlessSlide69
Questions…?
Thank you