PPT-In The Name Of GOD bilateral adrenal hyperplasia

and subclinical hypercortisolism Soheila sadeghi whats the diagnosis Are there any aberrant hormone receptors in this patient Does the patient need to a surgery Will we do in her follow up. Shedding Light on an Ambiguous Subject. Grand Rounds: September 25, 2015. Stephanie Gibson, MD Lisa Knight, MD. PGY-3 Assistant Professor of Clinical Pediatrics USC Pediatric Endocrinology. The most common form of Congenital Adrenal Hyperplasia (CAH) results from a deficiency of which of the following?. T. esticular . F. eminization Syndromes. Dr. Ahmed . Hussain. A. . Mujamammi. Objectives. Adrenal . steroidogenesis. Congenital adrenal hyperplasia syndrome. Types. Biochemical characteristics. Clinical manifestations. . Hyperfunction. (. Hyperadrenalism. ). 1. . Hypercortisolism. (Cushing Syndrome) . - In clinical practice, most cases are caused by the administration of exogenous . glucocorticoids. (Iatrogenic). By: . Shifaa. ’ . Qa’qa. ’. adrenal glands:. cortex . medulla. ADRENOCORTICAL. HYPERFUNCTION. (HYPERADRENALISM). Cushing syndrome. hyperaldosteronism. . adrenogenital. or . virilizing. . syndromes. By: . Shifaa. ’ . Qa’qa. ’. adrenal glands:. cortex . medulla. ADRENOCORTICAL. HYPERFUNCTION. (HYPERADRENALISM). Cushing syndrome. hyperaldosteronism. . adrenogenital. or . virilizing. . syndromes. More common in subgroup of resistant HTN. May be progressive, intermittent or curable. Staging is similar to essential HTN. CLASSIFICATION. 1- Renal disorders:. Renal parenchymal ( acute or chronic GN, . Recommended screening for all incidentalomas. Test. Cushing's syndrome. Cushing's syndrome. 1 mg overnight dexamethasone suppression test. Pheochromocytoma. Pheochromocytoma. 24-hour urine collection for fractionated metanephrines and catecholamines. Adrenalectomy. due to APA. a case report. Presented . by:B.Rezvankhah.MD. February 2017 . Recommendations . for . screening primary hyperaldosteronism:. All . patients with the combination of hypertension . masses. Dr. Ahmadi. 99.12.18. Problem List:. HTN. Hypokalemia. High ARR. Bilateral Adrenal masses(3cm). High . cortisol . level (. positive. . overnight. dexamethasone suppression test . ). Suppressed ACTH. . Khayamzadeh. MD. Retrospective analysis of 70 patients with bilateral adrenal masses presenting to a single tertiary care endocrine centre from western India (2002–2015). . The most common . aetiology. AlHawari. , MD, FACE, ECNU. Consultant Endocrinologist. Assistant Professor of . Medicine. Leena. Al-. Nsour. (MD student). D. epartment of Internal Medicine. School of Medicine. University of Jordan. Dr. Tariq Aladily. tnaladily@ju.edu.jo. Department of Pathology. The University of Jordan. Second semester 2021/2022. Hypercortisolism . AKA Cushing syndrome. Can be exogenous (iatrogenic) or endogenous (less common). , MD. FRCPA,KSUF,EBP. Adrenal Glands. The . adrenal glands: paired endocrine organs: cortex and medulla: 4 . Three layers in the cortex:. Zonaglomerulosa. Zonareticularis. abuts the medulla. . Intervening is the broad . Problem list:. Abdominal pain . Flank pain. LBP. Weight loss. Headache. Ataxia. N/V. Bilateral adrenal mass. Adrenal insufficiency. Cerebellar mass. Old MI. HF. Anorexia. weakness. diagnostic approach.