PPT-MORPHOLOGIC PATTERNS OF ACUTE INFLAMMATION

DRAYSER HAMEED LEC3 Many variables may modify the basic inflammatory response these include 1 The nature and intensity of the injury 2 The site and tissues affected 3 The responsiveness of the . Acute And Chronic. The cardinal signs of inflammation. Vascular changes in the acute inflammatory process. Cellular changes in the acute inflammatory process. Peripheal. blood smear. Perhaps the simplest indicator of acute inflammation is an increase in the white blood cell count in the . Jahangir . Sadeghi. MD. 1391. 1) . Inflammation. 2) . Infection. We approach to . RED Eye . through pathology . Inflammation is a response of tissue to a noxious stimulus . . This noxious stimulus may be infectious or noninfectious. What is inflammation?. it’s a body response against ( MILD or MODERATE) injury , it’s a first line of defense. .. Not severe enough to cause cell death (necrosis).. . Inflammation is of two types:. . classification of acute inflammation. :. according to the inflammatory fluid exudates, . -Exudates . are any fluid that filters from the circulatory system into lesions or areas of inflammation. 1- . Cedar Mountain post acute rehabilitation center provide a therapeutic environment for our residents Yucaipa. Individualized treatment programs are developed with the interdisciplinary team of nursing, social services, Healthcare and dietary in consultation with your physicians. MSc . Etab. A. AL-. Mosawe. Fibrinous pericarditis.  is an exudative inflammation. The visceral pericardium (epicardium) is infiltrated by the fibrinous exudate. This consists in fibrin strands and leukocytes. Fibrin describes an eosinophilic (pink) network, amorphous. Leukocytes (. Professor. DEPT OF PATHOLOGY. SKHMC. Inflammation is a complex reaction to injurious agents such as microbes & damaged usually necrotic cells , that consists of vascular responses ,migration, & activation of leucocytes & systemic reactions. . Prof.Dr. . . Baydaa. . H. . Abdullah. Inflammation. Is a protective response. The body’s response to injury. Interwoven with the repair process. Inflammation. Types. Acute (sec, mins, hrs). Chronic (days, weeks, months, yrs). د هبة احمد غيدان. LEC . 2. Cells of acute inflammation:. 1. . Neutrophils. : . Most prominent cells in acute inflammation during first 6hrs -24hrs.. 2. . monocytes. ----macrophages: . Inflammation is a set of symptoms that include pain, swelling, heat, and redness of an affected organ or tissue. It is the natural way the body’s immune system responds to injury, infection or attack. Dr. . Shakir. H. Mohammed . Al_Alwany. LEC 2,3,4. Hyperplasia & Physiologic . Hyperplasia. HYPERPLASIA . Increase . in the number of cells . results in increase in size of the . organ. 1-Resolution: . restoration. to . histologic. and . functional. . normalcy. :. -. Neutralization. or . removal. of the various . chemical mediator. s. .. -. N. ormalization. . of vascular . permeability. DR. AYSER HAMEED. The major local manifestations of acute inflammation, compared to normal . . Vascular . dilation and increased blood flow (causing erythema and warmth). . . E. xtravasation . and deposition of plasma fluid and proteins (edema), and . INFECTION. The disease state caused by the invasion and multiplication of a pathogen. A disease producing microorganism in body tissue. Pathogens- bacteria, virus, fungi or parasites. The body respond to the invasion by forming antibodies in a series of physiological changes.