PPT-Rheumatoid Arthritis Description

Autoimmune disease that causes chronic inflammation of connective tissue Joint tissue is permanently affected but any connective tissue of the body might be involved Etiology Associated with the production of abnormal antibodies that attacks or attaches to the bodies own cells and tissues Presence of the antibody called Rheumatoid factor RF in the affected individuals blood is indicative of the disease. Clare Hunt FY2. The plan. Overview of Osteoarthritis and Rheumatoid arthritis. Case scenarios 1 and 2. Symptoms and signs. Clinical findings. Epidemiology/ Risk factors. Management . Case scenario 1. ARTHRITISINFORMATION SHEET 1800 011 041 www.arthritisaustralia.com.au 1633_English Template.indd 18/3/10 2:11:48 PM ARTHRITISINFORMATION SHEET 1800 011 041 www.arthritisaustralia.com.au 1633_Engl Stephanie Arrington. Joint Replacement . Research suggests that more than a million people a year are getting a total joint replacement. . More than half will be younger than 65. . Joint replacement are most typically an artificial hip or knee.. (RA. ). . RA . is a chronic, systemic inflammatory disorder of unknown etiology characterized by the manner in which it involved . joints. Progressive joint destruction and deformity leads to variable degrees of . Dr R B Kalia,. Additional Professor ,. Department of Orthopaedics. Leaning Objective. Clinical Features of RA. Investigations . Diagnosis. Indications for Surgery in Arthritis. Various procedures possible. Rheumatoid Arthritis . Inflammatory vs Noninflammatory Arthritis. ACR/EULAR Criteria for Diagnosing RA Before Joint Damage. Limitations to Classification of RA. Biomarkers in RA. Differential Diagnosis. FRCPEdin. ; . FRCSEdin. “A . chronic progressive disease causing inflammation in the joints and resulting in painful deformity and immobility, especially in the fingers, wrists, feet, and . ankles”.. FRCPEdin. ; . FRCSEdin. “A . chronic progressive disease causing inflammation in the joints and resulting in painful deformity and immobility, especially in the fingers, wrists, feet, and . ankles”.. Gregory Gardner, M.D.. Gilliland-Henderson. Professor of Medicine. Division of Rheumatology. University of Washington. Outline of Discussion. Pathophysiology. Clinical Features. Treatment Update. Perioperative Management. Page 1 Rheumatoid Arthritis People have long feared r heumatoid arthritis (commonly called RA) as one of the most disabling types of improved for many people with newly diagnosed (detected) RA . Abstract Objective: Division of Rheumatology, Department of Internal Medicine, University of Michigan, Ann Arbor, MI, USA Address for Correspondence: Rajaie Namas, Division of Rheumatology, Department Sonya Abraham. Senior Lecturer/Honorary . Consultant. Imperial College Healthcare NHS Trust. Medical . Student Lecture (Yr 5). May 2012. Rheumatoid Arthritis. Historical. Epidemiology. Disease Pathogenesis. Test. & Pregnancy Test. . Assistant Prof. . Dr. . Fitua. Al-. Saedi. Department of Clinical Laboratory Science. College of . Pharmacy. Lab . training . Rheumatoid . factor (RF) test. ?. A . rheumatoid factor (RF) test measures the amount of rheumatoid . withlow. low-density lipoprotein have higher coronary atherosclerosis burden,. greater plaque progression and cardiovascular event risk. George . Karpouzas. , MD. Harbor. -UCLA Medical . Center. and the Lundquist Institute.