PPT-Hemodynamic Disorders, Thrombosis, Shock
Author : briana-ranney | Published Date : 2018-10-06
Matthew Velkey mattvelkeydukeedu 454A Davison Duke South Green Zone Fluid Homeostasis Total extracellular tissue fluid volume hydrostatic pressure colloid
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Hemodynamic Disorders, Thrombosis, Shock: Transcript
Matthew Velkey mattvelkeydukeedu 454A Davison Duke South Green Zone Fluid Homeostasis Total extracellular tissue fluid volume hydrostatic pressure colloid osmotic pressure . Thrombi . are solid masses or plugs formed in the. circulation from blood constituents. Platelets . and fibrin . form the basic . structure. . . Their . clinical significance. results from . ischaemia. Assistant Professor. Dr Talib Hussein Kamoona . (CABM). Hematologist. /Medical Oncologist. Hemostasis. * The . intimal. surface of blood vessels throughout the body is lined by monolayer of . endothelial cells. circulation from blood constituents. Platelets . and fibrin . form the basic . structure. . . Their . clinical significance. results from . ischaemia. from local vascular. Obstruction. . or distant . Rudolph Virchow, 1821-1902. Vessel wall injury. Stasis of blood. Hypercoagulability of blood. Arterial Thrombosis - 1. Interruption of blood flow causes . ischemic necrosis of involved . organ/limb. Vessel wall injury (. Shock may be defined as a condition in which circulation fails to meet the nutritional needs of the cells and at the same time. A final common pathway for many potentially lethal clinical events (hemorrhage,trauma, burns, large MI, massive pulmonary embolism microbial sepsis) fails to remove the metabolic waste products. Shock. Decreased tissue perfusion . . inadequate O2 delivery . . . tiessue. ischemia. Common Clinical Features of Shock. 1. HYPOTENSION. SBP < 90 mm Hg. MAP <60 mm Hg. Acute decreased in SBP of > 40 mm Hg. Assistant lecturer :. Noor Wafaa Hashim. Thrombosis is the development of a ‘thrombus’ consisting of platelets, fibrin, red cells and white cells in the arterial or venous circulation. If part of this thrombus in the venous circulation breaks off and enters the right heart, it may be lodged in the pulmonary arterial circulation, causing . 1 Deep Vein Thrombosis (DVT). Somali. Dhiig oo Arooriye ah oo Hoos Jira (Deep Vein Thrombosis) Vein Thrombosis), waxa kale oo loo yaqaanaa DVT, waa xinjir dhiig oo ku samaysanta xididada dhiigga ee j and emboli Peter Nagy • A thrombus is any solid object developing from the blood in vivo within the vascular system or heart . • Thrombosis is hemostasis in the wrong place . • Major compo Practical session. Dr.Mohanad. . Total extracellular tissue fluid volume . = hydrostatic pressure – [colloid osmotic pressure + lymphatic drainage]. Mechanism of edema formation in heart , renal and liver failure. DR. SUVANKAR GHOSH. BANKERS HEART INSTITUTE AND RESEARCH CENTRE. Physiology . Myocardial Oxygen Supply and Demand. a,b. Goals of Hemodynamic Support During High-Risk PCI. Types of Hemodynamic Support in the . Diagnosis and Management in the Family Medicine Setting. Pascal Bastien, MD FRCPC. On behalf of Thrombosis . Canada. Pascal . Bastien . has received . fees/honoraria from the following sources:. Sanofi. George W. Vetrovec, MD, MACC, MSCAI. Professor Emeritus, VCU Pauley Heart Center. Virginia Commonwealth University. Richmond, Virginia . USA. Disclosures:. Consultant: Abiomed, Merck, FDA. What Hemodynamic Support is Available?. An embolus: is a detached intravascular solid, liquid, or gaseous mass that is carried by the blood to a site distant from its point of origin. . . Embolism: - . This is the passage and eventual trapping within the vasculature of any of a wide variety of mass objects..
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