PPT-HypertriglyceridemiA -Induced Pancreatitis

Richa Shukla PGY5 Faculty Mentor Dr Suneal Agarwal September 4 2014 HPI Reason for consult pancreatitis 31F G2P1 24weeks pregnant patient who presented as an outside hospital transfer for management of pancreatitis. Directed Readings . In the Classroom. July/August . 2012 issue of . Radiologic Technology. .. Instructions:. This presentation provides a framework for educators and students to use Directed Reading content published in . CLARK A. HARRISON, MD. GASTROENTEROLOGY CONSULTANTS. RENO, NEVADA. GALlSTONE. . DISEASE: . THE . BIG PICTURE. CHOLELITHIASIS. = stones or sludge in the gallbladder. CHOLEDOCHOLITHIASIS . = stones/sludge in the bile ducts. The exocrine pancreas constitutes 80% to 85% of the organ and is composed of . acinar. cells that secrete enzymes needed for digestion.. The pancreas normally arises from the fusion of dorsal and ventral . Missy Purcell. 3/5/10. The Pancreas. a brief overview…. The pancreas has both endocrine and exocrine functions. Exocrine pancreas is made up of pancreatic . acinar. cells and a duct system that opens into the proximal duodenum. MBBCh. , . MS, FRCR . Consultant radiologist . Riyadh Military Hospital. Dr. Ahmed . Refaey. Normal CT anatomy of the upper abdomen. Anterior . pararenal. space. Normal Anatomy by CT. Pancreas is located in the anterior . Margery Swan, Dietetic Intern U of MD College Park. Clinical Case Study at . MedStar. Harbor Hospital . Presentation Outline . General Patient Information. Medical History. Social History . Hospital Diagnosis . DR : HICHAM AL MAWLA . . . emj. . lebanon. Establish the . Diagnosis. of Acute Pancreatitis. Establish the . Etiology. of Acute Pancreatitis. Initial . Management. of Acute Pancreatitis. Linson. February 2021. RAD 4001. (Matthew Lambert, Mindy Wang, Wylie Foss). Clinical presentation. HPI. 43 M with HTN, DM, and EtOH abuse presented to ED with nausea and 10/10 abdominal pain for past 12 hrs. Pain is at the umbilicus radiating to the back. . Endocrinology. Wyoming Medical Center. Disclosure. Holdings in . Amarin Corporation. Objectives. Triglyceride rich lipoproteins (TRLs). Genetic causes of hypertriglyceridemia (HTG). Hypertriglyceridemia . Present . by:J.s.hosseini. Anatomy. Retroperitoneal Organ . Weighs 75 To 100 G . 15 To 20 Cm Long. Head. Neck. Body. Tail. . 2. . Inflammation . or infection of the . pancreas. Normally . digestive enzymes secreted by the pancreas are not . ezzawi. Gastroenterology department . BMC. objectives. Anatomical and physiological back ground.. Difinition. and types of pancreatitis.. Clinical features and complications of disease and their management. . Sam Nourani MS MD. Digestive Health Associates. 5.12.2016. Reno, NV. Acute Pancreatitis. Acute inflammatory process of the Pancreas. Mortality ranges:. 3% for interstitial edematous pancreatitis. 17% for pancreatic necrosis. Mohammed . Fouad. . Mousli. Surgical Demonstrator. Acute pancreatitis. INTRODUCTION . :.  Acute pancreatitis is an acute inflammatory process of the pancreas. It is usually associated with severe acute upper abdominal pain and elevated blood levels of pancreatic enzymes. Most cases are associated with alcoholism or gallstones, but the precise . Due to premature activation of pancreatic enzymes within the pancreas, leading to a process of . autodigestion. . . Anything that injures the . acinar. cell and impairs the secretion of zymogen granules, or damages the duct epithelium and thus delays enzymatic secretion, can trigger acute pancreatitis. Once cellular injury has been initiated, the inflammatory process can lead to pancreatic .