PPT-Lead exposure and toxicity in adults

Ulrike Luderer MD PhD MPH Professor Division of Occupational and Environmental Medicine Dept of Medicine University of California Irvine Historical uses of lead by Romans Face powders rouges and . Susan Ripple, MS, CIH, Fellow. Global Manager – Industrial Hygiene. The Dow Chemical Company. Midland, MI. Value of Occupational Exposure Bands (OEB) to supplement other authoritative OELs. Value of OEBs to the industrial hygiene process (ERAM). Alex Battistini. Pharmacology of digoxin. Clinical Uses. Overview. Introduction. Toxicity and management. 1. 2. 3. 4. Digitalis: Cardiac glycoside that occurs naturally in plants. Its medical use dates back from the 18. Asst.Prof. . Dr. Ghaith Ali Jasim. PhD Pharmacology. . OPIATES: Alkaloid found as a natural product in . Papaver. . somniferum. (Poppy. ), un-ripened seed pods. . . . NARCOTIC: Broader group of agents and is predominantly used by law enforcement to designate a variety of controlled substances with . st. century. There is no innocuous level of exposure. Background blood lead level . is lower. Lead is not more hazardous now. Current information is consistent with past findings. But effects can be seen at a lower level. Pb. Emmanuel Obeng-Gyasi MPH, Ph.D.. Source—National . Institutes of Health . Background: Lead in gasoline. Major Source of Lead exposure. Belief . lead had the ability to boost engine performance by:. sporocyst. , redia and cercaria larva of . Fasciola . gigantia. .  . Dr. . Kumari. . Sunita. . Post Doctoral (UGC), New Delhi. Department of Zoology. D.D.U. Gorakhpur University, Gorakhpur. (UP) India 273009. OCCUPATIONAL MEDICINE SPECIALIST. PHYSIOLOGY. Regulation of electrolytes . Maintenance of acid-base balance. Regulation of BP . Remove wastes from the blood. Reabsorption. of H2O,G,AA. Produce hormones. —.  Mercury exists in elemental, inorganic, and organic forms, all of which may be toxic . .The . toxic manifestation depends on the form of exposure. This topic reviews sources of mercury exposure, the clinical manifestations, and the treatment and prevention of mercury toxicity.. Absorption of toxins in the GIT is by passive diffusion – this process requires that the substance cross cellular barriers . Toxins that are not absorbed from the GIT do not produce systemic effects but may produce local effects – diarrhea, bleeding and malabsorption of nutrients . ng closure and severe pain and this is However, there have been occasions where e, with the eyes closed and extremely recovery starts, it is a rapid processresult in a significant recovery 4. The HO QUICK TM 30 µg/dL should be seen by a doctor. Some harmful effects of lead are permanent.Requirements employers must follow to stop take-home lead exposure:Test workplace air for lead and blood le QUICK TM30g/dL should be seen by a doctor Some harmful effects of lead are permanentRequirements employers must follow to stop take-home lead exposureTest workplace air for lead and blood lead levels ToxCast data to predict acute toxicity. Dan Wilson, PhD, DABT. Science Leader – Cheminformatics. The Dow Chemical . Company. ddwilson@dow.com. (989)636-0712. EPA’s Computational Toxicology Communities of Practice. Exposure: Leg pain on walking, outcome: knee pain. Exposure: knee pain, outcome: . M15 . Polyarthrosis. Exposure: . M15 . Polyarthrosis. . , outcome: knee pain. Exposure: knee pain, outcome: . other specific joint derangements.