PPT-Chris Dobson Renal disease: AKI vs CKD

What is acute kidney injury AKI Hyperkalaemia What is chronic kidney disease CKD Caseoff So what is an AKI Significant deterioration in renal function over hoursdays How would you define that clinically. 13–18% of all people admitted to hospital. Format for the Session. 8.10 Acute . Kidney Injury (AKI). R. ecognise AKI/potential for AKI. Distinction from . chronic renal . failure. Establish underlying pathophysiology (causes). on . Chronic . Kidney . Disease. Maria Ferris, MD, MPH. PhD. Associate Professor. Director of the Pediatric Dialysis and Transplant Services. UNC Kidney Center, Chapel Hill, NC, USA. Akash. and his fan club. What do they do……………….?. IKA AGM. Dublin July 2014. Cardiac-Renal Centre. Cork University Hospital. Renal Services in 6 Hospital Groups. (*/^ – shared access contracted Units; [] planned Units). in Neonates. Jordan M. Symons, MD. University of Washington School of Medicine. Seattle Children’s Hospital. Seattle, WA. 9. th. International . pCRRT. Conference on . Pediatric Continuous Renal Replacement Therapy. Lowell Dilworth-Chemical Pathologist. CKD. CKD and Bone disease. Mineral . metabolism controlled by kidneys. , intestine, parathyroid glands and . bone. The . kidney plays a critical role in mineral homeostasis regulation and, therefore, renal disease exerts widespread effects on the skeleton and soft . Namita Singh, MD, FASN. Year 2021. DISCLOSURES. I have no financial relationships with commercial interests to disclose.. This presentation does not include discussion of off-label or investigational use. . . Structural . or functional abnormalities of the kidneys for . >. 3 months. . from early to late-stage disease, . Chronic Kidney Disease. as . manifested by either:. 1. Kidney . damage, with or without decreased GFR, as . Hyperkalemia, Metabolic Acidosis, Malnutrition, Depression & Acute Kidney Injury. Andrew . Narva. , MD, FASN & . Amy Barton . Pai. , PharmD, MHI, FASN, FCCP, FNKF. Andrew . Narva. , MD, FASN. In CKD and HD patients dysfunction of . immu. ne . sy. stem . a. lterations. . belonge. to . . involv. ing. . i. nnate. and . a. daptive. . i. mmunity. in T and B lymphocytes (. Reduced . T regulatory cells and memory cells and increased innate cells). . Heather Mascio, DO. Introduction. Chronic Kidney Disease (CKD) affects around 14% people in the United States. Individuals with CKD are 10 times more likely to die of CV disease than the general population and dialysis dependent individuals are at even higher risk (around 50% of them will die of CV causes). What we will cover:. AKI. CKD. Hyperkalaemia. Dialysis. UTI. Pyelonephritis. Case 1. . 92 year old lady with poor oral intake having routine bloods done. Her urea and creatinine come back raised compared to bloods yesterday. . Dr Jon Murray. Consultant . Nephrologist. James Cook University Hospital. Acute Kidney Injury v Chronic Kidney Disease. Creatinine 1250 . . mol. /l. What are the priorities?. How is the patient? Is this single or multi-organ failure?. Acute kidney injury. Acute kidney injury (AKI), previously referred to as acute renal failure, is not a diagnosis; rather it describes the situation where there is a sudden and often reversible loss of renal function. UHNM -CKD . 11/10/2017. Proportion of CKD 4 & 5 patients (not on RRT) with RA anaemia guideline(+/- ESA if available) .Spot audit December . 2016 . In December 2016, 222 patients with CKD 4&5,not on RRT, had a Hb checked. Of those patients 168 (76%) met RA anaemia guideline. 54 patients in the audit (24%) had Hb of less than 100..