PPT-VASCULITIS ( Part 2) Mustafa E. Al-Badran

Author : mackenzie | Published Date : 2023-05-20

CABM FIBMS Internist amp Rheumatologist Large Vessel Vasculitis Takayasu arteritis Mikito Takayasu  1860    1938  Japanese ophthalmologist CLINICAL FEATURES

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VASCULITIS ( Part 2) Mustafa E. Al-Badran: Transcript


CABM FIBMS Internist amp Rheumatologist Large Vessel Vasculitis Takayasu arteritis Mikito Takayasu  1860    1938  Japanese ophthalmologist CLINICAL FEATURES Diagnosis By angiography which reveals Coarctation occlusion ampaneurysmal dilatation. (. Grace). Melanie Kong. Jeremy Ng. Tina Ngo. Causes and Treatment of Granulomatosis with . Polyangiitis. PHM142H1. October 11, 2016. PHM142 Fall . 2016. Coordinator. : Dr. Jeffrey Henderson. Instructor: Dr. David Hampson. ANSWER THE QUESTIONS. DIRECTIONS: . Read the questions and carefully answer each one. . For multiple choice questions, you must write a . detailed reason explaining how/why. you chose your answer. . vasculitides. . Dr. . Aditya. . Jindal. 5/8/11. The . vasculitides. are a set of related disorders . characterized by . blood vessel . inflammation . leading to tissue or . end-organ injury. Mark A. McQuillan MD FACP SFHM. June 15, 2016. DISCLOSURES. None. Blake Roessler MD. David A. Fox, Division Chief. W. Joseph McCune. Ruba Kado MD. Josef Holoshitz MD. UMHS Rheumatology. William . Repaskey. . Rheumatica,Giant. Cell . Arteritis. , Wegener’s . Granulomatosis. , . Polyarteritis. . Nodosa. What is . Vasculitis. ?. Disease characterized by inflammation of blood vessel walls, leading to altered blood flow through obstructed walls. This causes ischemia and tissue damage. . By: Dr. Donna Braham. Objectives. Classify Vasculitis. Know when to consider Vasculitis. Rule out mimickers of Vasculitis. Diagnose cutaneous small vessel vasculitis – In . particular Henoch–. Schönlein. Prof. Dr. Gamze MOCAN KUZEY. NEU. Department of Pathology. Vascular Lesions. Atherosclerosis. Arteriolosclerosis. Varicose veins. Trombophlebitis. Lymphedema. Aneurysms. Dissection. Vasculitis. Vasculitis. Takayasu’s arteritis, giant cell arteritis and primary CNS angiitis all have granulomatous angiitis. Takayasu’s arteritis:. This affects the aortic arch, main arterial trunks and descending aorta including the carotid and subclavian arteries. The clinical features of the various . vasculitides. are protean and largely depend on the vascular bed affected (e.g., CNS vs. heart vs. small bowel). . Besides the findings referable to the specific tissues involved, the clinical manifestations typically include constitutional signs and symptoms such as fever, . Rheumatology winter clinical symposium 2019. Nina . narasimhalu. , md . CASE PRESENTATION. 33-year-old male presents with bilateral lower extremity edema, progressive . myalgias. , generalized weakness, periorbital swelling and rash for 2 weeks. OF AN . INFLAMMATORY CASE. DR NASIR FAROOQ BUTT. ASSISTANT PROFESSOR. DEPARTMENT OF MEDICINE . KING EDWARD MEDICAL UNIVERSITY. MAYO HOSPITAL LAHORE . INVESTIGATIONS . & MANAGEMENT. CASE . SCENARIOS. PRESENTER- Dr. B. PRADEEP.. CHAIR PERSON- Dr. T. SMRUTHI.. Vasculitis are group of disorders characterized by . immunoinflammatory. injury to vessel wall . leading to aneurysm, bleeding, stenosis, occlusion, thrombosis, embolism and ischemia. . CABM FIBMS. Internist and Rheumatologist . Epidemiology. . Ethnic differences . Knee OA higher in Africa, China, Japan and the Indian subcontinent . Hip O.A higher in European countries. Pathophysiology. CABM FIBMS. College . of Medicine\ University of Basrah. Objectives. What is cerebellum. The Functions of  cerebellum. Anatomical areas of cerebellum. Input (afferent) Pathways to the Cerebellum. Output (Efferent) Signals from the Cerebellum.

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