intro Functions of the Skin Protects body against bacteria Prevents fluid loss Regulates temperature Initiates immune response Sensation Aesthetic amp psychological importance ID: 913224
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Slide1
Burns
Tasneem Anagreh
Slide2intro
Functions of the Skin: Protects body against bacteria. Prevents fluid loss. Regulates temperature. Initiates immune response. Sensation. Aesthetic & psychological importance.
Slide3Burns are tissue injuries resulting from direct contact with
flames , hot liquids, gases, surfaces, caustic chemicals, electricity, or radiation. Most commonly, the skin is injured, which compromises its function as a barrier to injury and infection and as a regulator of body temperature and fluid loss. Like trauma, mortality
from burns occurs in
a
bimodal pattern
:
i
mmediately
after the injury or
weeks later
from sepsis and
multiorgan
failure.
Slide4Burns are classified into:
A. Thermal burns:B. Non-thermal burns:1. Electrical injuries2. Radiation injuries3. Cold injuries4.Chemical burns
Slide51.THERMAL BURNS:
Heat causes coagulative necrosis of tissue, by coagulation of the cellular proteins, this type of necrosis is characterized by, preservation of the shape of the tissue involved , the temperature that causes burn is greater than 45 degrees. Thermal burns is classified into:
A
. Dry heat (direct flame burn) direct exposure to fire.
B
. Moist heat (scald burn), exposure to hot liquids,
C.Contact
burn. contact with hot metals.
D.Friction
burns.
Slide6The burn wound and surrounding tissues classically have been described as a system of several circumferential zones
radiating from primarily burned tissues, as follows: 1. Zone of coagulation - A nonviable area of tissue at the "epicenter" of the burn. 2. Zone of ischemia or stasis
( Injury zone
): Surrounding tissues (both deep and peripheral to the coagulated necrotic areas), which are
not devitalized initially but, due to
microvascular
insult, can progress irreversibly to necrosis over several days if not resuscitated properly
.
3
. Zone of
hyperemi
a - Peripheral tissues that undergo vasodilatory changes due to neighboring inflammatory mediator release but are not injured thermally and remain viable.
Slide7ASSESSMENT OF THE SEVERITY OF BURN ( depth and percentage):
1. The depth of burn damage (degree): depends on the quantity of heat (temperature and duration of exposure), determinns the local management and outcome of the burn wound 2.The surface area involved in burn, This is the
percentage of the burned area to the total body surface area
.
This
determins
the systemic management
,
complications,
and
prognosis
( mortality rate).
Slide8Classification of the depth of burn injury:
1.First degree burn, thermal necrosis is limited to the epidermis, clinically there is pain, and erythema, it takes 1-6 days to heal and leaves no scars. 2.Second degree burn (
partial
thickness), necrosis of the
epidermis and varying depth of the dermis
, characterized clinically by
pain
( due to irritation of the dermal sensory nerves),
erythem
a,
blisters( bullae),
the burned area is wet with exudate (weeping), blanching denoting intact dermal vascularity, and preservation of skin elasticity. It takes 1-4 weeks to heal and leaves variable degrees of scarring.
3
.
Third
degree burn (
full
thickness), necrosis of the whole skin (
epidermis and dermis) and its skin appendages
, clinically there is an
eschar
which is simply -
the burned necrotic skin
,
it is
insensitive, leathery, hard, inelastic, and may show
thrombosed
dermal vessels
. It takes
months
to heal
and
leaves
significant scarring
, to avoid scarring it should be skin grafted.
Slide9Slide101st degree:
Slide11Second-Degree:
Slide12Second-Degree Burn with Sloughing:
Slide132nd:
Slide14A) superficial partial-thickness scald 24 hours after injury. The dermis is pink and blanches to pressure.
(b) At 2 weeks,the wound is healed but lacks pigment. (c) At 3 months, the pigmentis returning.
Slide153rd:
Slide16(a) A full-thickness burn on admission just prior toescharotomy. The wound is wrapped in cling film while in transit.
(b) Excision of the same full-thickness burn, down to healthy fat.
Slide172)ESTIMATION OF THE PERCENTAGE OF BURN:
This determines the prognosis, and the systemic management of burn, especially fluid management. There are three methods of TBSA estimation: 1. Rule of nines: the body is divided into 11 nines; Head &neck (9%), Upper limbs. (9% each), Anterior trunk (18%), Posterior trunk (18%), Lower limbs (18% each) and the remaining 1% for the genitals.
2. As the rule of nines is
not very accurate especially in children
, in whom the percentage of the surface area of the head (20% at birth) and decreases with age, while the percentage of the surface area of the lower limbs increases with age, it is more accurate to follow the
specially made charts available in the burns units and in the emergency rooms
.
Slide18Slide19Slide20Slide21I. ASSESSMENT
Mechanism of injury Identify burn source, duration of exposure, time of injury, and environment.-----------------------------------------------------------------Primary survey (ATLS)
treated as victims of multisystem
trauma:
1)Airway
:
Supraglottic
tissue edema
progresses over the first 12 hours and can obstruct the airway rapidly.Inhalation injury should be suspected if the patient was burned in an enclosed structure or explosion.
Slide22Physical signs include hoarseness, stridor, facial burns, singed facial hair, expectoration of carbonaceous sputum, and presence of carbon in the oropharynx
.minor inhalation injury humidified oxygen.
Major
injuries
endotracheal intubation
.(+ Bronchodilators can be given to treat bronchospasm whereas nebulized heparin and N-
acetylcysteine
can limit cast formation)
Slide23Carbon monoxide poisoning :Carbon
monoxide exposure is suggested by a history of exposure in a confined space with symptoms of nausea, vomiting, headache, mental status changes, and cherry-red lips.The arterial carboxyhemoglobin level is obtained as a baseline.Mx supplemental oxygen (40-minute half-life with 100% oxygen via nonrebreathing
mask
).
oxygen
therapy should
continue until normal levels are achieved.
Slide242. Breathing is evaluated for effort, depth of respiration, and auscultation of breath sounds.
Wheezing or rales suggest either inhalation injury or aspiration of gastric contents.3. Circulation. Circulatory support in the form of aggressive and prompt fluid resuscitation.4.Exposure. -------------------------------------------------------------------------
2) Burn-specific secondary
survey
Slide25When to transfer to a burn center?1.
Partial-thickness burns greater than 10% BSA. 2. Any full-thickness burn. 3. Burns that involve the face, hands, feet, genitalia, perineum, or major joints. 4. Any inhalation, chemical, or electrical injury.5. Burn injury in patients with
pre-existing medical
conditions that could complicate management, prolong recovery, or affect
mortality
6.
Burned
children in hospitals without qualified personnel
or equipment for the care of children.
8
. Patients
requiring specialized rehabilitation, psychological suppor
Slide26Management :
A. Emergency room 1. Resuscitation in patients with major injury. a. Oxygen: A 100% oxygen high-humidity facemask for those with possible inhalation injury .b. Intravenous access:
All patients with burns of
15% or greater
BSA require intravenous fluids.
Two 16-gauge
or larger peripheral venous catheters should be started immediately to provide circulatory volume support
.
Slide27Fluids :hemodynamically, the depletion of the intra-vascular compartment will cause hypovolemic shock, and its severity depends on the percentage of burn.
Practically, burn shock is seen in adults with burns greater than 15-20% and in children with burn more than 10-15% .
Slide28a. Consensus formula(Parkland formula):-
The estimated crystalloid requirement for the first 24 hours after injury is calculated on the basis of patient weight and BSA burn percentage.Lactated Ringer solution volume in the first 24 hours = 2 to 4 mL × %BSA (second-, third-, and fourth-degree burns only) × body weight (kg).One-half of the calculated volume is given in the first 8 hours after injury, and the remaining
volume is
infused over the next 16 hours
with
adjustments being made as clinical conditions
and size/depth of burns evolve
.
Slide29Example: An adult weighing 70 Kgm
, with 50% burn, should receive : 4x 70x 50= 14000 ml of Ringer lactate (Half of this amount is administered in 8 hours , and the remaining half over the next 16 hours).b. Colloid-containing solutions for resuscitation remains an ongoing debate. Some studies show they should be avoided as an intravenous therapy until after the first 24 hours postburn
,
at which time
capillary leak
diminishes. Some providers
option
for a compromise,
augmenting resuscitation with albumin later in the first 24 hours and in patients with large
burns
.
Slide30B. Additional Management:1. Foley
catheter(hourly monitored) : a minimum urine production rate of 1 mL/kg/hr in children (weighing ≤30 kg) and 0.5 mL/kg/hr in adults is the guideline for adequate intravenous infusion.2. Nasogastric tube
:
insertion
with low suction
is performed if patients are
intubated or develop nausea, vomiting, and abdominal distention consistent with
adynamic
ileus
.
3. Continuous pulse oximetry .4. Laboratory evaluation : baseline CBC, type and crossmatch, electrolytes and renal panel,
β-
human chorionic
gonadotropin (in women),
arterial
carboxyhemoglobin
,
ABG
,
and
urinalysis
,
ECG.
5.
Tetanus prophylaxis
Slide31C. Wound Care :1.
Early irrigation and debridement are performed using normal saline and sterile instruments to remove all loose epidermal skin layers. Nonviable tissue in the burn wound should be debrided early because the dead tissue provides a bacterial medium putting the patient at risk for both local and systemic infections2.Topical antimicrobial agents
:
gram-negative organisms
, particularly
Pseudomonas
aeruginosa
, and fungi
are the most common causes of invasive burn wound
sepsis.
(note:
Systemic antibiotics are not administered prophylactically)A biopsy of suspicious eschar and underlying unburned tissue is required to diagnose an invasive infection.
Wound
infection is defined by
more than
10^
5 organisms per gram of tissue.
Treatment
requires infected
eschar
excision and appropriate topical/systemic
antibiotic therapy
Slide32Slide333. Dressings:a. Biologic dressings include
allograft (cadaver skin) and xenograft (pig skin).B. Synthetic dressings (Biobrane , Trancyte , Integra)-----------------------------------------------------------------------
D. Operative
Management:
Escharotomy
may
be necessary in
full
-thickness
circumferential burns of the neck, torso, or extremities when increasing tissue edema impairs peripheral circulation or when chest involvement restricts respiratory efforts.
Slide34E. Nutrition. Severe burns induce a hypermetabolic
state proportional to the size of the burn up to 200% the normal metabolic rate.The daily estimated metabolic requirement (EMR) in burn patients can be calculated from the Curreri formula: EMR = [25 kcal × body weight (kg)] + (40 kcal × %BSA) Note supply
1.5 to 2
g/kg of
protein per
day
.
1.
Enteral
feedings
2. Total parenteral nutrition3. Daily vitamin supplementation(ascorbic acid, nicotinamide,riboflavin,thiamine,zinc)
Slide35F. Critical Care Considerations:1. Stress ulcer
prophylaxis (e.g., H2 blockers or proton-pump inhibitors).2. Venous thromboembolism (VTE).3. Sepsis. In patients who survive the first 24 hours after injury, burn sepsis is the leading cause of mortality!!!
Slide36Slide37Thank you ^^
*Reference: ( bailey &love + The Washington manual of surgery)