PPT-Hepatic Encephalopathy
Author : phoebe-click | Published Date : 2016-10-15
By Michelle Russell Case Study Presentation NUR 4216L 12412 1 Objectives Understand the pathophysiology of hepatic encephalopathy Recogonize the signs symptoms
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Hepatic Encephalopathy: Transcript
By Michelle Russell Case Study Presentation NUR 4216L 12412 1 Objectives Understand the pathophysiology of hepatic encephalopathy Recogonize the signs symptoms Understand relevance to clinical setting and patient scenarios. -FULMINANT HEPATIC FAILURE-................AETIOLOGY ................................................................................................................................................. EEG Part II - Encephalopathy. Teneille Gofton. September 2013. Objectives. To review the . subhairline. EEG changes seen with encephalopathy. To discuss specific EEG findings in encephalopathy. To outline basic EEG patterns associated with HIE. The Progression of Chronic Liver Disease. Staging Is Based on the Amount of Hepatic Fibrosis, . O. r Is It?. Scoring Methods . for. . Stag. ing Hepatic Fibrosis. Hepatic Function vs Hepatic Fibrosis. Other Brain Diseases. James S. Kennedy, MD, CCS, CDIP. President and Chief Medical Officer. CDIMD – Physician Champions. Smyrna, Tennessee. jkennedy@cdimd.com. – (615) 479-7021. Disclosures. This presentation is designed to provide accurate and authoritative information in regard to the subject matter covered. The information includes both reporting and interpretation of materials in various publications, as well as interpretation of policies of various organizations. This information is subject to individual interpretation and to changes over time. . Assistant . Professor of Medicine. Division of Gastroenterology and Hepatology. University . of Washington. Hepatic Encephalopathy. Last . Updated: . August 21, 2013. Disclosure Slide. Dr. Landis receives research support from the following:. Diagnostic and clinical manifestations. Hepatic disorders. Pancreatic disorders. The nursing processes. Functions of the liver. It receives nutrients-rich blood from GIT. It stores, transforms these nutrients into chemicals to be used by the body. Rand . Al-shayeb. Selena . Abboud. Definitions. Hypoxia or Anoxia: . A partial (Hypoxia) or complete (Anoxia) lack of oxygen in the brain or blood.. Hypoxemia: . D. ecreased arterial concentration of Oxygen. . There are three concepts of hepatic lobule. 1.Classical hepatic lobule. 2.Portal lobule. 3.Liver . acinus. Classical hepatic lobule. It is the structural unit of liver which appears hexagonal in outline. DR. SANJANA BHAGWAT. MODERATOR : DR. SHIMPA SHARMA. . DR. R MANE. TOPICS DISCUSSED : . Hepatic encephalopathy. Spontaneous bacterial peritonitis. Hepatorenal. syndrome. Hepatopulmonary. syndrome. Dr. Muntadher Abdulkareem Abdullah. M.B.Ch.B,CABM,FIBMS,FIBMS(GE.&HEP.). Acute Liver Failure:. Acute liver failure describes the clinical syndrome of severe impairment of liver function Within 6 months of the onset of symptoms, which include:. Journal of Current Med ical And Applied Sciences, 2015, July, 7(2), 133 - 138 . IJCMAAS,E - ISSN:2321 - 9335,P - ISSN:2321 - 9327. Page | 1 33 Prec ipitating Factors of Metabolic Ence Hepatic encephalopathy (HE) is abnormal function of the brain caused by severe liver disease. It occurs when the liver is sick and no longer able t hepatic vein variants. To the best of our knowledge, theremorphologic classification of the AHV, or the factorspredictive of the presence of an AHV, using multidetectorspatial, contrast, and temporal 99 Neurology AsiaMarch 2015 myelin. Coexistence of polyneuropathy may cause some patients with Wernickes encephalopathy to experience limb ataxia and dysarthria. It is estimated that 19% of pati
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