PPT-ACUTE DECOMPENSATED HEART

FAILURE 2010 HFSA GUIDELINES BART COX MD FACC ASSOCIATE PROFESSOR OF MEDICINE UNIVERSITY OF NEW MEXICO SCHOOL OF MEDICINE DIRECTOR ADVANCED HEART FAILURE PROGRAM DISCLOSURES NONE OBJECTIVES. Functions of the liver . Synthesis . (. gluconeogenesis. , . glycogenesis. , protein, cholesterol, . coag. factors, bile) . Breakdown . (drugs and toxins, . glycogenolysis. , . bilirubin. metabolism, ammonia). RHEUMATIC FEVER. Acute, recurrent inflammatory disease . in children 5-15 yrs old (rarely adults). Characterized by inflammatory changes in the fibrous tissue of different organs mainly the heart and joints.. (Lo Studio CARRESS-HF). Aurelio Limido. UO Nefrologia e Dialisi. AO Fatebenefratelli e Oftalmico. Milano. 2012;367 . 24 december. . :2296-304.. The acute . cardiorenal syndrome (type 1). is defined as worsening renal function in patients with acute decompensated heart failure. . W24. SOF/VEL. >. 18 years. Chronic HCV infection. Genotype 1 to 6. Naïve or treatment-experienced. No prior treatment with . NS5A or NS5B inhibitor. Child-Pugh B cirrhosis **. No . hepatocellular. About these slides. These slides give a comprehensive overview of the EASL clinical practice guidelines on the management of decompensated cirrhosis. The guidelines were first presented at the International Liver Congress 2018 and are published in the Journal of Hepatology. Salman Siddiqui. Professor of Airways Disease. EMBER Chief Investigator and WS4 lead. Work Strand 4 . Clinical . disease cohorts- 'breathomic' discovery and clinical validation & objective . - . ‘Breathomic. Shock may be defined as a condition in which circulation fails to meet the nutritional needs of the cells and at the same time. A final common pathway for many potentially lethal clinical events (hemorrhage,trauma, burns, large MI, massive pulmonary embolism microbial sepsis) fails to remove the metabolic waste products. Occurs as a result of complex interaction between GROUP A . Streptococcus . a susceptible host and environment.. An abnormal immune response to a GAS infection leads to an acute inflammatory illness that most commonly affects the joints, heart ,brain or skin.. ≥ 18 years. Chronic HCV infection. Genotype 1 or 4. HCV RNA > 10 000 IU/ml. Treatment-naïve or pre-treated. w. ith PEG-IFN ± RBV. Cirrhosis (> 14.5 . kPa. on . FibroScan. ). Portal hypertension or liver . Praktiknjo . M. 1. ,.  . Clees C. 1. , . Pigliacelli A. 2. , . Fischer S. 3. , . Jansen C. 1. , . Lehmann J. 1. , . Pohlmann A. 1. , . Lattanzi B. 2. , . Krabbe VK. 1. , . Strassburg CP. 1. , . and CLINICAL ASPEC. Nahar. . Taufiq. RS DR . Sardjito. Yogyakarta. Carditis. . Acute . Rhematic Fever (RF) and. Rhematic heart disease (RHD). Consequences of . Haemodynamic. When to Intervention?. Department of internal medicine #1 with cardiovascular pathology course. History and introduction. Epidemiology and aetiology. Pathogenesis. Clinical features. Diagnosis . Management. Old and New….. ASTRAL-4. Source: Curry MP, et al. N Engl J Med. 2015;373:2618-28.. Treatment Naïve and Treatment Experienced, Phase 3. Sofosbuvir-Velpatasvir in Decompensated HCV Cirrhosis. ASTRAL-4: Study Features. DR. SANDESH .P. GOJE. . ASSISTANT PROFESSOR . ( KAYACHIKITSA). SVAMCH, CHANDRAPUR. Inflamed pancreas. Acute pancreatitis . Introduction and definition . Acute pancreatitis is a sudden . inflammation.