PPT-Atherosclerosis is a progressive chronic inflammatory disease and the main contributor

The exact mechanisms underlying these diseases are still not fully understood but increasing evidence suggests that dietinduced monocytes and macrophage dysfunction plays a major causal role in the conversion of acute into chronic inflammation and . By Joshua Bower. Easter Revision 2014. J.Bower@warwick.ac.uk. Distinguish arteriosclerosis, atherosclerosis and arteriolosclerosis. ARTERIOSCLEROSIS – generic term meaning ‘hardening of the arteries’ which encompasses the other 2. Response. A review. The 3 Lines of Defense. FIRST line: Barrier protection. non-specific (treats . each pathogen the . same). acts to prevent the pathogen from getting into the body. skin, mucous membranes, tears, sweat. Dr. Omar Mansour. Consultant Colorectal & Laparoscopic . General Surgeon. Assistant Professor of General Surgery. Al-. Balqa. Applied University. FRCS FRCSI FEBC MSc MRCSI. What IS IBD. ? . Inflammatory bowel disease (IBD) is a group of inflammatory conditions of the colon and small intestine. . Dr. Omar Mansour. Consultant Colorectal & Laparoscopic . General Surgeon. Assistant Professor of General Surgery. Al-. Balqa. Applied University. FRCS FRCSI FEBC MSc MRCSI. What IS IBD. ? . Inflammatory bowel disease (IBD) is a group of inflammatory conditions of the colon and small intestine. . Why is Atherosclerosis known as a heart disease?. Atherosclerosis is when there is a buildup of plaque on artery walls. This creates a smaller passageway for blood to go through, increasing blood pressure. Overview: Why is it Called “Heart Disease”. Atherosclerosis is a type of arteriosclerosis (the hardening of the arteries.) . A thrombus (blood clot) can form because of the buildup of plaque.. Diseases that can develop from atherosclerosis : angina (chest pain), carotid artery disease, coronary artery disease, and kidney disease.. NORMAL ARTERIAL WALL. PROCESS OF ATHEROSCLEROSIS. RISK FACTORS OF ATHEROSCLEROSIS. COMPLICATIONS OF ATHEROSCLEROSIS. DEFINITION. Atherosclerosis. is defined as a progressive disease characterized by the accumulation of lipids and fibrous elements in the large arteries.. A review. The 3 Lines of Defense. FIRST line: Barrier protection. non-specific (treats . each pathogen the . same). acts to prevent the pathogen from getting into the body. skin, mucous membranes, tears, sweat. Featured Clinical Research – Late Breaking Clinical Trials . Residual Inflammatory Risk and Residual Cholesterol Risk Among Statin Treated Atherosclerosis Patients With and Without Chronic Kidney Disease:. 1 Frequent Diagnostic Pitfalls James Michael Mitchell, MD Problems encountered •Pathologist blinded or does not understand reason(s) for biopsies •Lack of clinical information •Signs an Atherosclerosis. Shaesta. . Naseem. Vessel wall structure. Arteriosclerosis. It literally means "hardening of the arteries with loss of elasticity. ”. . . Three patterns:. Atherosclerosis. :. The . When it occurs in the . heart. , it may cause angina, MI and sudden death; in the . brain. , stroke and transient . ischaemic. attack; and in the . limbs. , . claudication. and critical limb . ischaemia. Pathophysiology. Atherosclerosis is a chronic inflammatory disease. . Atherosclerosis . begins with fatty streak which is an accumulation of lipid laden foam cells in the intimal layer of the artery. . JR2 . chronic meningitis is inflammation of the meninges, with signs and symptoms persisting for at least 4 weeks.. Diagnosed when a characteristic neurological syndrome exists for more than 4 weeks and associated with a persistent inflammatory response in the cerebrospinal fluid(white blood cell count>5).