PPT-The Inflammatory Response

A review The 3 Lines of Defense FIRST line Barrier protection nonspecific treats each pathogen the same acts to prevent the pathogen from getting into the body skin mucous membranes tears sweat. lakeshorelearningcom response response response total recognized Writing Letters Key correctly recognized WR wrong response DK didn57557t know NR no response Key correctly written WR wrong letter written DK didn57557t know 57513 Lakeshore Alphabet C I-3 Fall 2012. Marieke Kruidering, Ph.D.. Pharmacology in I-3. Week 2. Anti-inflammatory. and immunosuppressive. drugs 1 & . 2. TBL. Week 4. Antibacterial drugs 1. Week 5. Antibacterial drugs 2. Antibacterial drugs . Group International . is an amalgamation of . Open Access publications.  and worldwide international science conferences and events. Established in the year 2007 with the sole aim of making the information on Sciences and technology ‘Open Access’, OMICS Group publishes . Soft Tissue Inflammatory. Multiple modalities. X-ray. Ultrasound. CT. MRI. Nuclear Medicine. Soft Tissue Inflammatory. Multiple modalities. X-ray. Evaluate for mass. Evaluate for soft tissue air. Evaluate for bone involvement. Response. A review. The 3 Lines of Defense. FIRST line: Barrier protection. non-specific (treats . each pathogen the . same). acts to prevent the pathogen from getting into the body. skin, mucous membranes, tears, sweat. Disturbances of growth. Inflammation and repair. Immune response. Non Adaptive. Degeneration. Neoplasia. . Dysplasia. Necrosis. I. NFLAMMATION. Definition. -it’s a body response against injury , it’s a first line of defense.. Literally thousands of inflammatory . dermatoses. have been described. . acute lesions . last from days to weeks and are characterized by inflammatory infiltrates (usually composed of lymphocytes and macrophages rather than neutrophils), edema, and variable degrees of epidermal, vascular, or subcutaneous injury. . Fred . Tabung. , . PhD(c. ), MSPH. Department . of Epidemiology and Biostatistics. Cancer Prevention and Control Program. Arnold . School of Public . Health, USC. 4. th. Annual . USC . Center for Research in Nutrition and Health Disparities, Annual Symposium. Dr Catherine Gwynne. Consultant Rheumatologist. Torbay and South Devon NHS Foundation Trust. What is Arthritis?. A condition causing pain or inflammation in a joint, often accompanied by structural changes. C. ount . P. redicts Outcomes after Cardiac . S. urgery in Infants . Katie Gilbert, Ray Lowery, . Sunkyung. Yu, Lauren . Retzloff. , Cynthia Smith, Suzanne Welch, . Donna Wilkin, . Caren. . Goldberg, . These are caused . by . infection or . immunological factors.. These conditions are . characterized by . inflammation of the synovial membrane . with resultant . changes in the . synovial fluid.. Systemic signs may include. . be used in PHL 322. Mouse . Rat . Frog . Don’t worry you will see it again in . Exam. Today . Last. . lab. . . Inflammation. It . is a reaction of living tissue to . an injury. . . 1- pathogenic organism . CARDINAL SIGNS OF INFLAMMATION. Rubor (redness). Tumor (swelling). Calor (heat). Dolor (pain). Functio laesa (loss of function). FACTORS INVOLVED IN PROTECTIVE RESPONSES AND BODILY REPAIR. Inflammatory reaction. NSAIDS) . Mr. Suhas Agey. Assistant Professor. Department of Pharmacology. HRPIPER, Shirpur. Contents . Introduction. Classification. NSAIDs Mechanism. Actions of NSAIDs. Pharmacology . of Aspirin. The nonsteroidal anti inflammatory drugs (NSAIDs) and antipyretic analgesics are a class of drugs that have analgesic, antipyretic and anti inflammatory actions..