PPT-Inflammation and the Spine: Mediators to Modulators

J Scott Bainbridge MD Denver Back Pain Specialists LLC wwwDenverBackPainSpecialistscom Overview Nociceptive vs Neuropathic Pain Vs Inflammatory Pain Lines blurred Stimuli and Mediators of Inflammation. Inflammation and Repair. Learning Objectives. List characteristics and clinical manifestations. Acute inflammation. Types of exudates: serous, purulent, fibrinous, hemorrhagic. Describe possible outcomes of an inflammatory reaction and its harmful effects. Inflammation lecture 1. Dr. H Awad. FRCPath. First trimester 2016/17. Contact details. Email: . h_awad@ju.edu.jo. Office hors: 11-12 Mondays. Office: third floor , JUH. Lectures will be available on my website page a day before they are delivered.. J. Matthew Velkey. matt.velkey@duke.edu. 454A Davison, Duke South (green zone). Cellular Adaptation to Injury or Stress. Injury or Stress. Increased demand. Decreased stimulation or lack of nutrients. , MD. .. Department of Pathology. Faculty of Medicine, . Naresuan. University. Learning Objectives . อธิบายความหมายของการอักเสบชนิดต่างๆ ได้. LEC.2. Binding of a particle to . phagocytic. leukocyte receptors initiates the process of active . phagocytosis. .. During engulfment, extensions of the cytoplasm (. pseudopods. ) flow around the particle to be engulfed, eventually resulting in complete enclosure of the particle within a . د هبة احمد غيدان. LEC . 2. Cells of acute inflammation:. 1. . Neutrophils. : . Most prominent cells in acute inflammation during first 6hrs -24hrs.. 2. . monocytes. ----macrophages: . Dr. . Shakir. H. Mohammed . Al_Alwany. LEC 2,3,4. Hyperplasia & Physiologic . Hyperplasia. HYPERPLASIA . Increase . in the number of cells . results in increase in size of the . organ. Faculty of Medicine. Al-Balqa’ Applied University. Email: ali.alkhader@bau.edu.jo. Overview. A protective response . …involving . host cells, blood vessels, and . molecules. . …for the elimination of:. Dr.V.Shanthi. Associate professor, Pathology. Sri Venkateswara Institute of Medical Sciences, Tirupathi. CHEMICAL MEDIATORS. Inflammatory mediators are the substances that initiate and regulate inflammatory reactions. Lecture 2. Dr. Mohanad Al-Hindawi. 2022. Cellular changes of acute inflammation.. A critical function of inflammation is to deliver leukocytes to sites of injury (recruitment), especially those cells capable of phagocytosing microbes and necrotic debris (e.g., neutrophils and macrophages).. Inflammation. . is. a . response. of . vascularized. . tissues. to . infections. and . tissue. . damage. . that. . brings. . cells. and . molecules. of . host. defense from the . circulation. Introduction.. What cause ankylosing spondylitis?. The ankylosing process.. Signs and symptoms.. Diagnosis.. Treatment.. Medication.. Introduction.... Previously known as-. Bechterew’s disease.. Bechterew’ syndrome.. Associate Professor. University of Kansas School of Medicine. Inflammation. Definition. . “. Inflammation is a reaction of a tissue and its microcirculation to a pathogenic insult. It is characterized by the generation of inflammatory mediators and movement of fluid & leukocytes from the blood into extravascular tissues.”. . . . A dynamic process that lasts from a few minutes to a few years. Depending on: . . the extent of the injury . . the type of injury . . the vascularity of the tissue.. Protective Role .