PPT-Reduced-dose vs. 10-unit insulin dosing in hyperkalemic ED patients
Author : amber | Published Date : 2023-07-18
ACKNOWLEDGEMENTS Special thanks to Dr Tom Jelic Dr Aaron Guinn Carol Cooke THE QUESTION In adult emergency department patients with hyperkalemia does a reduceddose
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Reduced-dose vs. 10-unit insulin dosing in hyperkalemic ED patients: Transcript
ACKNOWLEDGEMENTS Special thanks to Dr Tom Jelic Dr Aaron Guinn Carol Cooke THE QUESTION In adult emergency department patients with hyperkalemia does a reduceddose insulin administration strategy decrease the risk of hypoglycemia while still effectively lowering serum potassium when compared with the standard 10unit dosing strategy. . Dosing Advisor. 7/9/2012/. hf. Initial dose recommendation based on height, weight, and . creatinine. clearance (calculated from . SCr. ). Obesity factored into dose calculation. Serum . creatinine. G. lucose . C. ontrol. Mary Catherine . MacSween. MD FRCPC. The Moncton Hospital . Case DM. 74 year old male DM1 x 40 years.. MDI with . Levemir. 17 units . hs. and . NovoRapid. at meals.. Pre-op instructions take half usual . Dept of Pediatric Endocrinology. Stony Brook Children’s Hospital. Timing and Duration of Action of Insulin Preparations. Onset. Peak. Duration. Rapid Acting. Lispro. (. Humalog. ). 15-30. 30-90 min. Dept of Pediatric Endocrinology. Stony Brook Children’s Hospital. Timing and Duration of Action of Insulin Preparations. Onset. Peak. Duration. Rapid Acting. Lispro. (. Humalog. ). 15-30. 30-90 min. Insulin and Incretins: the perfect Partnership? Stephen Colagiuri, MD Professor of Metabolic Health Boden Institute of Obesity, Nutrition and Exercise University of Sydney Sydney, Australia Moderator CRRT BackgroundWhena patient is initiated on CRRT antimicrobial therapy often requires adjustment to ensure adequate drug concentrations are achievedCVVHDremoves solutes including drugs via diffusion Symbols D dose dosing interval CL clearance Vd volume of distribution ke elimination rate constant ka absorption rate constant F fraction absorbed bioavailability K0 infusion rate T duration By Dr Motamed. Fellow of Endocrinology and Metabolism. Endocrine Research Center. 1. 10 January 2012. Pathophysiology of Type 2 Diabetes. Peripheral Tissues. (Muscle). Glucose. Liver. Impaired insulin secretion. Dr. . . Ghadiri. , MD. Assistance professor of endocrinology. Shahid. . Sadoughi. . University of Medical Sciences . 2. The importance of glycemic control in minimizing complications related to diabetes has been well established in type 1 diabetes . Diabetes Educator. Program Coordinator . Diabetes Resource Center . Hopkinsville, KY.. Disclosure. to Participants. Notice of Requirements For Successful Completion. Please refer to learning goals and objectives. Yazan . karim. 09/03/2020. Objectives. Discuss background & etiology about acute DM management. Differentiate different points of care for ICU vs Non-ICU patients. Identify clinical goals for diabetic patients in an inpatient setting(ADA guidelines). Glucose homeostasis in blood. Gluconeogenesis in liver (making glucose from non-carbohydrate sources, e.g., lactate). Oral antidiabetic medication. Insulin . sensitisers. Biguanides. Thiazolidinediones. Diabetes consultant. University of Exeter. A.T.Hattersley@exeter.ac.uk. Why do HNF1B patients get diabetes?. Do all patients get diabetes?. When is diabetes likely?. How is the diabetes best treated?. . Created by: Andrew . Fratoni. , . PharmD. IV Aminoglycosides. Gentamicin. Tobramycin*. Amikacin*. *Restricted Antibiotics. Aminoglycoside Drug . Class Principles. Good bactericidal activity against gram negative organisms.
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