PPT-Inflammatory

M yofibroblastic Tumors In Children Dr Ibtisam Al Shuaili Clinical Fellow Pediatric Imaging Alberta Children Hospital Calgary Canada Disclosure No financial disclosure Introduction Pathogenesis. I-3 Fall 2012. Marieke Kruidering, Ph.D.. Pharmacology in I-3. Week 2. Anti-inflammatory. and immunosuppressive. drugs 1 & . 2. TBL. Week 4. Antibacterial drugs 1. Week 5. Antibacterial drugs 2. Antibacterial drugs . Soft Tissue Inflammatory. Multiple modalities. X-ray. Ultrasound. CT. MRI. Nuclear Medicine. Soft Tissue Inflammatory. Multiple modalities. X-ray. Evaluate for mass. Evaluate for soft tissue air. Evaluate for bone involvement. Diet. General. Fresh . is best . Fruits . and vegetables from all parts of the color spectrum. Omega-3 . fatty acids . Whole . grains. Ginger and curry . (. turmeric) for . anti-inflammatory effects . Response. A review. The 3 Lines of Defense. FIRST line: Barrier protection. non-specific (treats . each pathogen the . same). acts to prevent the pathogen from getting into the body. skin, mucous membranes, tears, sweat. Robert Murphy, MD. Clinical Neurophysiology Fellow. Disclosures. none. Outline. Definitions of Idiopathic Inflammatory Myopathies. Therapeuti. c Options. Polymyositis. Dermatomyositis. Necrotizing Myopathy. Frank H. Wians, Jr., PhD, MT(ASCP), MASCP, DABCC, FACB. Professor of Pathology. Texas Tech University of the Health Sciences Center El Paso. and the. Paul L. Foster School of Medicine. Technical Director, Clinical Chemistry, University Medical Center (UMC) El Paso. Literally thousands of inflammatory . dermatoses. have been described. . acute lesions . last from days to weeks and are characterized by inflammatory infiltrates (usually composed of lymphocytes and macrophages rather than neutrophils), edema, and variable degrees of epidermal, vascular, or subcutaneous injury. . Dr. Omar Mansour. Consultant Colorectal & Laparoscopic . General Surgeon. Assistant Professor of General Surgery. Al-. Balqa. Applied University. FRCS FRCSI FEBC MSc MRCSI. What IS IBD. ? . Inflammatory bowel disease (IBD) is a group of inflammatory conditions of the colon and small intestine. . Dr. Omar Mansour. Consultant Colorectal & Laparoscopic . General Surgeon. Assistant Professor of General Surgery. Al-. Balqa. Applied University. FRCS FRCSI FEBC MSc MRCSI. What IS IBD. ? . Inflammatory bowel disease (IBD) is a group of inflammatory conditions of the colon and small intestine. . Frank H. Wians, Jr., PhD, MT(ASCP), MASCP, DABCC, FACB. Professor of Pathology. Texas Tech University of the Health Sciences Center El Paso. and the. Paul L. Foster School of Medicine. Technical Director, Clinical Chemistry, University Medical Center (UMC) El Paso. Dr. . . Kumari. . Anjana. Assistant Professor. Deptt. . of Veterinary Pharmacology & Toxicology. Bihar Veterinary College, Bihar Animal Sciences University, Patna. NSAIDs . are heterogeneous group of drugs having analgesic, anti-inflammatory and antipyretic effect.. A review. The 3 Lines of Defense. FIRST line: Barrier protection. non-specific (treats . each pathogen the . same). acts to prevent the pathogen from getting into the body. skin, mucous membranes, tears, sweat. These are caused . by . infection or . immunological factors.. These conditions are . characterized by . inflammation of the synovial membrane . with resultant . changes in the . synovial fluid.. Systemic signs may include. NSAIDS) . Mr. Suhas Agey. Assistant Professor. Department of Pharmacology. HRPIPER, Shirpur. Contents . Introduction. Classification. NSAIDs Mechanism. Actions of NSAIDs. Pharmacology . of Aspirin. The nonsteroidal anti inflammatory drugs (NSAIDs) and antipyretic analgesics are a class of drugs that have analgesic, antipyretic and anti inflammatory actions..