PDF-REVIEW CB receptor antagonists new discoveries leading
Author : debby-jeon | Published Date : 2015-04-29
Kirilly X Gonda and G Bagdy 134 Department of Pharmacodynamics Semmelweis University Budapest Hungary Department of Clinical and Theoretical Mental Health K tv lgyi
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REVIEW CB receptor antagonists new discoveries leading: Transcript
Kirilly X Gonda and G Bagdy 134 Department of Pharmacodynamics Semmelweis University Budapest Hungary Department of Clinical and Theoretical Mental Health K tv lgyi Clinical Center Semmelweis University Budapest Hungary Group of Neurochemistry Semme. Teaser. Zak Fallows. 2013-07-03. http://datb.mit.edu. pharmacology@mit.edu. 1. How the Brain Works. You have about 100 billion brain cells, which are called neurons.. Each neuron has about 1,000 connections, called synapses. (This number is extremely variable.). Pharmacodynamics. Pharmacodynamics. The study of the biochemical and physiologic effects of drugs and the molecular mechanisms by which those effects are produced . The study of what drugs do to the body and how they do it . SOM Graduate Education Office. Paul MacDonald. Martin Snider. Robert Petersen. Monica Montano. George . Dubyak. Diana Ramirez-Bergeron. Joseph . Williams. Deborah . Noureddine. Malana. . Bey. Dan . Korgan. An Enrichment For High-Achieving Students. 2015-2016. What is Discoveries Unlimited or D.U.?. . Discoveries Unlimited is designed as an academically enriched time for high achievers. It is a wonderful opportunity to increase the quality of learning, the quantity of challenging experiences and extend the curriculum. Discoveries Unlimited classes meet for 45 minutes twice a week. Students will be challenged to expand thinking skills in these areas. Mike Harlos MD, CCFP(PC), FCFP. Professor and Section Head, Palliative Medicine, University of Manitoba. Medical Director, WRHA Adult and Pediatric Palliative Care. http://palliative.info. Objectives. β-adrenergic antagonists are commonly used to treat hypertension, angina, tremor, migraine, and panic attacks.. ACTION. β-adrenergic antagonists competitively antagonize the effects . of . catecholamines. in FEVE . Fluoropolymer. Resin Technology – Performance . Beyond . Weatherability. ” . Bob Parker . AGC Chemicals. October . 29, 2014. Recent Discoveries in FEVE Technology . Outline:. Explanation of FEVE . DR. ISHOLA I.O.. DEPT OF PHARMACOLOGY, THERAPEUTICS AND TOXICOLOGY. CMUL. ADRENERGIC TRANSMISSION. Adrenergic transmission is restricted to the sympathetic nervous system. Norepinephrine. is the transmitter at post-ganglionic sympathetic nerves – except sweat glands . Pain. . Pain. persists Pain persists. or increases or increases. 1. . Non. -. opioid. ± adjuvant. 2. Weak . opioid. ± non-. opioid. ± adjuvant. 3. Strong . opioid. 14/3/2020. 1. Learning Objectives 1. Cholinomimetics. • Cholinomimetics: explain how (a) directly acting and (b) indirectly acting cholinomimetic drugs produce their biological actions and state why the former are more selective in their actions. Graded Dose-response Relationships. Agonist . drugs mimic the action of the original endogenous ligand for the receptor (for example, isoproterenol mimics norepinephrine on β1 . receptors . of the heart). The magnitude of the drug effect depends on the drug concentration at the receptor site, which, in turn, is determined by both the dose of drug administered and by the drug’s pharmacokinetic profile, such as rate of absorption, distribution, metabolism, and elimination. . Know the characteristic pharmacokinetics & dynamics of different classes of antiemetic drugs.. Identify the selective drugs that can be used according to the cause of vomiting.. Learn the adjuvant antiemetics... Antiemetic drugs. Prof. . Alhaider. Nausea and vomiting may be manifestations of many conditions . However, . a useful abbreviation for . remembering causes of nausea and vomiting is VOMIT.. V. estibular . Graded Dose-response . Relationships:. Agonist drugs mimic the action of the original endogenous ligand for the receptor (for example, isoproterenol mimics norepinephrine on β1 receptors of the heart). .
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