PPT-UNDER PRESSURE : Intracerebral Hemorrhage
Author : faustina-dinatale | Published Date : 2018-11-02
Rhonda Finnie DNP APRN AGACNPBC ANVPBC Baptist Health Neurosurgery Arkansas Baptist Health Neurology Stroke Symposium October 26 2017 I have no disclosures Identify
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UNDER PRESSURE : Intracerebral Hemorrhage: Transcript
Rhonda Finnie DNP APRN AGACNPBC ANVPBC Baptist Health Neurosurgery Arkansas Baptist Health Neurology Stroke Symposium October 26 2017 I have no disclosures Identify risk factors etiology and clinical features associated with intracerebral hemorrhage. Ngo Minh . Triet. , MD. Department of Neurology, . University of Medicine and Pharmacy. Introduction. Intracerebral. . hemorrhage (ICH) results from the rupture of an . intracerebral. vessel. .. 10-35% percent of all strokes (in USA). . Hemorrhagic stroke vs. ischemic stroke. Intracerebral hemorrhage. Occurs from rupture of cerebral vessels . Can be a result of high blood pressure. Ischemic Stroke. . Caused by the development of a thrombus and/or embolus. Fred V. Plapp MD PhD. Pathology and Laboratory Medicine. Kansas University Medical Center. Financial Disclosure. Educational Objectives. Discuss intracerebral hemorrhage prevalence and etiologies. Review anticoagulation by warfarin and anti-Factor . ICH. MECHANISMS OF INTRACEREBRAL. HEMORRHAGE:. Hypertension . Vascular Malformation s . Intracranial . Tumors . Bleeding Disorders, . Antico. . agulants. , and . Fibrinolytic. Treatment . Cerebral Amyloid . Clinical picture of TIA. Neurologic deficits are . completelly and spontaneously reversible. . in less than 24 hours. No signs on CT. TIA is usually characterized by focal neurological symptoms. . There are 2 main groups of TIA’s symptoms:. Pathophysiological. . Facts :. For maintained normal brain function :. Continuous blood flow-15% of cardiac output. High oxygen requirement- 20% of total body O2 consumption . Maintenance of glucose. Wendy C. Ziai, MD, MPH, FAHA. Johns Hopkins Medical Institutions. Division of Neurocritical Care. Baltimore, MD, USA. June 2, . 2017. 2. October 14, 2013. ANA 2013. Presenter Disclosure Information. FINANCIAL DISCLOSURE. Brain Abscess Following Intracerebral Hemorrhage: A Case ReportJin Kyung Oh, M.D., Young Joo Kim, M.D., Eun Deok Chang, M.D. Tel. 82-31-820-3599 Fax. 82-31-846-3080 E-mail: violet2@catholic.ac.kr Eastern Journal of Medicine 18 (2013) 185-194 Original Article Table 2. Scores of the intracerebral hematomas according to their signal characteristics on DWI and ADC maps. The i Content. : . Vineeta. Singh, MD; Craig Williamson, MD; Jennifer . Erklauer. , MD. Slides. : Nicholas A. Morris, MD. Presenter:. Your name. Your institution. . . . . Conflicts:. No conflicts. Intracerebral. hemorrhage . accounts for . 8-13% . of all strokes and results from a wide spectrum . of disorders, . is more likely to result in death than ischemic stroke.. . Intracerebral. hemorrhage and accompanying edema may disrupt or compress adjacent brain tissue, leading to neurological dysfunction. Substantial . S. urvivors for Stroke . P. revent. I. on. and . RE. covery. Mean reduction of 9/4 mm Hg. Of course you want to join the first ICH prevention study – . ASPIRE. – in recent NIH history! . This is a great trial to contribute to an active clinical practice question and a wonderful way to build out your StrokeNet trial portfolio!. - . Cerebrovascular. disease. denotes . brain disorders caused by pathologic processes involving the blood vessels. . - The three . main pathogenic mechanisms are:. 1. Thrombotic occlusion of vessels. This is an abnormal accumulation of fluid in interstitial tissue spaces or body cavities. . A. Causes of edema. 1. . Increased hydrostatic pressure is exemplified by CHF.. 2. . . Increased capillary permeability occurs in inflammation or with injury .
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