PPT-Inflammation PRACTICAL

2 I Acute Inflammation Foundation Block Pathology Dept KSU Pathogenesis of Exudation The process of exudation aided by endothelial cell contraction and vasodilation which typically is most pronounced in venules. Acute And Chronic. The cardinal signs of inflammation. Vascular changes in the acute inflammatory process. Cellular changes in the acute inflammatory process. Peripheal. blood smear. Perhaps the simplest indicator of acute inflammation is an increase in the white blood cell count in the . What is inflammation?. it’s a body response against ( MILD or MODERATE) injury , it’s a first line of defense. .. Not severe enough to cause cell death (necrosis).. . Inflammation is of two types:. . classification of acute inflammation. :. according to the inflammatory fluid exudates, . -Exudates . are any fluid that filters from the circulatory system into lesions or areas of inflammation. 1- . . Manar. . Hajeer. , MD, . FRCPath. Plasma derived mediators of . acute inflammation. Complement . system. Coagulation . and . Kinin. systems. Complement system. Complement . components are , C1 – C9, circulate in the blood in an inactive form. Its purpose is to . localize. ,. . eliminate. the injurious . agent, . remove . damaged tissue . and . replace. it with healthy new tissue (. repair. ). . It consist principally of vascular changes associated with leukocyte infiltration and systemic reaction.. Kristen Dostie. April 8, 2015. Cadmium (Cd). Toxic heavy metal . Found naturally in environment. Extremely long biological half-life . of 18-33. . years. Intoxication leads to a variety of adverse effects. MSc . Etab. A. AL-. Mosawe. Fibrinous pericarditis.  is an exudative inflammation. The visceral pericardium (epicardium) is infiltrated by the fibrinous exudate. This consists in fibrin strands and leukocytes. Fibrin describes an eosinophilic (pink) network, amorphous. Leukocytes (. Zeinab Ghorbani. Ph.D. Candidate in Nutritional Sciences, School of Nutritional Sciences and Dietetics, Tehran University of Medical Sciences (TUMS).. Introduction. Top 10 level-4 causes of disability in GBD 2016 (global, both sexes, all ages). د هبة احمد غيدان. LEC . 2. Cells of acute inflammation:. 1. . Neutrophils. : . Most prominent cells in acute inflammation during first 6hrs -24hrs.. 2. . monocytes. ----macrophages: . . damage. in . the. CNS in . the. ART . era. Tricia . Burdo. , . Ph.D. .. Associate. Professor. Temple University. Lewis Katz School . of. . Medicine. Philadelphia, PA, USA. COI . Disclosure. . Supporting the Immune System . 1. Table of Contents.  . Module 1:. What is Inflammation? . Inflammatory markers. Signs and symptoms . Chronic disease risk .  . Module 2: . Fighting Inflammation . Acidity/alkalinity . Faculty of Medicine. Al-Balqa’ Applied University. Email: ali.alkhader@bau.edu.jo. Overview. A protective response . …involving . host cells, blood vessels, and . molecules. . …for the elimination of:. DR. AYSER HAMEED. The major local manifestations of acute inflammation, compared to normal . . Vascular . dilation and increased blood flow (causing erythema and warmth). . . E. xtravasation . and deposition of plasma fluid and proteins (edema), and . 1 IN THE HEALING PROCESS by Keith Wassung 2 The role of inflammation in the process of healing has been misunderstood for many years. Recent neurological and immunological research has shed light