PPT-Haemoglobin

Haemoglobin has ONE job to carry oxygen around the body It is a large protein with a quaternary structure What are proteins Proteins are made up of C H O N and some S and P Transport proteins such as haemoglobin carry oxygen. Mellitus Abbreviated Report of a WHO Consultation Starch, Glycogen and Cellulose. Starter. Haemoglobin summary on . kerboodle. Exam Practice. Haemoglobin is a protein.. A) What is meant by the quaternary structure of a protein? (1 mark). B) The tertiary structure of haemoglobin allows it to carry oxygen. Explain how. (2 marks). London Speciality School of Paediatrics. August 2014. Return to Acute Paediatrics. Aims. To . reassure you that not much has . changed. To give you some gems so you can sound knowledgeable from the first ward . Introduction. Iron is an essential nutrient needed by every human cell. Iron deficiency is the most common deficiency disease worldwide. More than one billion people have iron deficiency and about 700 million people have iron deficiency . PALI Haematology Session. 3. rd. October 2012. Christopher Mullen. Introduction. Classification. The Hypochromic . Anaemias. Megaloblastic. and other macrocytic . anaemias. Haemolytic . anaemias. Genetic disorders of haemoglobin. 1 Regular blood transfusions are needed to . maintainthe. . haemoglobin. over 10 g/. dL. at all times. . This usually requires 2-3 units every 4-6 weeks. Fresh blood. , filtered to . remove white . MAY9,L9o8.1]THETRAININGOFMENTALDEFECTIVES.[1J097possiblybeduetotheviscosityoftheblood.-ParkesWebershowedthistobemuchabovethenaturalinhiscases,andtheobsetvationhasbeenconfirmed.Butthiscouldonlyactbydel MD FRCEM. Emergency Medicine Consultant . Lister Hospital, Stevenage. Cyanosis. Cyanosis. What is Cyanosis?. Cyanosis. Abnormal bluish discolouration of the skin and mucous membranes.. Increased amounts of bluish-coloured haemoglobin – deoxygenated haemoglobin.. Abeer. . Anwer. Ahmed. Sickle cell disease is a group of . haemoglobin. disorders . resulting from . the inheritance of the sickle β‐. globin. gene. . Sickle cell disease (SCD) is . an. . autosomal. مؤيد . علو. ش. Lec. . 2. Thalassaemias. Objectives:. 1-Define thalassemia and its main types(. β. and . α. ). 2- Know the . genetic . lesions of each of . β . and . α. thalassemia. 3-Know the difference between thalassemia minor and major.. Ass. Prof. . Abeer. . Anwer. Ahmed. Sickle cell . anaemia. Ass. Prof. . Abeer. . Anwer. Ahmed. Sickle cell disease is a group of . haemoglobin. disorders . resulting from . the inheritance of the sickle β‐. anaemia. Assist.Prof. . :. Dr.Maysem. M. . Alwash. . Erythropoiesis. is the generation of red blood cells . .. .. -. All the circulating blood cells derive from . pluripotential. stem . cells in the marrow. مؤيد علوش. Introduction. THE HAEMOGLOBIN MOLECULE. Human . haemoglobin. is formed from two pairs of . globin chains . each with a . haem. . group attached.. Seven . different globin . chains are . The . sickling. phenomenon may be demonstrated in a thin. wet film of blood (sealed with a petroleum jelly/paraffin. wax mixture or with nail varnish). If . haemoglobin. S. is present, the red cells lose their smooth, round shape.