PPT-LDL is causal of atherosclerosis

Evidence from metaanalyses of Mendelian randomization studies prospective cohort studies and randomized controlled trials unequivocally establishes that LDL causes ASCVD. Dr. Gehan Mohamed Dr. Abdelaty . S. hawky. Learning objectives.  . Definition . of arteriosclerosis and mention the three patterns of it?. Mention definition, risk factors, gross morphology, and common sites of atherosclerosis?. By Krystian, Gabby, Yasmin, John. What is Atherosclerosis?. Atherosclerosis is a disease of the arteries characterized by the disposition of the plaques of fatty materials on their inner walls. . Arteriosclerosis is a general term for the thickening and hardening of arteries. Some hardening of arteries normally occurs when people grow older.. What is Atherosclerosis. Plaque builds up on artery walls causing the body to be deprived of oxygen.. Plague is made up of many substances from the blood.. Atherosclerosis is one of the main causes for many heart problems which is why it is sometime referred to as heart disease. . Terms. Arteriosclerosis – hardening of the arteries. Atherosclerosis – a form of arteriosclerosis. Venosclerosis. Arteriolosclerosis . Step One of Atherosclerosis. Endothelium injured. The tunica interna is injured in some way by bloodborne chemicals, hypertension, components of cigarette smoke or viral or bacterial infections. Almost any type of chronic infection – including periodontal disease can cause the damage.. Overview: Why is it Called “Heart Disease”. Atherosclerosis is a type of arteriosclerosis (the hardening of the arteries.) . A thrombus (blood clot) can form because of the buildup of plaque.. Diseases that can develop from atherosclerosis : angina (chest pain), carotid artery disease, coronary artery disease, and kidney disease.. Arterio. sclerosis. Thickening/Hardening of Arteries. Predominantly . Athero. sclerosis. Normal vessel structure. Pathophysiology of Brain & Body . USSJJQ-20-3. Normal Structure. Normal Structure. Thomboplastin. , Thrombin, . Prothrombin. , Fibrin, Fibrinogen. What do you know about atherosclerosis?. Atherosclerosis. L/O – What causes atherosclerosis?. ALL Describe the term Atherosclerosis. MOST Describe the steps that lead to Atherosclerosis. NORMAL ARTERIAL WALL. PROCESS OF ATHEROSCLEROSIS. RISK FACTORS OF ATHEROSCLEROSIS. COMPLICATIONS OF ATHEROSCLEROSIS. DEFINITION. Atherosclerosis. is defined as a progressive disease characterized by the accumulation of lipids and fibrous elements in the large arteries.. Society for Heart Attack Prevention and Eradication (SHAPE. ). SHAPE Guidelines. Prevention of Fatal Cardiovascular Events (Heart Attack & Stroke) based on the Detection . and Treatment of Subclinical . FamilialHypercholesterolemia:Screening,diagnosis andmanagementofpediatricandadultpatients ClinicalguidancefromtheNationalLipidAssociationExpertPanel onFamilialHypercholesterolemia AnneC.Goldberg,MD,FN Atherosclerosis. Shaesta. . Naseem. Vessel wall structure. Arteriosclerosis. It literally means "hardening of the arteries with loss of elasticity. ”. . . Three patterns:. Atherosclerosis. :. The . withlow. low-density lipoprotein have higher coronary atherosclerosis burden,. greater plaque progression and cardiovascular event risk. George . Karpouzas. , MD. Harbor. -UCLA Medical . Center. and the Lundquist Institute. Pathophysiology. Atherosclerosis is a chronic inflammatory disease. . Atherosclerosis . begins with fatty streak which is an accumulation of lipid laden foam cells in the intimal layer of the artery. . A cross-sectional study. 595 gout patients. T. ophi-free group (76.47%) and the tophi group (23.53%). Gout patients. Interaction with IR. Hepatic IR. (. HOMA index. ) . Peripheral IR. (. Gutt index.