PPT-Histologic and immunologic markers to predict renal recurrence in IgA vasculitis nephritis

Preliminary results K Abukasm AL Lapeyraque H Mathieu N Patey A Cambier Karma Abukasm Pediatric Nephrology Fellow CHU SainteJustine April 25th 2023 Introduction 1 IgA vasculitis formerly Henoch. Nephritis. What is Nephritis?. - It is essentially inflammation of the kidneys.. Types of Nephritis. Acute Nephritic Syndrome. Chronic Glomerulonephritis. A&P Review. Glomerulus- is an integral part of the nephron which is the basic unit of the kidney.. Prof. Hafiz . Ijaz. Ahmad. Department of Nephrology. Allama. . Iqbal. Medical College Lahore. Systemic Lupus . Erythematosus.  Systemic . Lupus . E. rythematosus. . (SLE) is a chronic inflammatory disease of unknown cause that can affect virtually any organ of the body. . Breast Cancer Recurrence. anytime, mostly occur in the first 3 – 5 years after initial treatment. can come back as a local recurrence (in the treated breast or near the mastectomy scar) or as a distant recurrence somewhere else in the body . . Dr. : Mohamed . Abdelhafez. . . . . Nephrology specialist . . MNGH. Agenda. DEFINITION AND EPIDEMIOLOGY. ETIOLOGY AND PATHOGENESIS. RENAL AND EXTRARENAL MANIFESTATIONS. IMMUNOLOGIC TESTS IN LUPUS NEPHRITIS. . Challenges and Options . Grand . Rounds. Presented by:. Sumona Kabir, DO. January 21. st. , 2015 . Overview . Case presentation. Definition of SLE. Pathophysiology. Classification . Details of each classification. Presented by: Olivia Wassef What is Systemic Lupus Erythematosus (SLE)? • An Autoimmune Chronic Inflammatory Disease ▫ Immune System produces proteins called autoantibodies that turns against The clinical features of the various . vasculitides. are protean and largely depend on the vascular bed affected (e.g., CNS vs. heart vs. small bowel). . Besides the findings referable to the specific tissues involved, the clinical manifestations typically include constitutional signs and symptoms such as fever, . OF AN . INFLAMMATORY CASE. DR NASIR FAROOQ BUTT. ASSISTANT PROFESSOR. DEPARTMENT OF MEDICINE . KING EDWARD MEDICAL UNIVERSITY. MAYO HOSPITAL LAHORE . INVESTIGATIONS . & MANAGEMENT. CASE . SCENARIOS. Lowell Dilworth-Chemical Pathologist. CKD. CKD and Bone disease. Mineral . metabolism controlled by kidneys. , intestine, parathyroid glands and . bone. The . kidney plays a critical role in mineral homeostasis regulation and, therefore, renal disease exerts widespread effects on the skeleton and soft . Signs and symptoms:. 1- . local. :. according. to the involved tissue. 2- . systemic. :(fever, . myalgia. , . arthralgias. , and malaise). Pathogenesis . 1- immune-mediated inflammation. . 2- . infectious pathogens. Hazem.K.Al-Khafaji. FICMS. Department of internal medicine. College of medicine. Al-Qadissyia University. Glomerular diseases. Lecture-5-. Acute Nephritic . Syndromes. Clinical types, associations & causes. when to biopsy? EMEESY Network educational day 2016 Dr. Louise Oni ( nee Watson ) NIHR Academic Clinical Lecturer in Paediatric Nephrology, Alder Hey Children’s Hospital, University of Liverpool My PRESENTER- Dr. B. PRADEEP.. CHAIR PERSON- Dr. T. SMRUTHI.. Vasculitis are group of disorders characterized by . immunoinflammatory. injury to vessel wall . leading to aneurysm, bleeding, stenosis, occlusion, thrombosis, embolism and ischemia. . Al-. Zahraa. Medical College and Scientific Research. Block: . Metabolic block. Lecture: . Renal involvement in systemic disease.