/
ANEMIA Key points Anemia is not a specific disease state but ANEMIA Key points Anemia is not a specific disease state but

ANEMIA Key points Anemia is not a specific disease state but - PowerPoint Presentation

clara
clara . @clara
Follow
371 views
Uploaded On 2022-02-14

ANEMIA Key points Anemia is not a specific disease state but - PPT Presentation

a sign of an underlying disorder There are several kinds of anemia A physiologic approach classifies anemia according to whether the deficiency in erythrocytes is caused by a defect in ID: 908799

iron anemia cell nursing anemia iron nursing cell blood interventions hemoglobin supplements vitamin sickle client b12 administer hgb monitor

Share:

Link:

Embed:

Download Presentation from below link

Download Presentation The PPT/PDF document "ANEMIA Key points Anemia is not a specif..." is the property of its rightful owner. Permission is granted to download and print the materials on this web site for personal, non-commercial use only, and to display it on your personal computer provided you do not modify the materials and that you retain all copyright notices contained in the materials. By downloading content from our website, you accept the terms of this agreement.


Presentation Transcript

Slide1

ANEMIA

Slide2

Key points

Anemia is not a specific disease state but a sign

of an underlying

disorder

There are several kinds of anemia.

A

physiologic

approach classifies anemia according to whether

the deficiency in erythrocytes is caused by a defect in

their production

(

hypoproliferative

anemia),

by

their

destruction (hemolytic

anemia),

or

by

their loss (bleeding).

Slide3

Key points a& Definitions

Anemia

is an abnormally low amount of circulating

RBCs,

Hgb

concentration,

Hct

concentration, or all.

Anemia results in diminished

O2-carrying

capacity and delivery to

tissues and

organs.

Therefore, the

goal of treatment is to restore and maintain adequate

tissue oxygenation

.

Causes of Anemia:

Blood loss.

Inadequate RBC production.

Increased RBC destruction.

Insufficient or defective hemoglobin.

Iron deficiency anemia is the most common anemia.

Slide4

Slide5

Slide6

Slide7

Risk Factors for Anemia

Acute

or chronic blood

loss

; menorrhagia, GI bleeding.

Increased hemolysis

; defective

Hgb

(sickle cell

disease), impaired

glycolysis

(G6PD) deficiency anemia, i

mmune

disorder

or destruction (transfusion

reactions

) Or mechanical

trauma to RBCs (mechanical heart valve,

cardiopulmonary bypass

)

Dietary inadequacy

; IDA,

megaloblastic

anemia, pernicious anemia, pica

Bone-marrow suppression

; exposure

to radiation or

chemicals, aplastic anemia

Slide8

Anemia?

Production?

Survival/Destruction?

The key test is the …..

Slide9

Slide10

Diagnostic Procedures and Nursing Interventions

Laboratory

Assessment

Complete blood count (

CBC)

RBCs

Hemoglobin

(

Hgb

)

Hematocrit

(

Hct

)

WBCs

Platelets

Slide11

Slide12

Slide13

Diagnostic Procedures and Nursing Interventions

RBC indices to determine the type and cause of most

anemias

:

Mean corpuscular volume (MCV): size of red blood cells

Normocytic – normal size

Microcytic – small cells

Macrocytic – large cells

Slide14

Slide15

Diagnostic Procedures and Nursing Interventions

Mean corpuscular hemoglobin (MCH): to determine the amount

of hemoglobin

per RBC

Normochromic – normal amount of

Hgb

per cell

Hypochromic – decreased

Hgb

per

cell

Mean corpuscular hemoglobin concentration (MCHC): to indicate

Hgb

amount relative to the size of the cell

Slide16

Slide17

Slide18

Diagnostic Procedures and Nursing Interventions

Iron studies

Total iron-binding capacity (TIBC) reflects an indirect measurement

of serum

transference.

Serum ferritin is an indicator of total body iron stores.

Serum iron measures the amount of iron in the blood.

Low

serum

iron and

elevated TIBC indicates iron deficiency anemia.

Slide19

Diagnostic Procedures and Nursing Interventions

Hemoglobin electrophoresis separates normal hemoglobin from abnormal.

It is used to detect thalassemia and sickle cell disease.

Sickle cell

test,

Schilling

test

(vitamin B12)factor,

used to differentiate between

malabsorption

and

pernicious anemia

.

Bone marrow examination diagnoses aplastic

anemia.

After

bone

marrow aspiration

:

Apply pressure to the site for 5 to 10 min.

Assess

VS

frequently.

Apply pressure dressing.

Monitor for signs of bleeding and infection for 24 hr.

Slide20

Slide21

Clinical Manifestation

Several factors influence

the development of anemia-associated symptoms:

the rapidity with which the anemia has developed,

the duration of

the anemia (

ie

, its chronicity

),

the metabolic

requirements of

the

client

,

other

concurrent disorders or

disabilities (

eg

, cardiac or pulmonary disease), and

complications

or

concomitant features

of the condition that produced the anemia

.

Slide22

Monitor for signs and

symptoms

May

be asymptomatic in mild cases

Pallor

Fatigue

Irritability

Numbness and tingling of

extremities

Dyspnea on exertion

Impaired skin healing

Brittle, spoon-shaped

nails (

Koilonychia

)

Cheilosis

Smooth, sore, bright red tongue

Sensitivity to cold

Brittle and ridged nails

Pain and hypoxia with sickle cell

crisis

Leg cramps

Pica

Slide23

Slide24

Slide25

Client’s respiratory status, activity tolerance, fatigueClient’s history regarding risk factors for anemiaGeneral appearance

Laboratory and diagnostic findings

Slide26

NANDA Nursing Diagnoses

Risk

for injury

Activity intolerance

Fatigue

Self-care deficit

Risk for

infection

Altered nutrition, less than

body

requirements

Altered

tissue

perfusion

Noncompliance

with prescribed therapy

Slide27

Nursing Interventions

Assess and monitor laboratory results.

Assess and monitor activity intolerance.

Encourage increased dietary intake of the deficient nutrient (iron, vitamin B12, folic acid).

Administer medications as prescribed; instruct the client on side effects.

Iron supplements

Oral iron supplements (ferrous sulfate, ferrous

fumarate

, ferrous

gluconate

)

Parenteral

iron

supplements

(

iron

dextran

)

Take with meals if gastrointestinal side effects occur.

Slide28

Nursing Interventions

Vitamin C improves absorption.

Do not take within 2

hr

of milk or

antacids (Al,

Ca

, Mg), proton pump inhibitor.

Stools will be black.

May cause constipation; increase intake of fluids and foods high

in fiber.

May use sustained release iron tabs

Use a straw with liquid iron to avoid staining of teeth.

Administer parenteral iron using Z-track method.

Erythropoietin (Procrit)

Cobalamin

(vitamin B12)

parenterally

or

intranasally

Folic acid supplements

Oxygen

Vitamin C supplements (to enhance iron absorption)

IV

fluids (sickle cell crisis, G6PD anemia)

Teach the client and family about energy conservation.

Slide29

Nursing Interventions

Teach the client the time frame for resolution.

Transfusions – lead to an

immediate improvement in blood cell counts

and client

signs and symptoms.

Typically only used when the client has significant symptoms

of

anemia

because of the risk of blood-borne infections.

Oral supplements – 4 to 6 weeks for increase in RBCs

Vitamin B12 supplements – increased RBCs in 1 week

Vitamin B12 supplementation

B12 supplementation can be given orally if the deficit is due to

poor

dietary

intake.

However

, with pernicious anemia it must be

given

parenterally

because it won’t be absorbed if given orally.

Administer parenteral forms intramuscularly or deep

subcutaneous to

decrease irritation. Do not mix other medications in the syringe.

Pernicious anemia must be treated for life.

Slide30

Nursing Interventions

Folic acid supplementation

Can be given orally or

parenterally

.

Large doses will stain urine dark yellow.

Slide31

Complications and Nursing Implications

Monitor

the client for signs of hypoxemia: dyspnea, decreased oxygen

saturation, central

cyanosis.

Administer oxygen therapy. Monitor SaO2.

Administer blood transfusions as ordered.

Assess the neurologic status of clients with macrocytic

anemias

.

Vitamin B12 is essential for neurological function.

Administer supplements. Notify

the provider of any changes in mentation.

Slide32

Complications and Nursing Implications

Sickle Cell Crisis

A

n

autosomal recessive

condition.

At least 40% of total hemoglobin is the abnormal hemoglobin S (

HbS

)

in sickle

cell disease.

When

exposed to hypoxic environments, it contracts and

clusters together

within the cell.

Cells become rigid, forming a “sickle” or “C” shape which causes them

to clump

together and impede blood

flow; leading to

further tissue hypoxia.

S &

Sx

: cyanosis, abdominal pain, leg and back pain,

low-grade fever, seizures, and stroke manifestations (weakness,

paresthesia

).

Administer oxygen and hydration therapy

. As well as

analgesia.

Organ failure can result if left untreated.

Slide33

Slide34

Slide35