PPT-Renal Physiology Overview role of the kidney

Remove metabolic products toxins and acid Removal of non volatile acids lactate sulphuric phosphate acetoacetate beta hydroxybutyrate toxins Salt balance Water balance Ph balance Produce . Part One - Overview. Digital Laboratory. It’s best to view this in . Slide Show . mode, especially for the quizzes.. This module will take approximately . 60 . minutes to complete. .. After completing this exercise, you should be able to (blue text in this module, tan text in subsequent kidney modules):. on . Chronic . Kidney . Disease. Maria Ferris, MD, MPH. PhD. Associate Professor. Director of the Pediatric Dialysis and Transplant Services. UNC Kidney Center, Chapel Hill, NC, USA. Akash. and his fan club. Jordan M. Symons, MD. University of Washington School of Medicine. Seattle Children’s Hospital. Seattle, WA - USA. 8th . International Conference On. Paediatric. Continuous Renal Replacement Therapy (. Part One - Overview. Digital Laboratory. It’s best to view this in . Slide Show . mode, especially for the quizzes.. This module will take approximately . 60 . minutes to complete. .. After completing this exercise, you should be able to (blue text in this module, tan text in subsequent kidney modules):. Does your GI tract absorb into blood all nutrients? ions? water?. Now imagine that you drink water: 1 glass, 2, 3, 4, 5 glasses (1L). . Does the additional 1 liter of water stay inside . the blood vessels (18% increase from 5.5L)?. CONGENITAL RENAL DISEASE IMAGING. F.Hallaji. MD. radiologist. HYDRONEPHROSIS . Pregnancy in fetuses with unilateral or bilateral ANH should proceed to term. , except if complicated by severe . in Neonates. Jordan M. Symons, MD. University of Washington School of Medicine. Seattle Children’s Hospital. Seattle, WA. 9. th. International . pCRRT. Conference on . Pediatric Continuous Renal Replacement Therapy. Renal System. 1. KIDNEY FUNCTIONS. . . MAINTAINING OF HOMEOSTASIS. maintain the blood volume and the normal composition of body fluid compartments. excrete waste products ( urea, creatinine, uric acid, NH₃(ammonia), which are toxic for the organism . Dr.Hazem.K.Al-Khafaji. MBCHB.D.M.FICMS . The clinical presentation of renal diseases include:-. 1- Nephritic syndrome. 2-Nephrotic syndrome. 3- Acute kidney injury(AKI),the old name acute renal failure.. (Increase frequency of urine) >1500ml/day. . Causes . Diabetes mellitus. . . . Acrete. Nephritis / Nephrotic syndrome. . . Post . Pitutary. tumour. 2. Oliguria (Decrease frequency of urine) < 500 ml/day. st. century?. By: . . Baskar . ,. . WDHB. , . Renal Service Auckland. . New Zealand. Presented at : “4. th. International Conference on Nephrology and Therapeutics” – Baltimore USA. International Society of Nephrology (ISN) and the International Federation of Kidney Foundations (IFKF) . . Digital Laboratory. It’s best to view this in . Slide Show . mode, especially for the quizzes.. This module will take approximately 60 minutes to complete.. After completing this exercise, you should be able to (blue text in this module, tan text in subsequent kidney modules):. ACUTE KIDNEY INJURY. Abrupt reduction in renal function over hours to days that we believe has the potential to be reversed . LAB TRENDS. HPI. AKI VS CKD. Acute Kidney Injury. Chronic Kidney Disease. Sh.A.Karbalaie. F.R.C.S.,F.I.C.S.,F.I.CM.S. Consultant urologist. Head of Dept. of surgery. learning objectives. 1-identify the secretory and excretory functions of the kidney.. 2-describe the normal anatomy of the kidney..