PPT-Living with Severe Rheumatoid Arthritis

By Danielle Lyner Goal To exhibit a wide variety of tools to help people that suffer from severe rheumatoid arthritis improve and facilitate their everyday living Jar Opener Enablers Jar Opener amp Closer. John Imboden MD. Rheumatoid arthritis: typical presentation. Prevalence 1%. Female > male (3:1). Peak onset: age 30s to 40s. Insidious onset of joint pain & AM stiffness lasting hours. By, Marissa Miuccio. Definition. What is it. Rheumatoid arthritis is a chronic inflammatory disorder that typically affects the small joints in your hands and feet. Rheumatoid arthritis affects the lining of your joints causing a painful swelling that can eventually result in bone erosion and bone deformity. . Stephanie Arrington. Joint Replacement . Research suggests that more than a million people a year are getting a total joint replacement. . More than half will be younger than 65. . Joint replacement are most typically an artificial hip or knee.. Osteoarthritic Patients. Steven Karpas, DMD. Geriatric Dentistry Fellow. Boston University School of Medicine/ . Dept. of Geriatrics. Boston University Henry M. Goldman. School of Dental Medicine. Paula K. Friedman, DDS, MSD, MPH. Dr R B Kalia,. Additional Professor ,. Department of Orthopaedics. Leaning Objective. Clinical Features of RA. Investigations . Diagnosis. Indications for Surgery in Arthritis. Various procedures possible. Rheumatoid Arthritis . Inflammatory vs Noninflammatory Arthritis. ACR/EULAR Criteria for Diagnosing RA Before Joint Damage. Limitations to Classification of RA. Biomarkers in RA. Differential Diagnosis. FRCPEdin. ; . FRCSEdin. “A . chronic progressive disease causing inflammation in the joints and resulting in painful deformity and immobility, especially in the fingers, wrists, feet, and . ankles”.. FRCPEdin. ; . FRCSEdin. “A . chronic progressive disease causing inflammation in the joints and resulting in painful deformity and immobility, especially in the fingers, wrists, feet, and . ankles”.. : Autoimmune disease that causes chronic inflammation of connective tissue. Joint tissue is permanently affected, but any connective tissue of the body might be involved.. Etiology. : Associated with the production of abnormal antibodies that attacks or attaches to the bodies own cells and tissues. Presence of the antibody called Rheumatoid factor (RF) in the affected individuals blood is indicative of the disease.. Gregory Gardner, M.D.. Gilliland-Henderson. Professor of Medicine. Division of Rheumatology. University of Washington. Outline of Discussion. Pathophysiology. Clinical Features. Treatment Update. Perioperative Management. Page 1 Rheumatoid Arthritis People have long feared r heumatoid arthritis (commonly called RA) as one of the most disabling types of improved for many people with newly diagnosed (detected) RA . Sonya Abraham. Senior Lecturer/Honorary . Consultant. Imperial College Healthcare NHS Trust. Medical . Student Lecture (Yr 5). May 2012. Rheumatoid Arthritis. Historical. Epidemiology. Disease Pathogenesis. Dr JW . Mojapelo. . MBChB. (. Pret. ). Dr M . Libala. . MBChB. (. Pret. ). Introduction. Arthritis is defined as a group of medical conditions accompanied by joint inflammation followed by various systemic symptoms and signs . withlow. low-density lipoprotein have higher coronary atherosclerosis burden,. greater plaque progression and cardiovascular event risk. George . Karpouzas. , MD. Harbor. -UCLA Medical . Center. and the Lundquist Institute.