PPT-Haemostasis Hemostasis :

The stoppage of bleeding or hemorrhage The normal haemostatic response to vascular damage depends on a closely linked interaction between the blood vessel wall circulating platelets. Bipolar electric coagulation allows controlling subcutaneous bleeding Muscular dissection is often per formed by monopolar electric coagulation Hemostasis of the muscular layers is performed by bipolar coagulator and maintained by mechanical compres Platelets aggregation. Studying Platelets Disorders. . Numbers. . CBC. PLT count. PLT morphology. Function. . Bleeding . Time (BT). Platelet Aggregation. Whole blood aggregation . Platelet-rich plasma aggregation. and . Basic Hardware. Dr. K. SURESH. SK Hospital and KIMS Hospital, Trivandrum. Vascular access. F. irst . important step in diagnostic / interventional . catheterization. Percutaneous approach has replaced the . Platelets aggregation. Studying Platelets Disorders. . Numbers. . CBC. PLT count. PLT morphology. Function. . Bleeding . Time (BT). Platelet Aggregation. Whole blood aggregation . Platelet-rich plasma aggregation. minimal thermal damage, targeted hemostasis and precise tissue removal COBLATION™ Plasma Technology COBLATION™ plasma technology Wands Our clinical use of plasma for tissue ablation and di Anaesthetic. Practice. G . Ogweno. Dept of Medical Physiology. Kenyatta University. Why Colloids over crystalloids?. Crystalloids. :. Extravasate. =only 25% remains in circulation after 20 . mins. used for . Sulistyo. . Rini. , . MSc. Five Stages of . Hemostasis. Vessel spasm. Formation of the platelet plug. Blood coagulation or development of an insoluble fibrin . clot. Clot retraction . Clot dissolution . Hematology Oncology Fellow. Hemostasis. Process of blood clot formation at site of vessel injury. Response must be. Quick. Localized. Carefully regulated. Phases of hemostasis. Formation of platelet plug. haemostasis. Vascular bleeding disorders:. .The underlying . abnormalities are. either in the . vessels themselves. . perivascular. connective tissue. .. Each could be. Inherited. Acquired. . autosomal. , thrombosis, NSAIDs and atherosclerosis. . . Ailbe. My guide to pharmacology. Pharmacology makes up most of LCRS 1 (a long with endocrinology & RDA). I would recommend spending a disproportionally small amount of time revising pharm, as the smaller topics like RDA, . . Alfonso Iorio, on behalf of the working group. DISCLOSURES. Financial conflicts of interests. McMaster University has received . funds for research. and . service agreements . from Bayer, . Baxalta. M. AND J. E. WYNANDS, M.D., C.IVI.~ THE V~Y F_~aLY STUVmS of this subject, many controversies existed regard- ing the various factors and mechanisms involved in the dotting process. One simply has to Julie Laloy. December. 1st, 2014. Nanotek. & Expo 2014 – San Francisco. Introduction. . – . Hemolysis. – . Primary. . haemostasis. – . Secondary. . haemostasis. – . Fibrinolysis. : . Haematology 5. . and . 6. 0900 . Lecture: . Haemostasis. : . Professor . David Lane. Film. . 10.00 . Lecture: Abnormalities of . Haemostasis. : . Dr Carolyn Millar. Department of Haematology. Hammersmith Hospital Campus.