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Cognition in psychopathology: can we say one way or another whether certain cognitive Cognition in psychopathology: can we say one way or another whether certain cognitive

Cognition in psychopathology: can we say one way or another whether certain cognitive - PowerPoint Presentation

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Cognition in psychopathology: can we say one way or another whether certain cognitive - PPT Presentation

Dr Tisha J Ornstein CPsych Department of Psychology Ryerson University Overview Why are neuropsychologists special particular way of thinking eg Phineas Gage research approach Discussion about Attention Deficit Hyperactivity Disorder ADHD and relevance as related to Traumatic ID: 628333

ocd adhd disorder inhibition adhd ocd inhibition disorder children deficits response cognitive executive attention control memory amp studies injury

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Slide1

Cognition in psychopathology: can we say one way or another whether certain cognitive processes are unique to a given disorder?

Dr. Tisha J. Ornstein, C.PsychDepartment of PsychologyRyerson UniversitySlide2

Overview

Why are neuropsychologists special – particular way of thinking, e.g., Phineas Gage; research approachDiscussion about Attention Deficit Hyperactivity Disorder (ADHD) and relevance as related to Traumatic Brain injuryLink between ADHD and Obsessive-Compulsive Disorder; research endeavours to be discussed by graduate student: Peter EgetoSlide3

Phineas Gage

Before

the accident, Phineas Gage had been a capable and efficient foreman who was looked on as

a

shrewd, smart business man.  Afterwards, he was fitful, grossly profane, impatient and obstinate. His friends said he

was,

“no longer Gage.”

Massive

damage to the frontal lobes can cause dramatic changes in personality while keeping sensation, movement, consciousness and most cognitive faculties intact. Slide4

Neuropsychology: As a Way of Thinking…

Humans have evolved a range of subtle cognitive control mechanisms to regulate more basic cognitive processes in the service of future-oriented goals in an uncertain environment

These mechanisms are known as executive control

Executive control includes anticipation, goal selection, planning, and response inhibition, among other processes.

Executive control depends on a series of partially segregated frontal subcortical loops (e.g., Alexander, Delong, & Strick, 1986; Lichter & Cummings, 2001).

Perturbations of these loops and the resultant effect on executive control are implicated in various neuropsychiatric disorders, such as attention deficit hyperactivity disorder, and obsessive compulsive disorderSlide5

Frontostriatal circuits and Neurocognitive Processes

Neurocognitive processes mediated by the circuits include attention, the inhibition of inappropriate responses, shifting between thoughts or actions, performance monitoring, decision-making, planning

ability,

and using working memory to help guide behavior.

Dysregulation

of frontostriatal circuits likely underlies many of the symptoms related to the disorders of

interest.

Would

it not be wonderful to identify key cognitive mechanisms associated with a disorder? What are some implications? Slide6

Research Approach

Disorders of the frontostriatal system:

Attention deficit

hyperactivity

disorder, Obsessive-compulsive

disorder

Other disorders that are frontal with respect to their cognitive characteristics:

Traumatic Brain injury

Understanding the nature of neuropsychological characteristics associated with various conditions:Consider neuropsychological test battery or neurocognitive measures sensitive to the disorder of interest;Comparative approach Co-morbidities (pain, anxiety, depression)Change and variability (early work) Long-term follow-up (determine impact of injury on development) (future endeavour)Ultimate goal

is to elucidate the mechanisms that underlie abnormal behaviourSlide7

Introduction to Attention Deficit Hyperactivity Disorder (ADHD)

Most common developmental disorderAffects children, youth, and young adults

Costly, impairing and persistent

Phenotypically complex

Cognitive deficitsSlide8

Neurocognitive Deficits

Neurocognitive deficits in ADHD, and more specifically executive deficits, extend to adults, although the research lags behind child/adolescent-focused studies.

DSM-5 states, “Individuals with ADHD may exhibit cognitive problems on tests of attention, executive function, or memory, although these tests are not sufficiently sensitive or specific to serve as diagnostic indices

.” “No biological

marker

is diagnostic for ADHD.” (p. 61).

Even so, certain neuropsychological domains have been proposed as crucial.Slide9

Neurocognitive TestsSlide10

Response

InhibitionMotor Control/

Fluency/Syntax

Self-Regulation

Of Affect/

Motivation/ArousalInternalizationOf SpeechReconstitution

WorkingMemory

Barkley’s Theory of ADHD

Barkley asserts that these deficits manifest in ADHD children through:

Ability to adapt to new information

Ability to predict what will happenAbility to generate responses to future infoADHD = “nearsighted when it comes to time” Slide11

Which tests should we use? Why?

Consider:Why is the subject being referredWhat is/are the presenting issues

How old is the subject; Male or female

Any presenting difficulties, like hearing impairment

What are the cognitive concerns presented in the literature

Neuroanatomical substrates

Test parametersSlide12

Stop-Signal Paradigm

Response inhibition considered a signature deficit of ADHD; recent meta-analysis reported an effect size of .62 for SSRT in ADHD as compared to controls (Lipszyc & Schachar, 2010), and not related to the ‘go’ response. Slide13

Caveat; even so…

Not all children are impaired; however,

Relatives of impaired children on the stop signal reaction time task performed worse than relatives of non-impaired ADHD and healthy control children;

has been replicated.

McAuley and colleagues

(

2014) said that certain of executive control processes, including response inhibition, are markers of genetic risk for ADHD. Looking to evaluate whether certain skills are state-independent. Children were reassessed as adolescents. They compared ADHD children to peers and then ADHD subgroups (persistent, remittent, and partially remittance forms of the disorder) on average five years later. They found that response inhibition distinguished ADHD children from their unaffected peers, but also that the deficit persisted regardless of the course of the disorder. Slide14

Traumatic brain injury (TBI)

Leading cause of death and disability 375 per 100,000 person years500,000 cases / yr. to ER

40% admitted

80% + are mild; 80% receive attention

7% moderate

8% severe30,000 suffer permanent disability

Children, youth and young adults50% < 19 years; 25% < 9 yearsPhenotypically complex Cognitive deficitsSlide15

Pre-injury mental illness

15-40% have pre-injury mental illness ADHD most common (upwards of 25% of TBI cases)

Aggression

Phobia, SAD, ODD, OCD, GAD, LD

Multiple problemsSlide16

Pre-injury predictors of new disorders

Mental illnessInjury severityNumber of previous head injuriesLow pre-injury adaptive functioning SES, psychosocial

adversity, family

functioningSlide17

Post-injury mental illness

Secondary ADHD (S-ADHD: ADHD develops after TBI)Persist, inattentivePersonality change disorder

Labile, aggressive, disinhibited, apathetic, paranoid

Oppositional defiant and conduct disorder

Post-concussive symptoms

Headaches, dizziness, fatigue, irritability, insomnia, loss

of concentration and memory, noise and light sensitivityComorbidityComorbidity predicts severity and increases health care needs. Slide18

Traumatic Brain Injury and Attention Deficit Hyperactivity Disorder

Association between ADHD and TBI:Those with ADHD are at a greater of suffering a TBI due to the nature of deficits – risk-taking behavior, impulse control issues (cf. Adeyemo, 2014)

TBI has been shown to result in

S-ADHD

Perhaps

TBI and/or inhibitory control performance shapes the expression of ADHD, leading to an etiologically distinct form of ADHD.Slide19

274

children6–14 yrs2 years post TBI

SSRTSlide20

6-16 yrs

Injured at ~ 10 yrs

141 at

6 monthsSlide21

126 followed at

12 monthsSlide22

NOTE:

OCD; SCHZ Adults includedSlide23

Obsessive-Compulsive Disorder

Estimated lifetime prevalence of 2 to 3%; approximately 600 000 Canadians will be affected by this illness in their lifetime.

Among adults, men and women are equally affected, but in adolescents boys are more commonly affected.

The disorder begins most often in adolescence or early adulthood; it may begin in childhood.; around one-half to one-third of adult OCD cases have their onset in childhood or adolescence.

It is usually a chronic course that waxes and wanes

.

OCD may be accompanied by depression, eating disorders, or other anxiety disorders. Slide24

Neuropsychology

Few early neuropsychological studies in adults with OCD; results were inconsistent.

More recent neuropsychological studies of executive functions in adults with OCD have reported increased difficulty with switching cognitive set, generating strategies (i.e., executive planning), and response inhibition; memory difficulties are said to be related to problems with strategic learning. Slide25

Few studies have examined executive functioning in children with OCD. Of the handful of published studies, findings are inconsistent.

It is important to study children with OCD for many reasons:

Executive processing plays an important role in children’s learning and problem solving.

Can assess the underlying deficit unbiased by the effects of treatment and the consequences of the disorder itself

.

NeuropsychologySlide26

Research ArticleDEPRESSION AND ANXIETY

 NEUROPSYCHOLOGICAL PERFORMANCE IN CHILDHOOD OCD:

A

PRELIMINARY STUDY

 

Tisha J. Ornstein, Ph.D., C.

Psych, Paul Arnold, M.D. F.R.C.P (C), Katharina Manassis, M.D. F.R.C.P (C), Sandra Mendlowitz, Ph.D., C. Psych, and Russell Schachar, M.D. F.R.C.P (C)Background: Neuropsychological deficits have often been found in studies

of adults with obsessive compulsive disorder (OCD). However, few studies have examined such impairment in children with OCD and of those studies published, the results are mixed. Methods: In the present study, 14 OCD children were compared to 24 healthy developing children of similar age and intellectual ability on a series of neuropsychological tests that assess response inhibition, abstract reasoning and problem solving, planning ability, verbal and nonverbal fluency, working memory, attention and information processing speed, and visual

and verbal memory and learning. Results: No significant differences emerged between the children with OCD and healthy controls for working memory, verbal fluency, attention, information processing speed, concept formation/abstraction, and response inhibition. We observed some deficits and a trend toward performance differences between the groups for psychomotor speed and attention, cognitive flexibility, nonverbal fluency, planning ability, and verbal memory and learning. Results are partially consistent with those found in adults with

OCD. Findings were not related to depressive symptoms or self-report feeling of anxiety. Conclusions: This preliminary survey indicates that OCD children may have deficits for cognitive flexibility and planning ability and differ from adults with OCD in not presenting with poor response inhibition or memory deficits. Larger, multi-site studies are warranted to help delineate the neurocognitive deficits associated with childhood OCD. Slide27

Child Literature

TestsFindings

Beers, 1999

Digit Span, Stroop, WCST, COWA, TOH, Go/No-Go,

CVLT, TMT

Null findings;

participant with OCD did better on some measuresShin, 2008CPT, WCST, TMT-B, RCFTNo differences on CPT, TMT-B, RCFTWooley, 2008Stop Signal Task, Motor Stroop

No differencesOrnstein, 2010Stop Signal, WCST, Spatial Span, n-Back, DKEFS ,TMT, CVLT, RCFTNo deficits, but trends towards deficits

in cognitive flexibility and planning abilityBehar, 1984Money’s Road Map Test of Directional sense, Maze Learning, Rey Word Learning List, RCFTDeficits in mental rotation in space & learning mazesAndres, 2007

RCFT, RAVLT, TMT, WCST, COWA, StroopDeficits in visual memory & visual organization. Slide28

ADHD and OC Behaviours

Background:Collaboration with Sick Kids Hospital (Drs. Schachar &

Crosbie

)

Children with ADHD and OC behaviours

ADHD: 5%

OCD: 1.2%ADHD+OCD: 10-33%

ADHD+OCB: 11.2%Slide29

ADHD and OC Behaviours

Background:ADHD & OCD: common neurobiological abnormalitiesCortico-striatal-thalamic pathwayAnterior cingulate cortex activity: ↓ in ADHD, ↑ in OCD

(

Brem

,

Grünblatt, Dreschler, Riederer &

Walitza, 2014)Inhibition impairmentsBehaviour & thought suppressionInhibition in ADHD+OCB: Arnold et al., 2005Somewhat ↑ inhibition in ADHD+OCB vs ADHDSlide30

ADHD and OC Behaviours

Measures: 3 inhibition tasksCognitive inhibition: Eriksen Flanker Task (distractor suppression)Response inhibition: Go/No-Go (response inhibition) Response inhibition: Stop

Signal Task (ongoing response

inhibition)

‘L’

‘X/O’

‘X’Slide31

ADHD and OC Behaviours

Results:

Task

Variable

ADHD (n=85)

OCB (n=10)

ADHD+OCB (n=14)

Controls (n=68)Post-hocGo/No-GoMCRT

734.9 (165.5)657.6 (165.6)683.54 (129.0)634.6

(121.1)ADHD > HCMCRT SD

253.0 (105.8)178.8 (78.9)231.9 (149.5)

188.8 (80.7)

ADHD > HC

Eriksen

Flanker

MCRT SD

283.9

(

95.1)

240.9

(

68.4)

241.7

(

67.0)

243.8

(

91.1)

ADHD > HC

MIRT SD

298.6

(

140.1)

293.8

(

314.2)

327.3

(

158.9)

228.5

(

125.7)

ADHD > HC

Stop Signal

All

n.s

.Slide32

ADHD and OC Behaviours

Conclusions:ADHD slower and more variableGeneral trend: ADHD > ADHD+OCB > controls > OCBProcessing speed vs variability?No differences on processing speed index (WISC-IV coding & symbol search)

OC symptoms: ↓ inhibition/inattention deficitsSlide33

ADHD and OC Behaviours

Future directions:Other inhibition measures (e.g., Stroop), larger sample, full OCD diagnosesOther studies:EEG and neuropsychogical

testing in OCD

Error related negativity (ERN), N2 (conflict monitoring), P3 (inhibition)Slide34

Current Endeavours

Decision-making in relation to:Inhibition;Quality of life and disabilityComponents of decision-making – Intolerance of uncertainty; reassurance seeking; impulsivity; subjective reporting (Frost Indecisiveness Scale…)Looking at these features in OCD and spectrum-related disordersSlide35

THANK YOU!

Acknowledgement: Ryerson research lab, Hospital for Sick Children, Sunnybrook Health Sciences Centre, and US collaborators.